Why does ataxia walk unsteadily?

Unsteady walking is a problem of ataxia, and there are many causes of ataxia, specifically in the following aspects:

1. Cerebellar ataxia: cerebellar hemisphere or cerebellar vermis lesions, such as cerebellar infarction or hemorrhage, cerebellar abscess, and tumor invasion, will lead to cerebellar walking instability and ataxia, such as walking too wide, staggering, etc. It is necessary to carry out ** for cerebellar lesions, the cerebellar compensatory ability is relatively strong, and the symptoms of unsteady walking will be reduced after **;

2. Deep sensory impairment: it will be further aggravated at night, resulting in subacute combined degeneration of the disease of deep sensory impairment, mainly due to the patient's impaired absorption of vitamins, intramuscular vitamin B12 can be given, and the situation of unsteady walking will be improved in the short term;

3. Vestibular organ lesions: when patients have vertigo, they may walk unsteadily and skew to the side of the lesion, and the vestibular disease needs to be corrected;

4. Frontal lobe lesions: lead to frontal lobe ataxia, resulting in unsteady walking.

Ataxia occurs due to cerebellar lesions and damage to the cerebellar spinal tract, and we know that the function of the cerebellum is to control balance, and when cerebellar lesions or cerebellar medullary surgery are damaged, there will be symptoms of unsteady walking.

Western medicine currently lacks a special **** method, telling that this disease cannot be cured, and can only take some nutritional neurohormone drugs or **** to improve symptoms, and these methods have no effect. The use of traditional Chinese medicine Rongjin Xing Yi decoction is suitable for the patient's condition to prescribe**, in order to achieve the medicine to the disease! Early detection, early attention, early recovery!!

Unsteady walking, most likely due to ataxia. Ataxia is further divided into cerebellar ataxia, sensory spatial disorder, vestibular ataxia and cerebral ataxia, all of which can lead to unsteady walking, as follows:

1. Diseases that can cause cerebellar ataxia, including hereditary cerebellar ataxia, as well as primary or metastatic tumors of the cerebellum, cerebellar infarction, hemorrhage, cerebellar inflammation, or cerebellar damage or traumatic malformation of the cerebellum due to chronic alcoholism and other reasons, can cause cerebellar ataxia and lead to walking instability. Clause.

2. Sensory ataxia, mainly caused by deep sensory impairment, is generally the most common in subacute combined degeneration of the spinal cord, etc., which can involve the posterior cord of the spinal cord. Clause.

3. Vestibular ataxia is mainly seen in diseases of the vestibular system, such as various damages including the inner ear labyrinth, vestibular nerve, brainstem, and vestibular nerve nucleus, such as labyrinthitis, vestibular neuritis, and subtentorial tumors. Clause.

4. Cerebral ataxia mainly includes frontal lobe ataxia caused by frontal lobe lesions of the brain, and temporal lobe ataxia caused by temporal lobe lesions.

Cerebellar atrophy is one of the common types of cerebral atrophy, but there are still many people who do not understand the nature of cerebellar atrophy. With the development of medicine, the research on cerebellar atrophy has been deepened, and there is a relatively clear statement on cerebellar atrophy. Understanding the common causes of cerebellar atrophy is very helpful for the correct **cerebellar atrophy.

There are many causes of cerebellar atrophy in daily life.

Spinocerebellar ataxia is the main type of inherited ataxia. Common features are middle-aged onset, autosomal dominant inheritance, and ataxia. In addition to cerebellar ataxia, clinical manifestations may be accompanied by ocular movement disorders, slow eye movements, optic nerve atrophy, retinitis pigmentosa, pyramidal tract signs, extrapyramidal signs, muscle atrophy, peripheral neuropathy and dementia.

**。Caused by abnormal amplification of CAG copy number in exons of genes to produce polyglutamine.

Clinical presentation. 1.Infancy.

Walking with unsteady gait and shaky limbs. Slowness of motor response and poor accuracy.

2.Medium term. Speech is slurred and the pitch cannot be controlled. If the eye movement is not smooth, the image is prone to "overlap". Muscle incoordination worsens, and it is impossible to write. Sometimes I feel that I have difficulty swallowing and I choke easily when eating.

3.Advanced. Speech is extremely unclear, and it is impossible to speak. Limb weakness, unable to stand, need to rely on a wheelchair to walk. The ability to comprehend gradually decreases, and finally loses consciousness and falls asleep.

Examine. 1.Clinical examination of the cranial nervous system.

2.Magnetic resonance imaging (MRI) test.

3.Genetic testing.

**。This disease is a degenerative disease, and there is currently no drug that can be used, and the focus is on rehabilitation, so that patients can maintain the highest level of self-care ability as much as possible. It can also be done through stem cells**.

After **, the patient can recover the ability to take care of himself, move, speak, and write. Levodopa relieves tonic and parkinsonian symptoms, chloraniline butyric acid reduces spasticity, and amantadine improves ataxia.

The main purpose of ataxia is to control the condition and prevent progression, including balance training and coordination training, which need to be combined with symptoms, mainly in the following ways:

1. Improve the basis of the patient's exercise posture, enhance proximal stability, improve balance, and strengthen small-range movement. 2. Improve the synergistic effect of agon, synergistic and antagonist muscles, so that the patient's motor balance becomes stable and smooth. 3. Appropriate anti-gravity exercises, such as standing, sitting down for gymnastics exercises, repeating the eyes open and closed, from sitting to standing, you can also carry out left and right hand throwing exercises, repeated training along the horizontal line and below the knee joint, training from sitting to standing.

It is recommended that the condition should be assessed by a professional ** teacher and then carried out under the guidance of the ** teacher to improve efficiency.

Clinical presentation. 1.Infancy.

Walking with unsteady gait and shaky limbs. Slowness of motor response and poor accuracy.

2.Medium term. Speech is slurred and the pitch cannot be controlled. If the eye movement is not smooth, the image is prone to "overlap". Muscle incoordination worsens, and it is impossible to write. Sometimes I feel that I have difficulty swallowing and I choke easily when eating.

3.Advanced. Speech is extremely unclear, and it is impossible to speak. Limb weakness, unable to stand, need to rely on a wheelchair to walk. The ability to comprehend gradually decreases, and finally loses consciousness and falls asleep.

Walking unsteadiness is one of the symptoms of ataxia, but not all walking steadiness is ataxia. Let's take a look at the symptoms of supply imbalance.

The symptoms of ataxia mainly include the following: 1. Changes in posture and gait: worm lesions cause trunk ataxia, unsteady standing, staggering gait, feet far apart when walking, shaking, and even difficult to sit firmly in severe cases, the upper vermican is damaged and falls forward, the lower vermire is damaged and falls backwards, and the ataxia of the upper limbs is not obvious.

Cerebellar hemisphere lesions deviate or tilt toward the affected side when walking. 2. Disorder of voluntary movement coordination: cerebellar hemisphere damage leads to ataxia of ipsilateral limbs.

Manifests with poor distance resolution and intention tremor, with heavier upper extremities and more pronounced tremor as the movement gets closer to the target. Coarse nystagmus may be seen when the eye is fixed to the side of the lesion. Ataxia of the upper limbs and hands is the most severe, inability to complete coordination and fine movements, inability to coordinate, and abnormal rapid recovery and alternating movements.

The more you write the handwriting, the bigger it gets (capitalization). 3. Speech disorder: due to the ataxia of articulatory muscles such as the lips, tongue and larynx, the speech is slow and slurred, and the voice is intermittent, frustrated or explosive, showing poetic or explosive language.

The main symptoms of ataxia are unsteadiness when walking and shaky limbs. Slowness of motor response and poor accuracy. Later, he developed a slurred pronunciation when speaking and was unable to control the pitch.

Irregular eye movements. Muscle incoordination worsens, and it is impossible to write. Sometimes I feel that I have difficulty swallowing and I choke easily when eating.

Later stage, speech is extremely unclear, or even unable to speak. Limb weakness, unable to stand, need to rely on a wheelchair to walk. The ability to comprehend gradually decreases, and finally loses consciousness and falls asleep.

As long as it is analyzed according to the specific situation, sometimes the rest is not good, there is no energy, and the walking will be unstable.