Can anyone tell me the symptoms of Brain Protein Demyelination and how to treat it with medication?

Clinical symptoms are occasionally trance-like, restless, dizzy, depressed, anxious, obsessive-compulsive, claustrophobia, claustrophobia, headache, abnormal vision or brain discomfort, as well as mild local numbness, muscle weakness, etc., which are not appropriate and prone to recurrence of nerves, and can invade the entire center in severe cases, resulting in spastic paralysis and life-threatening. In the early stage, most of them are hormones and nutrition, but the efficacy is difficult to control, and it will lead to immunity to the body, and even viral infections and inflammatory infections will cause nerve damage again and further aggravate the symptoms. If the correct ** damaged nerve is not obtained, it will ** and late multifocal sclerosis or softening of the lesion, and re-damage the nerve will cause secondary dementia or spastic paralysis.

The best recovery depends on early**. No information can only provide you with a theoretical ** plan: Chinese and Western compound to enhance immunity, improve the body's ability to suffer from high disease, chronic anti-inflammatory to prevent inflammation from damaging nerves again, improve blood flow of affected nerves to nourish nerves, regulate nerve malac scarring, prevent late ischemia and further aggravation of lesions, and at the same time stimulate and activate nerves to regenerate and repair lesions, prevent the condition from restoring the best nerve function as soon as possible.

Need help sending magnetic resonance** for your guidance.

After the edema of the white matter will lead to the loss of myelin, also known as the white matter demyelination disease or demyelinating encephalopathy, demyelinating encephalopathy has primary, secondary, ** and late type and ** many, the primary demyelination is mostly formed during pregnancy or childhood, and there is a difference in severity, the degree of nerve damage is related. Severe patients are more likely to have postpartum cerebral palsy, mild damage and damage to non-functional brain tissue areas can occur at any age, and mild patients often develop neurological disorders or dizziness, headache, depression, anxiety, insomnia, forgetfulness and cerebrospinal syndrome after the age of 50 before MRI (cervical spondylosis and lumbar spondylosis occur after muscle spasm and stretching of the spine due to cranial nerve function involvement). However, the magnetic resonance report will mostly report pathologies such as cavity infarction to the lesion, so many patients have neck and low back pain symptoms After cervical and lumbar spine surgery, the symptoms not only cannot be relieved, but also more neurological symptoms occur (such as aggravation of limb numbness, spasms, dizziness, headache, depression, anxiety, etc.), so this phenomenon is called secondary nerve injury.

With the unclear diagnosis of the disease and the delay in diagnosis and treatment, many patients will eventually suffer from chronic ischemia and delayed spinal cord nerve redamage, resulting in spastic insufficiency and paralysis. Therefore, the diagnosis and treatment of white matter demyelinating diseases need to consider many aspects, find and estimate the source of the disease, correctly make the microcirculation of the affected nerves get sufficient blood supply, nutrition and excitation activation, and regenerate and repair the affected nerves, so as to essentially alleviate the symptoms and prevent the disease from continuing to deteriorate and achieve the best recovery state.

Demyelination of the white matter is a lesion in which the white matter of the brain undergoes demyelinating changes. The white matter is a part of the nerve fibers in the brain parenchyma, which is wrapped by complex cells to form a myelin sheath, which plays the role of ensuring the efficient transmission of nerve impulses. Age-related demyelination of the white matter is a degenerative disease that does not require special **.

If the white matter is demyelinated due to pathological factors, the following measures can be used:

1. If it is a change in the demyelination of the white matter caused by a nutritional metabolism disorder, such as vitamin B12 deficiency, vitamin B12 can be supplemented to help control the symptoms;

2. If it is a demyelinating change in the white matter caused by ischemia and hypoxia, such as multiple lacunar cerebral infarctions, it is necessary to use drugs to improve local microcirculation, promote blood circulation and eliminate blood stasis, and anticoagulation**;

3. If it is a demyelinating change in the white matter caused by immune-related factors, such as multiple sclerosis, acute disseminated encephalomyelitis, etc., glucocorticoids need to be actively used**, and immunosuppressants can be added after the disease control is stable**.

Regular rest, avoiding staying up late, avoiding overwork, quitting smoking and drinking, improving bad living habits, and controlling high-risk factors for cardiovascular and cerebrovascular diseases have a certain effect on preventing the demyelination of the white matter.

There are many kinds of white matter demyelinating lesions, and they are also very complex, and the clinical manifestations of different ones are different, but pathologically they are all manifested as demyelinating changes in the white matter, which are mostly caused by genetics and autoimmune factors, which are more difficult and require long-term treatment, and the efficacy is very slow and the prognosis is not optimistic, as follows.

1. Drugs, demyelination should be done as soon as possible, from controlling the deterioration of the condition, reducing **, commonly used cortisol drugs, such as prednisone, adrenaline, dexamethasone, methylprednisolone, etc., according to the condition.

2. Avoid triggers, such as trauma, fatigue, agitation, upper respiratory tract infection and other infection symptoms, which can induce aggravation of the condition and should be avoided.

3. In the acute attack period, first of all, bed rest is given to facilitate the alleviation of the condition, and secondly, ** training is given, such as massage when the limbs are twitching, and ** training of the affected limbs is given to prevent joint stiffness and pain.

The above is a response to the question "How to do white matter demyelination changes?" "The advice on this issue, I hope it helps you, I wish you good health!

1. Oral medication**.

Oxiracetam capsule is used in clinical practice for **senile dementia or traumatic brain injury, and can also control the development of patients with white matter demyelination, reduce memory loss and multiple sclerosis and other uncomfortable symptoms. Each patient should take the medication orally two to three times a day according to the doctor's instructions, and the dosage should be reasonably arranged. Patients with renal insufficiency should be cautious with medication, and if this product must be used**, the dose should be reduced.

2. Family**.

The attending doctor will choose the best drug according to the patient's condition, reaction, etc., and should stay away from trauma, fatigue and agitation and other related causes during the onset of the disease, otherwise it will aggravate the condition and is not conducive to the recovery of physical health. It should be noted that pregnancy is very likely to accelerate the deterioration of the condition, so women should avoid pregnancy for two years after the onset of the disease, so as not to affect the health of the fetus and themselves.

3. General**.

Patients with white matter demyelination require bed rest to provide relief. However, for patients with paralysis of the limbs, some passive exercises are needed to prevent symptoms such as joint stiffness and pain. If the patient is in the advanced stage, the family members or medical staff should turn over frequently, strengthen the nursing work, and go to the hospital regularly for re-examination, so as to understand the recovery of the body.

Demyelination of the white matter of the brain, today we use a popular explanation to understand. Simply put, it is an abnormal nerve conduction caused by the detachment of the epidermis that protects the nerves of the brain. In traditional Chinese medicine, it is called perjury, which refers to the delay in the conduction of the myelin sheath.

Just like this wire, the wire inside the broken skin is abnormally convenient, causing poor contact and losing working power.

Early phenomena: The general early phenomena are: dizziness, headache, low immunity such as fever and cold that do not go away for a long time.

Walking is not fast, loss of balance, generally this term is a term for early symptoms that are symptomatic but not obvious, and can lead a normal life. Generally, at this stage, as long as you pay attention to your eating habits, you can completely cooperate with drugs.

Medium-term phenomenon: Medium-term phenomenon means that one or two seizures have been experienced. For example:

Urinary retention, incontinence, hypertonia of the lower limbs, functional impairment of peripheral nerve paralysis, memory loss, blurred strength, and confusion. This kind of just entering the lesion period is often used hormones, immunoglobulin**, this kind of can also be turned into a prognosis instrument, mainly Chinese medicine, the best effect.

Late stage phenomenon: Generally, patients in the advanced stage are very painful, mainly due to mental pressure. For example, coma, loss of consciousness, dementia, paralysis, etc., are often acute lesions caused by long-term lesions or poor physical fitness.

In the face of this situation, it is basically impossible to **, the speed of lesion incursion is disproportionate to the speed of drug absorption, that is to say, the repair speed of the drug cannot keep up with the destruction of the lesion, so it cannot be **.

Demyelinating lesions of the white matter refer to areas of demyelination of different sizes on the inner surface of the central nervous system, which actually include three conditions:

1. Demyelination caused by axonal damage.

2. Defective myelination and inability to complete normal development is the so-called leukodystrophy, which is mostly a genetic defect and is only seen in children.

3. The myelin sheath is damaged and the axonal cord is not damaged or the damage is mild, the pathological changes are divided into acute and chronic, acute lesions are common in the large demyelination necrosis of the white matter under the sebum, there are a large number of lattice cells concentrated near the blood vessels, chronic lesions often show the intact brain structure, the light case is only part of the myelin sheath loss and thinning, and the severe case may be complete loss of multiple occurrences, which is more likely to occur in the central nervous system of young adults. Genetic factors of demyelinating diseases have a certain influence on the occurrence of this disease, and different symptoms appear according to the lesions in different parts.

The method of demyelination of the white matter is as follows:

1. The examinee has no symptoms and no underlying diseases.

2. In patients with more severe degrees, if there are cloud-like large demyelinating changes on both sides of the three ventricles of the cerebral hemisphere, it especially indicates that the patient may have obvious cerebral arteriosclerosis. At this time, a corresponding plan should be formulated according to the comprehensive diagnosis of the scope and degree of the lesion and the underlying disease.

**Hormones, immunosuppressants, immunoglobulins, gamma globulin, etc. can be used. In addition, it is necessary to pay attention to the control of hypertension and the monitoring of blood glucose and blood lipid levels. Quit smoking, limit alcohol, get enough sleep, and exercise appropriately.

The specific medication should be combined with clinical practice, and the doctor's face-to-face guidance shall prevail.

Disease analysis: Mainly to damage the white matter of the brain and spinal cord (neuroleukodegeneration) autonomic nerve caused by autonomic nerve dysfunction, some patients early onset is not obvious, with the neglect and symptoms of the diagnosis after going to the doctor for magnetic resonance and diagnosis, **improper disease** and delayed nerve damage lead to aggravation of the disease, severe onset can invade the anterior horn cells of the spinal cord and brainstem nerve nucleus and cerebral motor cortex pyramidal cells resulting in neurological damage to life. In the early stage, most of them are hormones and nutrition, but the efficacy is difficult to control, due to the loss of myelin caused by the disease, the neurological damage is serious and the body immunity is extremely low, and the neurological symptoms are further aggravated by the occasional infection.

If the lesion is not correct, the lesion will be degenerated late to thin, harden or soften, and re-damage the nerve, secondary dementia or paralysis, secondary complications may be life-threatening. The key to the disease** lies in the early secondary control of the disease.

Suggestions: **Scheme: If the hormone of late-onset demyelinating encephalopathy is ineffective against the disease, the body's immune function should be enhanced, the body's ability to resist disease should be improved, and at the same time, the combination of traditional Chinese and Western medicine should expand the microcirculation so that the damaged residual nerves can be fully supplied with blood, and the disease should be controlled and prevented from continuing to develop.

And the use of nerve regeneration drugs to stimulate the nerves, activate the paralysis and shock nerves, so that the damaged nerves can be regenerated and repaired to achieve early **.

Beijing Deshengmen Hospital of Traditional Chinese Medicine - Wen Jihuan - Deputy Chief Physician - Department of Encephalopathy - What should I do with white matter demyelinating lesions**?

Nowadays, many people use traditional Chinese medicine**.

White matter 158, demyelination 1004**9095

You can find a Chinese medicine doctor**. Now Chinese medicine is an expert in this area.

It is a neurological disease, and the immunity to colds is low.

It is a neuroimmune disease that causes demyelination for many reasons, such as viral infection, trauma, carbon monoxide poisoning, etc.

Demyelination disease will not be well grasped by many people, in fact, the harm caused by this disease problem to the body is relatively large, the common reason is some subacute infectious, multi-nervous system radiculoneuritis, or other viruses ** infection, there are nutritional disorders and their oxygen insufficiency, etc., will cause this disease, so after mastering the cause, then people should also pay attention to prevention in daily life.

Demyelination is a whole class of acquired diseases with different causes and different clinical symptoms, but similar characteristics, and its characteristic clinical manifestations are the loss of myelin of nerve fibers while the relative maintenance of neuronal cells is detailed.

After healing, it is not easy to have seizures, the actual effect is stable, and the symptoms are also treated, from the overall point of view. Careful and meticulous examination and truth-seeking, careful TCM syndrome differentiation and treatment.

White matter is a type of nerve fiber, and human brain tissue can be divided into gray matter and white matter. The myelin sheath is a membrane that wraps around the axons of nerve cells, and its main function is to insulate and protect neurons from transmitting nerve impulses from neuronal axons to another neuronal axon. Detachment of the myelin sheath can be due to poisoning, cerebral ischemia, or cerebral hypoxia.

1. On ischemic demyelination of white matter.

The clinical manifestations of demyelinating diseases vary from patient to patient, but they are all diseases in which the myelin shedding of nerve fibers and single nerve cells remain relatively intact. The insulating and protective effects of neurons are also lost when the myelin sheath is shed, resulting in abnormal transmission of nerve impulses. The myelin sheath of the nerve can be regenerated, but its functional recovery is uncertain, which may or may not be complete.

Ischemic demyelination of the white matter of the brain is mainly a variety of pathological changes that occur by damaging the nerves of the white matter of the brain and making its regulation disordered and unable to regulate itself voluntarily to innervate the tissues of the region. In severe cases, it can cause spastic paralysis of the entire center, which can endanger the patient's life.

1. Pre-diagnosis. Demyelinating changes in the white matter of the brain can be detected and observed by CT or magnetic resonance imaging of the brain. It is necessary to clarify the cause before proceeding to the next step** white matter ischemic demyelination, diabetes, poisoning and hypertension can lead to the occurrence of white matter ischemic demyelination.

2. Symptoms and **. To carry out ischemic demyelination of the white matter, after the cause is clarified, it is necessary not only to have the primary disease, but also to cooperate with nutritional neurodrugs. The early symptoms of most patients are only occasional dizziness and other brain discomfort, and the early **nutrition** and hormones**, but its efficacy is not stable.

As symptoms worsen, the situation becomes more difficult to control, so the sooner the white matter ischemic demyelination** is better, and the early** can achieve the best recovery.

In patients with ischemic demyelination of the white matter, it is important to have a remission of the disease after learning of the disease. Therefore, bed rest should be done appropriately, and the patient's paralyzed limb requires appropriate passive movement to prevent joint stiffness. For advanced bedridden patients, guardians should help them turn over and carry out ** nursing.