Can brain parasitic diseases be cured, a brief introduction to brain parasitic diseases

Brain parasitic diseases can be ** because the parasite infects the human brain, causing brain damage or inflammation in the patient, which is called parasitic disease. Common cerebral parasitic diseases mainly include neurocysticercosis, cerebral schistosomiasis, cerebral pulmonary flukes, cerebral echinococcosis, etc. There are many symptoms of brain parasitic infection, mainly related to the site of the parasite, which can cause headaches of varying degrees and may also cause seizures in patients.

Anti-parasitic drugs, such as praziquantel and albendazole, should be modified from small doses, and patients should be given symptomatic treatment to reduce cerebral edema and anti-epileptic seizures. If surgery or medication is not good, consider surgical removal of the patient's parasites.

The parasite in the brain is capable, but the risk it faces is relatively large, because the parasite is sometimes not a parasite in the skull, it may be a multiple parasite, so it should also be decided according to the situation at that time. Generally speaking, the corresponding drugs are actively used for deworming treatment, and in some cases, surgical resection can also be taken, and the first functional exercise is actively carried out in the later stage.

Brain parasites are capable! It's just that the risk it faces is greater than that of other parts!

Traumatic brain injury, cerebral hemorrhage, cerebral infarction or other diseases in the brain, if the doctor says that cerebral herniation will generally be critically ill, most of them cannot be saved, what kind of disease is this, so serious!

1. What is brain herniation?

When there is a space-occupying lesion in a certain position in the normal brain, the pressure is higher than that of the adjacent tissues, and the brain tissue shifts from the high-pressure area to the low-pressure area, which compresses some brain tissues, nerves and blood vessels, and the cerebrospinal fluid circulation is impaired and the corresponding symptom group is generated, which is called brain herniation.

To put it simply, one part of the brain tissue goes to another place, causing the important brain tissue to be compressed, and the symptoms are fatal.

When the diagnosis is confirmed, the pre-craniotomy preparations should be completed quickly according to the condition, and the surgical removal** should be performed as soon as possible, such as removing the intracranial hematoma or removing the brain tumor.

2. Brain herniation**.

The development of space-occupying lesions in any part of the brain to a certain extent can lead to brain herniation due to uneven pressure in various cavities of the brain. Common lesions that cause brain herniation are:

1. Various intracranial hematomas caused by injury, such as acute epidural hematoma, subdural hematoma, intracerebral hematoma, etc.;

2. Various intracranial tumors, especially tumors located in one cerebral hemisphere and posterior fossa tumors;

3. Intracranial abscess;

4. Intracranial parasitic diseases and other chronic granulomas.

5. Congenital factors, such as cerebellar tonsillar herniation malformation. In addition, in patients with increased intracranial pressure, lumbar puncture releases too much cerebrospinal fluid, resulting in an increased pressure difference between the intracranial cavities, which can promote the formation of brain herniation.

3. Principles of management of brain herniation.

1. Urgently treat the increased intracranial pressure, and quickly infuse hyperosmolar intracranial pressure lowering drugs with intravenous infusion, quickly clarify **, and make preoperative preparations for craniotomy at the same time.

2. Different surgical decompression methods are adopted according to the ** and the patient's condition.

3. If the occipital foramen magnum hernia or severe hydrocephalus is combined, rapid lateral ventricular extracorporeal drainage can often quickly reduce the intracranial pressure and turn the patient from critical to safe.

Can brain parasitic diseases be a**? Mine is that the current medical development of our country is very good, any disease can be **, brain parasitic disease, I believe it can also be **.

1 Pinyin 2 Introduction.

3 Clinical manifestations.

nǎo jì shēng chóng bìng

Common pediatric brain parasitic diseases are: Cerebral schistosomiasis: caused by the invasion of schistosomiasis eggs into the brain.

The pathological manifestations are meningeal thickening, adhesions, and exudates. Miliary nodules made of eggs are seen in the brain, which are gray and hard in color. Multiple small nodules agglutinate together to form a large granuloma mass, and the surrounding brain tissue is significantly edematous.

Neurocysticercosis: cysts parasitic in the brain, mostly found in the cerebral cortex near the motor center, leptomeninges, cerebral white matter and ventricles can also exist, the size and number of cysts are different. In the surrounding tissues of cyst parasitism, there is hyperemia, edema, and inflammatory cell infiltrate.

Cerebral fluke: It is an adult parasite in the lungs, metastasizes into the skull, and lays eggs to form a multilocular cyst with eggs, pus cells, and needle-like crystals, and the cyst wall is composed of glial hyperplasia of surrounding brain tissue, and early lesions mostly occur in the temporoparietal lobe. Cerebral echinococcosis:

Echinococcus invades the brain and is distributed in the brain along with the middle cerebral artery, forming hydatid cysts of varying sizes, and can also have many ascomycetes in a mother sac. There is no adhesion with the surrounding tissues, the lesions are mostly seen in the frontal and parietal regions, the cerebellum, ventricles and the base of the skull are less, the hydatid cysts are white translucent, the volume is different, and there are cyst fluid and scolices in the sac.

The clinical manifestations of cerebral parasitic diseases share common characteristics. In the acute stage, there are many functional disorders such as headache, dizziness, insomnia, irritability, apathy, memory loss, convulsions, and vomiting. After the acute stage, symptoms of increased intracranial pressure, optic nerve edema in the fundus, and symptoms of brain damage such as generalized or local convulsions, facial paralysis, tongue protrusion, hemiplegia, and aphasia.

Differential diagnosis should be based on residence in endemic areas and history of infection. history of schistosomiasis infection with cerebral schistosomiasis; Neurocysticercosis has subcutaneous cystic nodules; Cerebral fluke disease with a history of eating raw crabs or crickets; Cerebral echinococcosis has a history of close contact with animals (dogs, sheep). Laboratory tests showed eosinophilia.

Stool examination for cerebral schistosomiasis may show schistosomiasis eggs; Eggs can be found in the feces of neurocysticercosis; Eosinophilia and protein content in cerebrospinal fluid in cerebral echinococcosis are increased. Chest x-ray of cerebropulmonary fluke disease has a shadow of a pulmonary nodular cyst, and a positive complement fixation test can be used as a diagnostic reference.

Neoplastic lesions of intracerebral parasites are indicative of surgery**. If the intracranial pressure is too high, the optic nerve ** edema in the fundus is severe, and there is no obvious lesion on ventriculography, subtemporalis decompression can be performed to protect vision, and postoperative deworming is adjuvanted**.

Go to a big hospital for a consultation, it's complicated, and you can't just ask for a doctor.

Hello, you said that this situation is considered to be a neurocyst.

1. Cysticercosis. Commonly used medications are:

1 Praziquantel: It is a broad-spectrum antihelminth drug, which also has a good ** effect on cysts. The usual dose is 120 mg kg, taken orally over 6 days (three times a day).

Taking the drug cyst can cause swelling, degeneration and necrosis, resulting in inflammatory reaction and allergic reaction in the brain tissue around the cyst, and some patients can also have cerebral edema of varying degrees, increased cerebrospinal fluid pressure and cell number, and even intracranial hypertension crisis in severe cases.

2 Prothhimazole, also a broad-spectrum antihelminthic drug. The usual dose is 15-20 mg kg per day for 10 days. Common side effects include itching, hives, dizziness, fever, seizures, and increased intracranial pressure.

3 Mebendazole: The commonly used dose is 100mg, 3 times a day, for three consecutive days, and the common side effects are abdominal pain, diarrhea, itching and headache.

In order to reduce the allergic reaction caused by a large number of deaths in the body in the process of anticyst **, generally start with a small dose and gradually increase the dose, such as praziquantel from 100mg first, 3 times a day, if there is no adverse reaction, increase by 100mg each time, until the ** dose is reached, and then continue to use for 6 days and then stop use. Dehydration drugs such as mannitol** should be used promptly after symptoms of increased intracranial pressure, and steroids should be used as appropriate. If severe intracranial hypertrophy occurs, in addition to stopping anticystic drugs and dehydration and anti-allergic treatment, subtemporal muscle decompression can also be used to prevent intracranial hypertension crisis.

2. Surgery**: Patients diagnosed with ventricular type should be surged**. Second, surgery may also be considered in patients with persistently elevated intracranial pressure and limited neurologic signs and CT-confirmed lesions**.

3. Tapeworm repellent**: For those who still have tapeworm parasites in the intestine, in order to prevent their own reinfection, tapeworm repellent** should be performed. The commonly used drug is chlorsulfamine (niclosamide), 2 grams, chewed and swallowed once, and laxative should be given once 3-4 hours after taking the drug to expel proglottids and eggs.