How to check for cerebral thrombosis and solve for further examination of cerebral embolism

Netizen consultation: Patient is a 43-year-old male. Dizziness, nausea caused vomiting several times.

Test result: slight cerebral thrombosis. Wonder?

How is it**? Experts from Shanghai Blue Cross Brain Hospital answered: ? 1. Symptom examination:

Before the onset of the disease, the patient had signs of limb numbness, ineffective movement, slurred speech, dizziness, and blurred vision. It often occurs during sleep or in the morning, with weakness or inability to move the affected limb, slurred speech or aphasia, and choking on water. Most patients are unconscious or mildly impaired.

facial and hypoglossal nerve palsies, nystagmus, decreased or increased muscle tone and abdominal reflexes, positive pathological reflexes, decreased or absent abdominal wall and cremasteric reflexes. 2. Physical and chemical examination: 1. Electroencephalogram:

The two sides are asymmetrical, and the lesion side is slow, low amplitude, and slow rhythm. 2. Cerebral angiography: showing arterial stenosis, occlusion and abnormal blood vessels around the lesion.

3. Brain ultrasound: 24 hours after the disease, the midline wave can be seen to be displaced to the opposite side. 4. CT scan:

There is a low-density area with reduced absorption in the vascular distribution area at the infarct site. 5. Hemorheology: the viscosity of whole blood increases, the platelet aggregation increases, and the length of thrombus in vitro increases.

Hospital features**: Stroke of integrated traditional Chinese and Western medicine**Six steps First, wake up the brain and open the mind: suitable for cerebral hemorrhage, large-scale cerebral infarction, trauma and other serious head injury, resulting in the patient coma, delirium, or vegetative state, etc., the method to lower intracranial pressure, promote brain cell function**, so that the nerve cells in a state of shock as soon as possible to restore function, as well as prevent complications and other comprehensive**.

2. Hematoma fragmentation and aspiration: suitable for patients with cerebral hemorrhage, subdural hematoma, cerebral contusion and laceration intracranial hematoma, using a special hematoma grinder, with a diameter of about 3mm, to carry out all-round scouring, crushing, separation and drainage of the hematoma without craniotomy and incomplete anesthesia, and will not bring any minimally invasive hematoma hematoma to the brain. 3. Thrombolysis method:

It is suitable for the acute and subacute stages of cerebral infarction, especially for patients with cerebral infarction and cerebral infarction (except for large-scale cerebral infarction) and advanced cerebral infarction. On the one hand, it can dissolve thrombosis, dredge blood vessels, and restore the recovery of brain cells in the ischemic area, on the other hand, it can also dissolve, remove, or make the wall smaller, which has a good effect on preventing cerebral infarction. Fourth, the limb ** method:

It has a significant recovery effect on the patient's limbs. Severe patients or patients with paralysis within half a year can basically take care of their limbs, and patients in the acute and subacute stages can basically recover their paralyzed limbs, including Western medicine, Chinese medicine, acupuncture, electroacupuncture, medicinal oxygen, limb function training and equipment training. 5. Acupoint injection method:

It is suitable for any patient with facial and limb function injury, and the application of different drugs, different acupuncture points and different techniques can play a good role in the recovery of the injured nerves. Tips: The examination of cerebral thrombosis must go to a professional and regular hospital, and do not believe in small roadside advertisements.

Shanghai Blue Cross Brain Hospital has a good reputation in the field of cerebral thrombosis and is a trustworthy hospital.

Answer] :(1) Electrocardiogram examination can find evidence of myocardial infarction, arrhythmia, and myocardial ischemia, which is suggestive for cerebral embolism of cardiac socks.

2) Cardiac ultrasound can confirm the existence of cardiac emboli, which is suggestive of cardiogenic cerebral embolism.

3) Carotid artery ultrasonography can evaluate carotid artery lumen stenosis, blood flow and carotid plaque, which is of suggestive significance for carotid artery source brute cerebral embolism.

4) CT or MRI of the brain can show changes in ischemic well-bridging infarcts or hemorrhagic infarcts, the latter of which is more supportive of the diagnosis of cerebral embolism.

？Early** cerebral thrombosis can significantly improve the sequelae of patients and reduce the damage caused by the disease to patients

Complete blood count. In the early stage of cerebral thrombosis, there are generally no special changes. The white blood cells are not high, which is different from a brain hemorrhage.

When there is a co-infection, such as a respiratory or urinary tract infection, white blood cells are elevated. Urinalysis. Cerebral thrombosis has no effect on urinalysis.

When patients have arteriolar sclerosis in common, urinalysis may be abnormal. Male patients with chronic prostatitis may have pusular and protein in the urine. Urine glucose tests may detect previously asymptomatic diabetic patients.

Stool routine. There is generally no change, and the purpose of examination of stool in patients with cerebral thrombosis is to detect parasites and other intestinal diseases. Fecal occult blood to detect bleeding from the gastrointestinal tract.

2) Blood biochemical tests: blood glucose measurement and glucose tolerance test: ischemic cerebrovascular disease often has low glucose tolerance during transient ischemic attack, acute phase and recovery period, and persists.

In cerebrovascular patients with low glucose tolerance, coagulation factor VIII-related antigens, fibrinogen, and triglycerides were significantly higher than normal, and blood hypercoagulability was one of the causes of cerebrovascular disease. Lipid measurement: triglycerides and high-density lipoprotein (HDL) have an inverse relationship, i.e., HDL decreases when triglycerides are high.

Decreased HDL predisposes to atherosclerosis, causing ischemic cerebrovascular disease. Serum potassium, sodium, chloride, and carbon dioxide binding tests: timely detection of electrolyte imbalances and acid-base imbalances.

Through the above introduction of relevant examinations for patients with cerebral thrombosis, I hope to increase your understanding of cerebral thrombosis and choose a scientific and effective method.

The incidence of cerebrovascular diseases is getting higher and higher, and the current medical conditions can be understood through CT, MRI, and related angiography examinations in the brain, and can also be eliminated through relevant blood biochemical examinations such as blood pressure, blood sugar, blood lipids, etc.

For patients with cerebral thrombosis, hypertension, obesity and diabetes are key factors leading to cerebral thrombosis. In the early stage, dizziness, dizziness, nausea and vomiting, nausea and retching, blackness in front of the eyes, inability to see, numbness of the upper limbs, stroke, facial paralysis, aphasia and other diseases will occur. If the above symptoms are found, it is recommended to go directly to the hospital for brain and blood routine tests and electrocardiogram examinations to see if it can cause cerebral thrombosis.

Early ** can alleviate the development trend of the disease, control the disease, and improve the ** rate.

Cerebral thrombosis is manifested as dizziness, weakness of limbs, weakness of the whole body, stroke, and even fainting, which can be significantly life-threatening. This type of disease often appears in the morning, when the patient is suddenly unable to speak, the human body is unable to exercise or the language expression is not very good. There are also specific manifestations of stroke and facial paralysis, and if this kind of thing occurs, it is necessary to go to the emergency department as soon as possible, grasp the opportunity for medical treatment, make early diagnosis, and do medical treatment as soon as possible, without delaying the symptoms.

Immediate diagnosis should be confirmed by medical attention, defensive intravenous thrombolysis should be performed under the guidance of a doctor, and lifestyle modifications should be made in addition to <>

To prevent cerebral infarction, we should actively control the body, quit smoking and alcohol to avoid risks, avoid the intake of high-sugar, high-calorie, high-calorie foods, high-fat, and trans-fatty acid-containing foods in the diet, and improve aerobic exercise with moderate tensile strength**, such as brisk walking, regular work and rest, and ensure adequate sleep and rest. Cerebral thrombosis is a type of cerebral infarction, which is a series of conditions caused by arterial narrowing and thrombosis caused by myocardial infarction at the top of the head, resulting in blockage of blood supply to the brain.

Cerebral thrombosis often vacates more serious symptoms, so immediate ** is critical for the recovery period. Nutritional adjustments. Three meals are regular and quantitative, diet is matched, it is not suitable to be too supportive, avoid overeating, pay attention to control fat and caloric value of ingredients, choose ingredients that can avoid cerebral thrombosis, and eat more fruits and vegetables rich in dietary fiber, coarse grains, etc.

Have a regular schedule. Disordered work and rest schedule can easily lead to metabolic disorders and accelerate thrombosis, so we should pay attention to regular work and rest, avoid sleeping after meals, avoid reduced blood supply to the brain, slow blood flow, and accelerate thrombosis.

Headache, dizziness, tinnitus, hemiplegia, can be single limb or single limb, or the upper limb is heavier than the lower limb or the lower limb is heavier than the upper limb, and there are a variety of conditions such as dysphagia, slurred speech, nausea, vomiting, etc. Coma. Each patient may have several of these clinical manifestations.

These are common symptoms of cerebral infarction. To determine whether there is cerebral thrombosis, you can choose to observe the symptoms at usual times, or you can choose a specialty**. Judging by combining these two methods, the effect is more obvious.

Once the onset of thrombosis patients will have symptoms such as weakness in the limbs, headache, numbness of the tongue, speech disorder, drooling, etc., so it is very important to have a physical health examination once a year, so that the condition can be detected early. If the above symptoms appear, you must pay attention to it, check it in time, and avoid blood clots.

Cerebral thrombosis. It is mainly because of the blood disorder in the brain, which leads to cerebral ischemia, hypoxia, and avascular necrosis of brain tissue.

Cerebral thrombosis and diabetes.

Hypertension. Rheumatic heart disease.

Long-term obesity, arrhythmia, trauma, shock, smoking, fatigue, alcohol consumption, low blood pressure, etc. are involved. Long-term and poor control of the above triggers can easily aggravate atherosclerosis, resulting in vascular circulation disorders and ischemia and hypoxia.

The clinical symptoms of cerebral thrombosis vary, mild can cause dizziness, headache, nausea, and vomiting; In more severe cases, there may be limb inactivity, speech disorders, incontinence and even limb paralysis, coma, death, etc. Generally, the diagnosis is confirmed by blood pressure, blood routine, macrochemistry, electrocardiogram, cranial CT or MRI.

How to get cerebral thrombosis Most cerebral thrombosis is due to the presence of lesions in the cerebral blood vessels themselves, such as atherosclerosis.

Changes in blood composition are also factors in cerebral thrombosis.

Atherosclerosis is the most common lesion of the cerebral artery wall and the first cause of thrombosis**;

Secondly, various cerebral arteritis, as well as congenital vascular dysplasia and vascular amyloidosis are also common factors causing vascular wall lesions.

The CT findings of intracranial hematoma are mainly images of the hematoma itself, peripheral brain changes, and mass findings. Fresh hematoma is a high-density area with clear and uniform density in the brain, with a CT value of about 60 80 hu. The high-density hematoma is surrounded by a low-density edema zone, and there are also mass manifestations such as compression of the ventricles, cerebral cisterns, and sulci and midline structural displacement caused by hematoma and cerebral edema.

The CT findings of intracranial hematoma at different stages were consistent with the pathological process of hematoma formation, resorption and cystic changes.