Can high blood pressure cause kidney disease, and can high urea nitrogen cure it? How to treat it?

There are two types of elevated urea nitrogen. The most common is renal insufficiency, including acute renal insufficiency and chronic renal failure, which is mainly renal impairment and urea nitrogen is not well excreted in the urine. The use of traditional Chinese medicine is to block the progression of renal fibrosis, fundamentally repair the damaged kidney function, dilate the renal artery, increase the effective blood perfusion of the damaged kidney, increase the oxygen supply to the kidney, improve the renal microcirculation, reduce the intraglomerular pressure, and promote the repair of the glomeruli, the structural change and functional recovery of the damaged lesion.

This increases urine output, excreates blood creatinine and urea nitrogen from the body, and relieves the symptoms of poisoning. Hello, high blood pressure for a long time can cause hypertensive nephropathy, which causes renal insufficiency, which is manifested as an increase in creatinine. The diagnosis of hypertensive nephropathy is like this:

The history of hypertension is often many years, and there is no kidney disease when hypertension is found, and kidney disease is not discovered until several years later, and usually manifests as tubular damage. Suggestions: If it is clear that kidney disease is caused by high blood pressure, then the focus is to control high blood pressure, and then relatively apply some drugs to protect the renal tubules, such as Jinshuibao capsules, Bailing capsules, etc., even if it is a slow process to recover.

Urea nitrogen is basically determined by the balance of urea production and excretion, so it is closely related to protein uptake, protein metabolism, and kidney function. Therefore, if this value exceeds 50mg dl, it is probably kidney failure. Possible diseases when urea nitrogen shows high values:

Disorders of urea nitrogen excretion cause kidney failure (low function of the renal globules), dehydration, edema, obliterative urinary tract disease, ingestion of high-protein foods due to excessive urea nitrogen production, infections, cancer, diabetes, hyperthyroidism, surgery, and gastrointestinal bleeding. In the early stage of kidney disease, lowering serum creatinine and urea nitrogen is the most important means to prevent renal insufficiency. View the original post

A slight increase in elevation is generally not of much clinical significance, and a combination of clinical symptoms and other tests is required for diagnosis.

Clinical significance of elevation:

1. Organic renal damage: chronic renal failure caused by various primary glomerulonephritis, pyelonephritis, interstitial nephritis, renal tumor, polycystic kidney disease, etc. In acute renal failure, there is no change in BUN when renal function is mildly impaired, but the GFR (glomerular filtration rate) falls below 50% for BUN to be elevated.

Therefore, blood BUN measurement cannot be used as an early indicator of renal function. However, the degree of elevation in chronic renal failure, especially uremia, is generally consistent with the severity of the disease: the GFR in the compensated phase of renal failure decreases to 50 ml min, and the blood bun is 9 mmol L; decompensated renal failure, blood bun 9mmol L; In the stage of renal failure, the blood bun is 20mmol l.

2. Prerenal oliguria: such as severe dehydration, massive ascites, cardiac circulatory failure, hepatorenal syndrome, etc., resulting in hypovolemia, reduced renal blood flow, and insufficient perfusion resulting in oliguria. At this time, the bun is elevated, but the creatinine is not significantly elevated, and bun cr(mg dl) 10:

1, called prerenal azotemia. Urine output can increase after volume expansion, and BUN can decrease spontaneously.

3. Excessive protein decomposition or intake, such as acute infectious diseases, high fever, upper gastrointestinal bleeding, extensive burns, severe trauma, post-major surgery and hyperthyroidism, high-protein diet, etc., but blood creatinine generally does not rise. After the above conditions are corrected, the blood bun can be reduced.

Whether or not to reach the uremia stage is mainly based on the blood creatinine and the calculation of creatinine clearance.

Attached: 1. Calculation of creatinine clearance.

Creatinine clearance CCR = [(140-age) body weight (kg)] serum creatinine (for females, as calculated

2. Stages of chronic renal insufficiency.

Normal renal function: creatinine clearance CCR 120 80 ml Partial renal insufficiency compensatory phase: creatinine clearance CCR 80 50 ml Partial azotemia phase:

Creatinine clearance CCR 50 20 ml Divided into renal failure phase: creatinine clearance CCR 20 10 ml Divided into uremic phase: creatinine clearance CCR 10 ml min.

If there are no other underlying diseases, high urea nitrogen alone is basically meaningless.

If there are no other underlying diseases, it is meaningless to simply have a high creatinine, and if there are no other problems, the creatinine clearance rate is less than 60 to determine chronic kidney disease.

Is creatinine normal??? It may be caused by your regular consumption of melon seeds, peanuts and other nuts.