Why do patients with cerebral infarction always say that they are tired and do not sleep at night???

According to what you said, it is a sequelae of stroke, and some patients can suffer from syndrome, ** the disease must have a long-term psychological preparation. The sequelae phase is a period when there is no further recovery in the motor function of the paralyzed limb. After active training, it generally enters the sequelae period 6 months after the onset of the disease.

Some patients still have symptoms to varying degrees after active **** in the early stage and recovery period, such as abnormal posture, inability to speak, and even difficulty in sitting and standing. In the sequelae stage, the patient's functional recovery is impossible or incomplete, and the ** for this part of the patient is mainly for psychological and emotional, that is, the patient's family must face the reality and give the patient spiritual care. For those who are bedridden with little activity or for a long time in the early stage, the recovery of motor function can last longer, and exercise tolerance and activities of daily living can still be further improved.

Due to the damage of the nerves in the brain of patients with cerebral infarction, the reaction is slow, the movement is slow, and the lack of energy is caused by sleeping more, and the tiredness is certain, because there is always a lack of oxygen in the brain, so they will feel tired.

Since he can't move, the side that can't move is of course tired, and he was right to say tired. Not sleeping at night is because his consciousness has been rested during the day, and it is normal not to sleep, so there is no need to worry.

Cerebral infarction refers to a series of symptoms caused by cerebral vascular blockage, after the blockage, the brain tissue is ischemia and hypoxia, energy is insufficient, and of course you will always want to sleep.

Condition analysis: cerebral infarction is due to cerebral atherosclerosis, vascular intimal damage narrows the lumen of the cerebral artery, and then local thrombosis is formed due to a variety of factors, which aggravates or completely occludes the arterial stenosis, resulting in ischemia and hypoxia buried in the slag of brain tissue. **Mind Brain Dredging Capsule Captopril Aspirin mannitol.

Guiding opinions: (1) Instruct patients to stay in bed in the acute stage, preferably in a supine position, to ensure cerebral blood supply and reduce brain ischemia.

2) Maintain the functional position of the paralyzed limb and help the patient to do the passive movement of the affected limb and joint.

3) Cause the current symptoms, only by restoring the brain nerve function, the condition will be significantly improved, after the condition is stable, encourage the patient to do active exercise. Get out of bed as soon as possible, train and guide from getting up, balancing the affected limb, standing, and walking, gradually increase the range and number of activities, and finally help to train up and down the stairs, so that the affected limb can be exercised and conducive to the recovery of function.

In addition to the appropriate **, you can also eat Dilong protein-related foods for conditioning improvement. Dilong protein has the ability to improve microcirculation, dissolve blood clots, soften blood vessels, reduce blood viscosity, regulate blood pressure in both directions, and lower blood sugar.

Problem Analysis:

Cerebral infarction is due to cerebral arteriosclerosis caused by cerebral ischemia and hypoxia of brain cells, resulting in brain cell necrosis, and the appearance of limb dysfunction, unfavorable speech, unconsciousness and other symptoms, the early **thrombolysis is the mainstay, the mid-stage ** mainly focuses on blood circulation and stasis, and promotes brain cell metabolism. After discharge, take brain rehabilitation and aspirin**.

Suggestions: Hello; **It depends on the condition of the acute stage of large-scale cerebral infarction**The key is to control intracranial pressure, reduce cerebral edema, prevent the formation of cerebral herniation, and promote the recovery of diseased brain tissue function. Hyperosmolar dehydrators, diuretics, and hormones can be given promptly**. Brain cell activators may also be given as appropriate.

The application time of dehydrating agent should depend on the condition, generally after 1 2 weeks**, if the patient's consciousness disorder disappears and the intracranial pressure has returned to normal, vasodilators and blood circulation and blood stasis removal drugs can be given. However, it should be mentioned here that vasodilators and blood stasis drugs must be used at the right time, and cannot be used blindly or too early, otherwise, there will be "blood steal syndrome" and aggravate the condition. At the same time, attention should be paid to controlling blood pressure, maintaining water and electrolyte balance, preventing and **complications**.