Meniere s syndrome, what else do you know

Meniere's syndrome is characterized by sudden vertigo, visual rotation, severe vomiting, inability to move, tinnitus, deafness or nystagmus, with episodic and aspirative characteristics, that is, vertigo has obvious episodes and intermittent periods. Most of the patients are middle-aged, and there is no significant difference in the gender of the patients, with about 65% of patients having a first episode before the age of 50, and most patients have monocular disease.

Vertigo and tinnitus warn of Meniere: As the saying goes, "The patient is dizzy, the doctor is dizzy".

Vertigo is one of the common clinical symptoms, and the diagnosis and treatment of vertigo are extremely disordered because the severity of vertigo is often difficult to determine. Yes, the inner ear is the sensory organ for hearing and balance, and more than 96% of Meniere patients have episodes of vertigo or dizziness. This vertigo lasts from 1 hour to 2 days, from severe to mild, and occurs irregularly, about once or several times a year.

After an attack, patients often feel very tired, fall asleep quickly, and wake up feeling better. Generally, patients suffering from Meniere's syndrome can be conditioned according to ** with traditional Chinese medicine, if there are patients with vertigo, because vertigo is mainly related to the five internal organs, kidney yin is insufficient, and spleen yang is weak, which will cause vertigo, and patients with vertigo will be weaker, and weakness will be supplemented, so traditional Chinese medicine **, for vertigo in Meniere's syndrome has a better curative effect.

No Love Disease is an inner ear disease named after a French doctor, Prosper Meniere. The main symptoms of this disease are regurgitative vertigo, hearing loss, tinnitus, and ear stuffiness. In 1861, Dr. Meniere published an article suggesting that the inner ear has the function of regulating balance in addition to hearing.

This view was not taken seriously at the time due to the limitations of medical anatomy, and it was not until the 20th century that it was gradually verified in medicine. By 1989, China's Natural Science Terminology Examination and Approval Committee named the disease Meniere's disease according to the French pronunciation. At present, it is generally believed that Meniere's disease is caused by hydropsatophylaxis in the inner earmembrane, which leads to hydrops.

Electrophysiology of hearing is the most important in Meniere's examination, and CT and magnetic resonance imaging are also required to rule out vertigo due to intracranial tumors in pure tone audiometry, glycerol testing, electrocochleography, and vestibular function testing. It is recommended to go to a specialized ENT hospital for a detailed examination.

A confirmed case of Meniere's syndrome, typically characterized by paroxysmal vertigo and hearing loss, accompanied by tinnitus or a feeling of tightness in the ears. Vertigo is characterized by sudden rotation, lasting from about 20 minutes to 12 hours. In severe cases, it may be accompanied by autonomic symptoms, such as nausea, vomiting, paleness, sweating, etc.

Hearing loss is characterized by fluctuating hearing loss, which is mainly low-frequency type at the beginning, and with the reversal of the disease, it will develop into irreversible high-frequency sensorineural hearing loss, and there will be persistent tinnitus or ear stuffiness symptoms during the intermittent period. Diagnostic criteria for Meniere's syndrome require at least two episodes of vertigo and audiologic findings for sensorineural hearing loss.

1. Genetic factors.

If more than one person in the family has had Meniere's disease, the other people are much more likely to develop the disease than the general population, and the way the disease is inherited is very diverse. In order to reduce the chance of developing the disease, people with genetic risk need to strengthen their physical protection.

2. Viral infection.

Viral infections are a common factor in the development of Meniere's disease. If a patient's endolymphatic vessels and endolymphatic sac are accidentally infected and destroyed by the disease-causing virus, the risk of developing the disease is significantly increased. Once this factor appears, what patients need to do is to actively seek medical attention to fight the infection**, so that the disease can be curbed more quickly.

3. Endolymphatic sac dysfunction.

After the occurrence of endolymphatic sac dysfunction, it is easy to cause abnormal glycoprotein secretion or production, which leads to problems in the internal environment of endolymphtic stability, and Meniere's disease may occur at this time. Moreover, this factor is difficult to prevent, so after the occurrence of such a trigger, it is necessary to deal with it according to the guidance of a professional doctor as soon as possible.

The above are some of the causes of Meniere's disease, and it is recommended that patients actively prevent common adverse triggers in their daily lives. If it is not prevented, the symptoms have already appeared. Patients must seek medical attention in time and choose the best measures according to the specific state of the condition.

After the illness**, patients should rest more, maintain good eating habits and a relaxed emotional state. At the same time, it is also necessary to exercise appropriately and strengthen physical fitness, which is conducive to the recovery of the condition.

The condition of Meniere's syndrome is not well understood, and it may be related to various infectious factors, injuries, otosclerosis, syphilis, genetic factors, allergies, tumors, leukemia, and autoimmune diseases.

Meniere's disease (formerly known as Meniere's syndrome) is an unexplained inner ear disease characterized by hydrops, memranous labyrinthus, and is clinically characterized by episodic vertigo, fluctuating hearing loss, tinnitus, and/or aural fullness.

Attacks are often accompanied by pallor, cold sweats, nausea, vomiting, and intermittent periods of dizziness. Hearing loss occurs during seizures, while hearing may be partially or fully restored during intermittent periods. As the disease progresses, hearing loss no longer fluctuates and gradually worsens.

Tinnitus usually appears before the onset of vertigo, worsens during vertigo, and disappears with the relief of vertigo during the intermittent period, but the tinnitus in patients with reverse action can persist. The reverses of Meniere's disease have a serious impact on the quality of life of patients.

The typical symptoms of Meniere's disease are vertigo, deafness, tinnitus, and a feeling of fullness in the ears, as follows:

1. Vertigo: mainly rotational vertigo, the duration of the attack is about 20 minutes to 12 hours, accompanied by nausea, vomiting, sweating, tinnitus, ear fullness, urinary urgency, etc., and tinnitus and ear fullness can persist in the later stage of Meniere's disease;

2. Hearing loss: hearing loss is fluctuating, hearing loss is obvious when vertigo occurs, hearing can improve after vertigo is relieved, and hearing damage is irreversible in the later stage of Meniere's disease.

Hello, the general clinical manifestations are reverse rotational vertigo, fluctuating hearing loss, tinnitus and ear fullness may occur, severe is vomiting, it is recommended to go to the hospital for detailed examination, it is best to be hospitalized. Avoid leaving sequelae. Regular check-ups are preferred.

Thank you, hope it helps! 1

Meniere's disease, also known as Meniere's syndrome, is an inner ear disease characterized by episodic vertigo, fluctuating deafness and tinnitus caused by edema of the inner ear membrane. It is usually monocral, and it is more common in young adults. **Unknown, may be related to congenital inner ear anomalies, autonomic dysfunction, viral infections, allergies, endocrine disorders, salt and water metabolism disorders, etc.

At present, it is generally believed that endolymphtic return obstruction or malabsorption is the main cause, and the main clinical manifestations are episodic vertigo, fluctuating deafness, and tinnitus. Diagnosis and treatment guidance: 1. To determine whether it is Meniere's disease, it is necessary to pay attention to sudden deafness, labyrinthitis, oto-drug poisoning, vestibular neuronitis, acoustic neuroma, positional vertigo, sudden deafness, vertebrobasilar artery insufficiency, cardiovascular disease and hunt syndrome.

2. Vestibular function examination, hearing examination, glycerol test, and electrocochlear electrogram examination can be performed3. 1. During the seizure period, bed rest, low-salt diet, and less water should be drunk. 2. Sedatives: Valium can be used during the attack period to inhibit dizziness and vomiting.

3 Vasodilator drugs. 4. Vitamin drugs: vitamin B1, vitamin B6, vitamin E, etc.

5 Diuretics. 6. Surgery**: For those who have frequent attacks, conservative attacks**, and vertigo and relocation affecting work and life, surgery can be considered**.

Including: endolymphatomy, balloontomy, labyrinthectomy, transcranial fossa or translabyrinthine vestibular neuroresection, etc.

The main clinical manifestations of Meniere's disease are: vertigo is a sudden onset, mostly rotating or shaking, accompanied by spontaneous nystagmus and autonomic reflex symptoms such as nausea, vomiting, paleness, cold sweats, slow pulse, and decreased blood pressure. Vertigo lasts for a short period of time, usually lasting a few seconds to a few minutes, and rarely exceeds a day.

Tinnitus usually precedes the onset of vertigo, starting with low-pitched windy tinnitus and then turning to high-pitched cicada-like tinnitus. Tinnitus is severe during vertigo episodes and mild during intermittent periods. Hearing loss is mostly fluctuating, with a feeling of fullness in the affected ear before the attack, hearing loss during the attack, and hearing recovery or incomplete recovery after the attack.

The overall trend of hearing loss is worse with the number of seizures, but total deafness is rare. In addition, some patients experience a feeling of fullness, heaviness, or pressure in the affected head or ear during the attack. Otoscopy showed that the tympanic membrane was normal, the Eustachian tube was functioning well, the hearing impairment was cochlear (positive hearing test, negative fatigue test, etc.), and the low vocal range damage was severe, and the deafness was mostly sensoriac.

Vestibular function is tested between episodes and is mostly semicircular canal paralysis on the affected side; Glycerol tests are mostly positive. The combination of the above symptoms and examination data can be of diagnostic value for this disease.