Does the crescent moon speed up kidney failure, and what does the crescent mean?

This depends on the pathological results of the crescent type, size and number.

It is generally believed that cellular crescent + large crescent (occupying more than 50% of the full capsule) + extensive crescent formation (more than 50% of glomeruli have crescents) - indicates active lesions and will accelerate the deterioration of renal function.

Crescent is a pathological concept and one of the basic lesions of glomerular morphology. The appearance of crescent often indicates that the patient's renal pathological damage is relatively severe, which is an important sign of poor prognosis of IgA nephropathy. The definition of a crescent is:

The Baumann sac is filled with capillary leakage of blood cells, cellulose, infiltrating monocytes, and proliferating parietal epithelial cells, forming a hemispherical structure on the outside of the capillary bulb.

What is crescent-type IgA nephritis.

Crescent formation occurs when the glomerular capillary wall is severely damaged and fractured due to various causes, and if the glomeruli formed by the crescent in the Baumann sac accounts for more than 50 of the total glomeruli, and the crescent occupies more than 50 of the glomerular area, it is called crescent nephritis (CGN). If this crescent occurs in a patient with IgA nephropathy, it can be called IgA crescent nephritis (this is just a non-strict name).

Crescents are often found in many other glomerular lesions, and their formation is often indicative of severe disease and a poor prognosis.

The appearance of crescents in different diseases is also different.

Foreign scholars analyzed the pathology of 6,000 cases of kidney biopsy and found that: 95 patients with anti-glomerular basement membrane (anti-GBM) antibody nephritis had crescent formation; Antineutrophil cytoplasmic antibody (ANCA)-associated glomerulonephritis with crescents accounts for about 90 patients; The percentage of crescent lesions in immune complex crescent nephritis is smaller than in the first two, and the disease progresses slowly. The prognosis of IgA nephropathy or lupus nephritis is directly related to the percentage of crescent formation.

However, the prognosis is not always the same for different disorders with similar crescent numbers. For example, a 25-crescent group with lupus nephritis has a worse prognosis than a streptococcal infection with the same percentage of crescents.

Crescents appear in IgA nephropathy and have a poor prognosis.

Crescent formation has a non-negligible negative role in the pathological damage of the kidneys in IgA nephropathy, mostly segmental, cellular crescents, of which mononuclear macrophage infiltration is common, crescents are more common in patients with severe diffuse mesangial hyperplasia and/or diffuse intracapillary hyperplasia, found by renal biopsy in IgA nephropathy: in the five stages of IgA nephropathy, crescent formation occurs from the second stage, and it is generally believed that the later the stage, the greater the number of crescents, The more severe kidney damage due to crescents, is also an important pathological factor in the poor prognosis of IgA nephropathy.

This is usually the result of renal puncture The crescent is a pathological concept that is one of the basic lesions of glomerular morphology. The appearance of crescents often indicates that the patient's kidney pathological damage is relatively severe The formation of crescents is seen in severe damage and rupture of glomerular capillary walls caused by various causes.

Crescent nephritis, also known as rapidly progressive nephritis. The disease has a rapid onset, rapid decline in renal function, poor prognosis for patients, and more than 80 patients enter renal failure within 3 months and require hemodialysis**; The mortality rate is also high.

1. General** includes treatment for hypertension, water and sodium retention, acidosis, electrolyte disorders, uremia and infection, cardiac insufficiency, pericarditis, etc. The approach and management are similar to those of general renal insufficiency. 2. Western medicine** 1 corticosteroid methylprednisolone 0 5 1 0g dissolved in 5 glucose or normal saline 20o 500ml intravenous drip, once a day, 3 times for a course of treatment, an interval of 3 15 days to start the next course of treatment, generally with 3 courses of treatment.

Pay attention to secondary infections and water and sodium retention. Prednisone is 1 mg per kilogram of body weight per day and gradually reduced to a maintenance dose after 3 months. 2 Cytotoxic drug 1 0g of cyclophosphamide dissolved in 5 glucose injection 250 50 ond intravenously, once every half a month to 1 month, the total amount of 6 8g discontinued.

Pay attention to recheck blood routine, liver function, etc., to avoid the occurrence of ***. 3 Anticoagulant heparin 75 100mg added to 5 glucose injection 250ml 500ml intravenous drip once a day; Rapid avoidance of coagulation 0 4ml intravenous push, once a day. 4 Platelet depolymerization drug Dipyridine 50mg, taken 3 times a day; Dencreids 0 25 orally 2 times a day.

A relative of mine is 57 years old and had kidney disease a long time ago.

At first, I was covered in water, nausea and vomiting, and protein 3+

Occult blood 2+, 24-hour urine protein quantification, online.

Found Dr. Liu :(shenbing 5) Reasonable Ann.

Diet has helped me a lot and I am full of hope for life.

Gaussian elimination method, but the binary equation is relatively simple, and it can also be used by the formula method.

Depending on your input format, you will also have to parse the string, which is more convenient in a scripting language, or limited to only the coefficients.

The solution of x=(b2*c1 - b1*c2) (-a2*b1 + a1*b2) is: x=(b2*c1 - b1*c2) (-a2*b1 + a1*b2)x=(b2*c1 - b1*c2) (-a2*b1 + a1*b2).

y=(a2*c1 - a1*c2)/(a2*b1 - a1*b2) thing that science assures him is

To put it simply, the crescent moon is the result of the accumulation of cells or fibrous tissue in the renal sacs, forming a crescent-like shape. These include cellular crescents, cellular fibrous crescents, and fibrous crescents. Nephrology talks about crescent nephritis, which is divided into three types, and there are all differences in prognosis.

If it is not easy to say for you personally, you have provided too little information. Further information would be provided. It is mainly a detailed report of kidney wear and ** process.

Crescent formation is a severe damage and rupture of the glomerular capillary wall caused by various causes, and is a pathological type of kidney biopsy. If this crescent occurs in patients with IgA nephropathy, it is clinically called IgA crescent nephritis . Crescent is often seen in many other glomerular lesions, and its formation often indicates a serious disease with a poor prognosis, so it is not a terminal disease, but it should be paid enough attention.

Because the crescent body shape can easily cause a sharp deterioration of kidney function, uremia is formed.

Crescent nephritis, also known as rapidly progressive glomerulonephritis, presents with an acute onset of hematuria, proteinuria, edema, and hypertension. In some patients, gross hematuria occurs, and red blood cell casts may be seen in urine sediment. In addition, some patients will have oliguria and anuria in the short term, most of them will deteriorate sharply in renal function, and some patients may rapidly progress to uremia and rely on dialysis.

Some patients with ANCA-associated small vessel vasculitis and systemic lupus erythematosus may present with multi-organ involvement, and pulmonary hemorrhage can occur in Goodpasture syndrome. In addition, some other diseases such as IgA nephropathy may have infection-related hematuria and elevated serum IgA levels, and Henoch-Schonlein purpura may have purpura, joint pain, and abdominal pain.

In addition to puncture, there is no way to know whether there is a star and moon body--- and people with more red blood cells are more likely to have an star and moon body

The crescent body must be punctured by the kidneys to be clear, and it cannot be seen by symptoms alone.

Only a puncture can be known.

Mr. Xie: Hello!

Judging from your pathological results and blood creatinine level, it is unlikely that you will recover, your pathology is IgA nephropathy, the medical history should be relatively long and not found and timely**, take medicine and maintain dialysis first, control blood pressure well, add some anticoagulant drugs such as aspirin, warfarin, etc.

Dr. Song, Department of Nephrology, First Affiliated Hospital of the PLA General Hospital.

Song Yan of the First Affiliated Hospital of the General Hospital of the People's Liberation Army.

It's okay to pick the right one. The mentality should be correct, which is very helpful for the ** of kidney disease.

The patient has edema, this disease can be cured, and you must have confidence.

Be brave, there will be a way, take care.

It's okay!! I'm 13 years old and have hardening!! It will be good if you have a happy problem?!