How is hyperlipidemia formed, and how is hyperlipidemia caused?

The long-term acidification of human body fluids makes the acidic substances in the blood in the body continue to increase, especially the plasma in the acidic environment, which cannot effectively remove the acidic substances produced by metabolism, such as non-protein nitrogen, fatty acids, excessive lactic acid produced by amino acid decomposition, especially fatty acids and cholesterol, low-density lipoprotein, etc., which increases blood lipids over time. Nutritional regulation of small molecule active peptide health products.

Fat is a type of energy. Fats are made up of glycerol and fatty acids. The types and lengths of fatty acids vary.

There are three main categories of fatty acids: saturated fatty acids, monounsaturated fatty acids, and polyunsaturated fatty acids. After eating, the food is absorbed and turned into energy to maintain the body's metabolic function and daily activities.

Once the food (energy) consumed cannot be consumed by the body immediately, the surplus energy will be converted into neutral fat and stored in the fat cells.

In the blood, high-density lipoprotein (HDL) and low-density lipoprotein (LDL) are responsible for transporting fat. Low-density lipoprotein (LDL) transports triglycerides, and he's like a garbage truck, dropping as he goes. High-density lipoprotein (HDL), on the other hand, is a sweeper that is responsible for removing blood fat.

Normal condition 2 is in a balanced state to ensure the balance of blood lipids in the human body. When LDL increases, and cells don't consume as much fat, blood lipids increase.

Countermeasures: Eat more foods rich in omega 3, fish oil, seal oil, etc., which can increase high-density lipoprotein (HDL) and play a role in regulating blood lipids. There are also olive oil, tea oil, etc.

There are two aspects of hyperlipidemia**: one is that the diet is fatty and sweet, which damages the spleen and stomach; The second is that after middle age, the kidney qi gradually declines, the yin and yang of the kidney are deficient, and the fire is reckless, resulting in hyperactivity of the liver yang, which is consistent with the conclusion of modern medicine that dyslipidemia and fat metabolism disorders lead to atherosclerosis and coronary heart disease, and are one of the most important elements of ischemic heart disease and cerebrovascular disease.

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What can not be eaten for high blood lipids.

High cholesterol foods.

Excessive intake of cholesterol foods will lead to an increase in cholesterol content in the human body, and an increase in cholesterol content will lead to narrowing or even blockage of blood vessels, which is the root cause of atherosis.

Such as: animal offal, eggs, etc.

High-fat foods.

Why can't you eat high-fat foods? Hyperlipidemia itself is due to the accumulation of fat in the body, which cannot be metabolized, resulting in an increase in blood lipids in the body. If you want to eat more fat, you will only increase the level of blood lipids in your body, resulting in a continuous increase. Harmful to the body.

Such as: meat, cream, peanuts, etc.

High-sugar foods.

In the body, excess sugar is converted into triglycerides and stored. That is, it is found in the human body in the form of fat. It goes back to the first point.

Eat a salty diet. Put less salt in your daily diet, salt contains sodium, and if the sodium content increases in the body, it is easy to increase blood pressure. Therefore, salt intake should be restricted.

Drink less alcohol. When the liver metabolizes alcohol, it consumes coenzymes, resulting in insufficient oxidation of fatty acids and the synthesis of triglycerides. This leads to an increase in triglyceride levels, resulting in hyperlipidemia.

Don't smoke. Patients with hyperlipidemia must not smoke, because the toxic substances in the smoke will damage the blood vessel wall, causing blood to form a blood clot when the blood flows through the blood vessels, so it is important to pay attention.

Hyperlipidemia is usually caused by family genetics, bad lifestyle habits, secondary diseases, etc. If elevated blood lipids are found, intervention should be given in a timely and early manner to avoid progression to hyperlipidemia and affect physical health.

1. Familial inheritance: there is a certain genetic tendency to hyperlipidemia, if one of the patient's parents has hyperlipidemia, the probability of hyperlipidemia will also increase, and most of them will have lesions in childhood.

2. Bad living habits: If you often overeat and overeat greasy foods, such as fried chicken, etc., it will lead to the accumulation of cholesterol, triglycerides and other lipids in the blood, and you will exercise less often, which will increase the blood viscosity and cause hyperlipidemia.

3. Secondary diseases: such as diabetes, hypothyroidism, Cushing's syndrome, etc., insufficient insulin secretion in diabetic patients, resulting in the conversion of excess blood sugar to other metabolic forms, such as blood lipids, will cause the occurrence of the above conditions. Patients with hypothyroidism secrete less thyroxine, which affects the metabolism of fat, and patients with Cushing's syndrome have increased secretion of glucocorticoids by the adrenal glands, resulting in metabolic disorders.

In addition, if patients often take contraceptives, especially long-acting contraceptives, the estrogen contained in them will affect blood lipid levels, and long-term use can easily cause hormone level disorders and metabolic disorders in the body, resulting in increased blood lipids.

Long-term high blood lipids can cause cardiovascular and cerebrovascular diseases, and common complications include cerebral infarction, stroke, coronary heart disease, myocardial infarction, heart failure, etc. The main manifestations of hyperlipidemia are high triglycerides and serum total cholestein, which can form atherosclerosis, and atherosclerosis can cause cardiovascular and cerebrovascular diseases, and sudden death will occur in severe cases, and the mortality rate is very high.

Predisposing factors for high blood pressure. Many patients with high blood pressure have hyperlipidemia, and some medical experts regard hypertension as one of the complications of high blood lipids. But no matter how it is expressed, it is to show that high blood lipids can cause high blood pressure.

Hyperlipidemia is abnormal in fat metabolism, fat in the blood is deposited in the blood vessel wall, blood vessels are blocked, arteriolar resistance is enhanced, and blood pressure naturally increases.

Fatty liver with hyperlipidemia. Fatty liver and hyperlipidemia are difficult brothers, and their "encounters" have the same thing, both of which are due to the excess of body fat levels. Fatty liver refers to the fat content of the liver area exceeding 5% of the liver weight, 10% is severe fatty liver, and more than 25% is severe fatty liver.

If it is not cured for a long time, cirrhosis of the liver will occur, and the case fatality rate is very high.

There are many causes of hyperlipidemia, the common cause is poor diet, followed by lack of exercise, body fat can not be metabolized normally, resulting in high blood lipids in the body. Don't be too nervous if you find high blood lipids during the examination, in addition to developing good living habits, appropriate physical exercise, and a light diet. If the blood lipids are very high, drug intervention is still required.

Oral drugs such as atorvastatin calcium to lower blood lipids. The most critical reason for hyperlipidemia is its own cause.

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Different types of hyperlipidemia are different, and even within the same type there is heterogeneity. In general, genetic and environmental factors are involved in the pathogenesis. The disease is more common in obese or overweight people, people with a family history of the disease, and diabetic patients.

First, the main **.

1. Primary hyperlipidemia:

1) Genetic factors: mutations in genes related to lipid metabolism can lead to a decrease in the activity of lipoprotein-degrading enzymes, and defects in lipoprotein structure or receptors reduce the clearance of lipoproteins in the body or slow down catabolism; or increase the synthesis of lipoproteins, affect the absorption of fats in the diet, etc., causing various types of primary hyperlipidemia.

2. Secondary hyperlipidemia:

1) Excessive dietary fat: It is a common non-pathological factor that causes hyperlipidemia. The absorption of lipoproteins from the gastrointestinal tract is greatly increased, contributing to the development of hyperlipidemia.

2) Obesity: weight gain promotes the synthesis of apolipoprotein B by the liver, which increases LDL production; On the other hand, it promotes the synthesis of cholesterol in the liver.

3) Disease state: There are many diseases that cause abnormal blood lipid profile, such as diabetes, nephrotic syndrome, hypothyroidism, gout, obstructive hepatobiliary disease, pancreatitis, systemic lupus erythematosus, glycogen accumulation, myeloma, acute porphyria, polycystic ovary syndrome, alcoholism, etc.

4) Long-term heavy drinking: It can inhibit the oxidation of fatty acids in the liver and increase the synthesis of fatty acids.

2. Predisposing factors.

1. Age factor: With the increase of age, the weight will also increase; In the elderly, low-density lipoprotein (LDL) receptor activity is reduced, and LDL catabolic rate is reduced; Decreased bile acid synthesis, increased intrahepatic cholesterol content, etc.

2. Drug factors: many drugs can aggravate dyslipidemia, among which the two most common classes of drugs are antihypertensive drugs and steroid hormones. Among the former, diuretics and receptor blockers have the greatest effect on blood lipids.

The most common of the latter is estrogen, and whether used for hormone replacement** or as an oral contraceptive, estrogen can raise plasma triglyceride levels, especially in patients who already have triglyceridemia.

3. Smoking: Smoking can increase plasma triglyceride levels. Epidemiological studies have confirmed that smoking increases plasma triglyceride levels by 10% compared to the normal average.

4. Lack of exercise: People who are used to sitting still have higher plasma triglyceride (TG) concentrations than those who insist on exercising. Both long-term and short-term exercise can lower blood triglyceride levels.

5. Gender: The incidence rate of men is higher than that of women, 3 to 4 times higher in middle age, and the incidence of women increases after menopause, but the ratio of men to women is still about 1 times.

The main harm of hyperlipidemia is to lead to atherosclerosis, which in turn leads to many related diseases, the most common of which is coronary heart disease. Severe chylomicronsemia, which can lead to acute pancreatitis, is another fatal condition. So what causes hyperlipidemia?

What daily behaviors are prone to hyperlipidemia?

Four causes of high blood lipids.

1. High intake of saturated fatty acids and high cholesterol.

For example, some people like to eat fatty meat and animal offal, and some like to stir-fry vegetables with lard or other animal oil. If this is done for a long time, blood lipids will quietly rise.

2. Unhealthy lifestyle.

Long-term sitting, alcoholism, smoking, nervousness or anxiety can all cause elevated blood lipids. However, we can eliminate these harmful environmental factors by controlling our diet, weight, exercise, and quitting smoking.

3. Lack of physical activity and physical exercise.

Nowadays, most of the work of young people is engaged in mental work, and the busy work during the day makes the office workers exhausted, and when they come home for dinner at night, they stay at home to watch TV or play on the computer, and lack of appropriate physical exercise, which induces the appearance of hyperlipidemia.

4. Genetic factors.

Inheritance can cause hyperlipidemia through a variety of mechanisms, some of which may occur at the cellular level, mainly by defects in cell surface lipoprotein receptors and defects in certain intracellular enzymes (such as defects or deficiencies in lipoprotein lipases), or in lipoprotein or apolipoprotein molecules, often due to genetic defects.

High blood lipids may be caused by genetics, lifestyle habits, age, diseases, and medications, and blood lipids are the general term for all lipids contained in plasma. If you are in a state of high blood lipids for a long time, it is easy to lead to cardiovascular and cerebrovascular diseases such as atherosclerosis and cerebral thrombosis. It is recommended that patients with high blood lipids go to a regular hospital for identification** to avoid delaying the condition.

1. Heredity: Some patients with high blood lipids may have single or multiple gene mutations, often with a certain familial aggregation. If the parents have high blood lipids, the children may also be affected by genes, and the blood lipids may also be high.

2. Lifestyle habits: mainly including long-term high-fat diet, insufficient exercise, smoking, alcoholism, etc., which can lead to high blood lipids;

3. Age: plasma cholesterol levels will increase slightly with age, and the activity of low-density lipoprotein receptors in the elderly will decrease, which will reduce their catabolism, resulting in high blood lipids;

4. Diseases: mainly including obesity, hypothyroidism, Cushing's syndrome, liver and kidney diseases, systemic lupus erythematosus, myeloma, polycystic ovary syndrome, etc., these diseases can affect the synthesis, transport and metabolism of lipids or lipoproteins through different mechanisms, resulting in elevated blood lipids;

5. Drugs: Long-term application of certain drugs may lead to secondary increase in blood lipids, such as the use of diuretics such as hydrochlorothiazide, which can cause an increase in serum total cholesterol, triglycerides, and low-density lipoprotein, and a decrease in high-density and low-lipoprotein. In addition, receptor blockers such as propranolol hydrochloride and bisoprolol can cause an increase in serum total cholesterol and low-density lipoprotein cholesterol, and a decrease in high-density low-lipoprotein cholesterol.

Long-term application of a large number of glucocorticoids such as prednisone acetate and dexamethasone can promote fat decomposition and cause an increase in plasma total cholesterol and triglycerides. If these drugs are taken orally frequently, it is recommended to check blood lipid levels regularly.