What are the specific symptoms of acute myelitis?

Acute ascending myelitis has a rapid onset, the lesion rises rapidly within a few hours or 1-2 days, and the paralysis rapidly spreads from the lower limbs to the upper limbs or medullary innervation muscles, resulting in dysphagia, dysarthria, paralysis of respiratory muscles, and even death. The most common disease of the nervous system is acute myelitis, which can cause great harm to patients. Knowing its early symptoms helps us to stay in time and thus avoid unnecessary troubles.

The early symptoms of acute myelitis are mainly as follows:

1. Acute onset of acute transverse myelitis, often developing to complete paraplegia within a few hours to 2-3 days. It can occur at any age, is more common in young adults, has no sex differences, and has a sporadic onset. Fever, general malaise, or symptoms of upper respiratory tract infection are common in the days or 1-2 weeks before the illness of the disease, and may be triggered by overwork, trauma, and cold.

The symptoms are mostly numbness and weakness of the lower limbs, banding sensation of lesion spots, or root pain, and then develop into complete transverse damage to the spinal cord, with the thoracic cord being most commonly affected. Motor, sensory, and autonomic dysfunction below the level of plaque.

1) Movement disorder: Spinal shock is common in the early stage, showing paraplegia, limb and hypotonia and loss of tendon reflexes, without pathological signs. The shock period is usually 2-4 weeks or longer, and it is longer in patients with severe spinal cord damage, complications of lung and urinary tract infections, and pressure sores.

During the recovery period, muscle tone gradually increases, tendon reflexes are hyperactive, pathological signs appear, and limb muscle strength gradually recovers from the distal end.

2) Sensory impairment: all sensory loss below the lesion segment, there may be hyperesthesia area or band-like paresthesia at the upper edge of the sensory loss level, which gradually decreases with the recovery of the disease, but is slower than the recovery of motor function.

3) Autonomic dysfunction: early urine and fecal retention, no feeling of bladder fullness, tension-free neurogenic bladder, overfilling of the bladder and filling incontinence; As spinal cord function is restored, the bladder capacity decreases, and urine fills to 300-400ml when it urins spontaneously, which is called reflex neurogenic bladder. No or little sweating below the level of injury,** desquamation and edema, crunchy stents, and hyperkeratosis.

2. Acute ascending myelitis has a rapid onset, the lesion rises rapidly within a few hours or 1-2 days, and the paralysis rapidly spreads from the lower limbs to the upper limbs or medullary innervation muscles, resulting in dysphagia, dysarthria, paralysis of respiratory muscles, and even death.

3. Demyelinating myelitis is mostly acute multiple sclerosis (MS) spinal cord type, and the clinical manifestations are similar to those of post-infectious myelitis, but the progression is relatively slow, and the disease often reaches its peak within 1-3 weeks. Anterior infection may be subtle, usually incomplete transverse lesions, with one or both lower extremities weakness or paralysis with numbness, sensory deficits with little or two planes, and urinary and fecal disorders. Evoked potentials and MRI may reveal lesions elsewhere in the CNS.

Typical symptoms: pain, numbness, fatigue, sensory disturbances, fever and bone pain, difficulty walking, local redness and swelling, etc.

Common symptoms: The early symptoms of acute myelitis are mostly weakness and numbness of the lower limbs, back pain at the lesion site, and a feeling of banding, and some patients have sudden paralysis without any other symptoms. Sudden numbness and weakness of unilateral or bilateral lower limbs, pain in the corresponding parts, movement disorders and sensory loss, and even urinary retention, paralysis and other manifestations.

May be accompanied by systemic symptoms such as fever.

Motor dysfunction , sensory deficit , dysphagia, dysarthria, shock nerve involvement, spinal intermittent rupture, urinary and urinary retention.

Sensory impairment: In patients with complete spinal cord injury, all sensations below the level of impairment are lost, while in some patients with impairment, part of the sensation is retained depending on the degree of impairment.

It can be cured with Chinese medicine.

Acute myelitis is more painful to attack, so what are the clinical manifestations of acute myelitis?

The clinical manifestations of acute myelitis are acute onset, low-grade fever, radicular pain, numbness and fatigue of the limbs, skin lesions, paralysis, and no other symptoms. Within a few hours or days, they mostly develop motor hunger disorder, bladder and rectal sphincter dysfunction. The early manifestations of motor disorder are spinal shock.

After 2 to 4 weeks, muscle tone gradually increases. Tendon reflexes are active, pathological reflexes are present.

As the disease recovers, the patient's sensory plane gradually decreases, but the recovery of motor function is slower and poorer. Autonomic dysfunction manifests early as stool retention, and later as spinal cord function is restored, a reflex neurogenic bladder can develop. Most patients with myelitis begin to recover within 8 weeks of onset and the rate of recovery begins to slow after 3 to 6 months, with one-third of patients leaving no sequelae and one-third of patients leaving moderate sequelae.

Another third of patients were repatriated with severe sequelae.

Acute myelitis refers to a disease in which the spinal cord reacts to an acute inflammatory response, and it has a variety of possibilities, including:

Viral infections: Viruses are the most common cause of acute myelitis, including poliovirus, epidemic virus, herpes simplex virus, etc.

Bacterial infections: Myelitis can also be caused by bacterial infections such as tuberculosis, Lyme spirochetes, meningococcals, etc.

Autoimmune diseases: Rarely, acute myelitis may be caused by autoimmune diseases, such as multiple sclerosis, SLE, etc.

Poisoning: Acute myelitis can also be caused by toxins and drug overdoses, such as heavy metal poisoning, arsenic poisoning, radiation poisoning, etc.

Hypoxia: In some cases, hypoxia can lead to acute myelitis, such as altitude sickness and asphyxia.

These are some of the common causes of acute myelitis, but this list is not exhaustive and in some cases** it is not clear. If symptoms of myelitis appear, you should seek medical attention as soon as possible to get a correct diagnosis and**.

The typical clinical symptoms of myelitis are motor disturbances, sensory deficits, and autonomic dysfunction. If you do not actively accept regular ** in time, the injury and inflammation of the spinal cord may be aggravated and spread, leaving sequelae and even paralysis.

1. Sensory impairment: all the depth and shallowness of sensation below the lesion segment are lost, and there may be a sensory hypersensitivity area or band-like abnormality at the upper edge of the sensory disappearance file, and with the recovery of the disease, the sensory plane gradually decreases, but the recovery of sensation is slower than that of movement, and it is not obvious. In some patients, sensory and motor symptoms gradually increase after the onset of the disease, even affecting the medulla oblongata, which is called ascending myelitis.

2. Movement disorders:

1) Acute onset, rapid progression, early spinal shock, manifested as quadriplegia or flaccid paralysis of both lower limbs, hypotonia, loss of tendon reflexes, negative pathological signs, patients often complain of inability to complete certain movements, such as upper limb weakness can not hold objects firmly, weakness of arms and difficulty in buttoning clothes.

2) Weakness of the lower limbs manifests as toe mopping, difficulty in going up and down stairs and sitting up. The spinal shock phase can last for 3-4 weeks.

3) After the spinal shock period, muscle strength begins to recover from the distal end, the injured segment is positive with the inferior pyramidal tract sign, muscle tone and tendon reflexes are gradually restored, and patients often complain of easy fatigue, stiffness of the lower limbs or clumsiness of movement when walking. Severe spinal cord injury often results in increased flexor muscle tone.

3. Autonomic dysfunction of reed fiber: urinary and fecal dysfunction, manifested as urinary retention, overfilling of the bladder, and filling incontinence; Fecal incontinence or constipation. Below the level of the lesion ** dry, no sweat, brittle nails.