Is radiculitis life threatening and what is radiculitis?

Acute**Infectious polyradiculitisUpdate time: [Overview] Guillain Barre syndrome, also known as Guillain Barresyndrome, is a common and serious disease of the nervous system. The main lesions are in the spinal nerve roots and spinal nerves, which may involve the cranial nerves.

Associated with viral infection or autoimmune reactions. The clinical manifestations are acute, symmetrical, flaccid limb paralysis.

1. Medical history and symptoms: A few days before the onset of the disease, there may be a history of cold, upper respiratory tract or digestive tract infection. The limb is acutely symmetrical, flaccid paralysis, often beginning in the lower extremities.

2. Physical examination findings: cranial nerve involvement can cause facial tongue paralysis, eye movement disorders, and bulbar palsy: in the early stage, there may be paresthesia such as limb numbness and pain, muscle tenderness, and a positive straight leg raise test.

Muscle weakness, decreased muscle tone, decreased or absent tendon reflexes, muscle atrophy. In severe cases, intercostal muscles and diaphragm are involved, resulting in life-threatening paralysis of respiratory muscles. 3. Auxiliary examination:

1.Cerebrospinal fluid (CSF) examination: protein is elevated, and the number of cells is low or mildly elevated"Protein cell isolation"。

2.White blood cell count and differential may be elevated in coinfection. Electromyography showed lower motor neuron damage, decreased motor unit potential in the acute phase, prolongation of non-latent time, and slowing of MCV and SCV.

**Measures] 1. If there is respiratory muscle paralysis, tracheotomy should be done as soon as possible. Strengthen airway management, keep the airway open, and maintain effective artificial respiration. 2. Reduce nerve root edema and improve microcirculation, severe patients can use hormones for a short time, dexamethasone 10-15mg, intravenous drip once a day, 706 generations of plasma 500ml intravenous drip once a time d7 for 10 days.

3. Nutritional neurological drugs: citicoline 250mg intramuscular injection once a day, melonin 20mg intramuscular injection once a day, nerve growth factor 10 million u1 times intramuscular injection once a day. High-dose C-ball (200-400 mg kg d) intravenous drip effect is better.

High-dose vitamins taken orally. 4. Use of antibiotics in co-infected patients. 5. Maintain the balance of water and electricity to strengthen nutrition, and patients with bulbar palsy should be fed a high-protein and high-calorie diet.

6. During the recovery period, traditional Chinese medicine, acupuncture, and physiotherapy can be used to promote the recovery of the function of the affected limb.

No, but as soon as possible**.

Acute radiculitis.

Generalized acute radiculitis, or guillain-barr'e syndrome, includes acute inflammatory demyelinating polyneuropathy (AIDP), acute motor axonal neuropathy (AMAN), and Miffer syndrome miller fisher syndrome, mfs)。 The clinical manifestations of AIDP and AMAN are similar, while MFS is dominated by ophthalmoplegia. It is important to note that acute radiculitis should be considered a syndrome and therefore can be diagnosed as long as the clinical presentation is consistent with the following section and other possible diagnoses have been ruled out; Some clinical manifestations of acute radiculitis are not completely consistent with those described in the literature, and the clinical symptoms can be found in the next section:

Motor, sensory, and autonomic manifestations are described.

Clinical manifestations The typical symptom is numbness in the hands and feet within a few days, extending from the distal end to the proximal end (or from the proximal end to the distal end); Motor nerve symptoms begin at the same time as or later after sensory symptoms occur; Within a few days, the muscles of each part are affected in turn, and the severity of the disease reaches its peak: from the onset of the disease to the onset of the most severe symptoms, no more than two weeks; About one-third of patients may have a history of mild colds or diarrhea in the weeks leading up to the onset of illness. Depending on the severity and severity of the nerves, motor, sensory and autonomic nerves can be affected.

Motor nerves: often begin with muscle weakness at the ends of the extremities (especially the lower extremities) and then invade the proximal muscles; However, some of them start from the proximal muscles; Depending on the progression of the disease, the muscles innervated by the respiratory muscles and cranial nerves, including swallowing, facial expression, and eye movement muscles, may cause dyspnea, dysphagia, facial paralysis, and diplopia.

Sensory Nerves: Macrosensory Deficiency is mainly a disorder of large-diameter sensory nerves, which is related to proprioceptive sensations, and the early symptoms of patients can be non-specific numbness, pinprick sensation or pain at the end of the limb, and as the disease progresses, proprioceptive disorders appear in stages, such as balance loss and sensory ataxia.

Autonomic nervous system: its symptoms include arrhythmia, abnormal sweating, pupillary contraction disorder, constipation, diarrhea, difficulty urinating, etc.

Neuritis is a disease of nerves or neuritis, and the clinical manifestations can include glove-like, stocking-like sensory impairment in the affected area, tingling, tenderness, numbness in the inflammation of the nerve, as well as decreased muscle tone or muscle atrophy, loss of tendon reflexes, etc., and autonomic dysfunction, such as cold extremities, dull nails, and physical exercise, light diet, and warmth.

Predominantly symmetrical limb motor, sensory, and autonomic dysfunction.

1. Sensory symptoms: at the beginning of the disease, it is mostly numbness or pain in the extremities, and there can also be hyperesthesia or abnormality, such as ant walking, and then the sensation decreases or even disappears, and the typical case is glove, socks and socks sensory disorder.

2. Motor symptoms: flaccid paralysis mainly at the distal end of the limbs, decreased muscle tone, tendon opposition decreased or disappeared, and muscle atrophy may occur later.

3. Autonomic nerve dysfunction: such as vasomotor of the hands and feet, sweating, paleness, coldness or redness and heat, tenderness or hyperkeratosis, dryness and cracking, etc.; In addition, due to different **, the clinical manifestations have their characteristics, such as the dyskinesia is not obvious when nitrofuran drug poisoning, and the pain and autonomic symptoms are prominent. If there is a single cranial neuropathy, peripheral facial neuritis (Bell's palsy) is common

The facial muscles of the hemilateral face are paralyzed, the frontal lines disappear, the eyes are incomplete, the nasolabial folds on the affected side become shallow, the corners of the mouth are drooping, and the corners of the mouth are crooked to the unaffected side when the teeth are exposed.

It may be for you to survive, which is generally fine, but it is more difficult**.

It has a certain impact on life and work, and it is recommended to ** as soon as possible.

Spinal radiculitis is a general term for inflammatory or degenerative lesions of spinal nerve roots caused by various causes. The cervicothoracic and lumbosacral segments are the most common, and they are called cervicthoracic radiculitis or lumbosacral radiculitis.

Clinical signs:1The onset can be acute or slow, and there is often a history of infection, poisoning, nutritional and metabolic disorders, spinal diseases, paravertebral muscle trauma and inflammation, transverse process trauma, etc.

2.Radiational numbness, pain within the posterior root innervation of the damaged nerve root, such as intercostal neuralgia due to thoracic radiculitis; cerothoracic radiculitis is ulnar and/or radial pain in the shoulder, neck and upper extremities; Lumbosacral radiculitis is manifested by pain from the lumbosacral region to the medial and/or lateral side of the lower extremities and the feet. Symptoms are often induced or aggravated by cold, cough, bowel movements, etc.

3.In the distribution area of the anterior root of the affected nerve roots, there are varying degrees of lower motor neuron paralysis: muscle weakness, muscle atrophy, and decreased or absent tendon reflexes.

For example, cervicothoracic radiculitis symptoms mostly occur in the shoulder girdle and upper limbs; Symptoms of lumbosacral radiculitis are seen in the lower extremities, with severe sacral nerve root damage and dystonic bladder and sexual dysfunction.

4.Cerebrospinal fluid may be mildly lymphocytosis, muscles within the affected area may show denervating electromyography changes, peripheral nerve motor and sensory conduction velocities may be slowed, and sensory nerve evoked potential latency may be prolonged.

5.When the lesion involves the arachnoid membrane, it is called meningoradiculitis, and if it involves the spinal cord at the same time, it is called spinal meningoradiculitis, which can produce spinal arachnoiditis symptoms.

It can lead to limb paralysis, and some patients may also have complications of cardiovascular dysfunction such as unstable blood pressure and tachycardia. Complications include paralysis and tachycardia.

It is uncomfortable everywhere in the body, which affects life.

Radiculitis is an autoimmune peripheral neuropathy characterized by demyelinating lesions of peripheral nerves and nerve roots and infiltration of small vessel inflammatory cells, and the clinical manifestations are acute symmetrical flaccid limb paralysis.

Also known as acute inflammatory demyelinating polyneuropathy, Guillain-Barre syndrome.

Cellular and humoral immune-mediated diseases** are not fully understood.

The clinical symptoms first manifest as muscle weakness, gradual motor and sensory impairment, and cranial nerve palsy.

Complications: respiratory failure, infection, hypotension, arrhythmia, etc. It is a neurological acute and severe disease, which can cause patients to die of serious complications such as respiratory failure.