The difference between thrombosis and arterial embolism

Thrombosis refers to the damage to the intima of blood vessels caused by arteriosclerosis or other reasons, when the blood in the blood vessels flows to the damaged site, the platelets or blood cells in the blood vessels will gradually be deposited in the damaged parts, and the endogenous coagulation mechanism is initiated, resulting in platelets or other components, which are deposited more and more, resulting in thrombosis in this area. The main difference between the two is that thrombosis is formed slowly at the site of the thrombosis, and the embolism is caused by the clots flowing here from other parts of the body, causing blockage, like the common cerebral thrombosis, which is the thrombus formed by cerebral arteriosclerosis and cerebral arteriosclerosis, and cerebral embolism is due to the thrombus that has been formed in the heart, which flows out from the heart to the aorta after falling off, and then flows through the cerebrovascular site, caused by embolism, if the lining of the veins of the lower extremities is damaged. Or the blood viscosity increases, and venous thrombosis can also form.

However, once the venous thrombosis falls off, it will flow into the pulmonary artery with the blood circulation, thereby causing embolism of the pulmonary artery, and the second thrombosis has a process, which is generally relatively slow, from the damage of the blood vessel to the formation of thrombosis, it takes a long time, even several years, and the embolism is very fast, and it can occur instantly.

Clause. 1. The thrombosis is different, the arterial thrombosis is mainly caused by the embolus falling off of the heart or large blood vessels, and the venous thrombosis is mainly due to the embolus reflux and blockage in the blood vessels of the whole body. Clause.

Second, the location of embolization is different, arterial thrombosis is mainly embolized in the peripheral arteries, and venous thromboembolism is embolized in venous vessels. Clause.

3. The components of thrombosis are different, arterial thrombosis is mainly platelets, and venous thrombosis has other blood cells and tissues in addition to platelets, such as red blood cells. Clause.

Fourth, the symptoms are different, arterial thrombosis is mainly pain, skin temperature is reduced, sensation is reduced, arterial pulse is weakened, venous thrombosis is mainly swelling, there may be ** ulcer inflammation, and the pain is relatively light. Clause.

Fifth, the causes of thrombosis are different, ** is also different, arterial thrombosis is mainly platelet aggregation to form embolus, ** is also mainly antiplatelet, venous thrombosis due to the slower blood flow velocity, and the coagulation mechanism is greater than the anticoagulation mechanism to form embolus, ** is mainly anticoagulation.

Arterial thrombosis is more than the plaque formed by atherosclerosis, which is easy to loosen and fall off and form thrombus with the blood flow. Thromboembolism is when an embolus is blocked in a certain stenosis in the bloodstream to form an embolism. Causes associated symptoms.

It is recommended that the key is to control the diet and actively prevent high-risk factors such as hypertension, diabetes, hyperlipidemia, and obesity.

Take the kidney as an example: renal artery embolism refers to the blood flow of embolus from other parts (such as deep veins from the lower limbs, heart, etc.) to the kidney, resulting in renal infarction.

Renal thrombosis refers to the pre-existing lesion of the renal artery, and there is a thrombus in the wall of the renal artery, which leads to renal infarction as the thrombus expands.

Most of the arterial thrombosis is formed on the basis of atherosclerotic plaque rupture, and to prevent arterial thrombosis, it is necessary to focus on preventing the formation of atherosclerotic lesions, stabilizing atherosclerotic plaques from rupture, and using effective antithrombotic drugs to prevent the formation of thrombis and blockage of blood vessels after plaque rupture. Hypertension, dyslipidemia and hyperglycemia are the main risk factors for cardiovascular and cerebrovascular diseases, which are related to the occurrence and development of atherosclerosis, plaque rupture and thrombosis. For the prevention of arterial thrombosis, efforts should be made to control risk factors that lead to atherosclerosis, such as hypertension, hyperlipidemia, diabetes, and smoking cessation, and effective antithrombotic drugs such as aspirin should be taken on this basis.

The formation of venous thrombosis, in addition to surgery and injury, is mostly related to slow blood flow and poor drainage. Venous thrombosis leads to poor blood return in the affected area, resulting in congestion and edema, and even local necrosis. Venous thrombosis can travel with the blood to other areas, leading to more serious consequences, such as pulmonary embolism.

To prevent thrombosis in the venous system, it is necessary to focus on avoiding factors that cause blood flow to slow down, and patients who are bedridden for a long time, have trauma to the lower limbs, postoperative patients, and those who take airplanes for a long time are all high-risk groups. Therefore, patients should leave the bed as soon as possible after surgery, and those who fly for a long time should pay attention to regular movement of their lower limbs.

Blood clots can cause arterial embolism. However, arterial embolism is not always caused by blood clots. That's the difference between the two.

Thrombosis refers to the damage to the lining of blood vessels caused by arteriosclerosis or other causes, and the blood clots form clots during the flow of blood. Embolism, on the other hand, is due to the fact that a blood clot that has formed falls off and circulates throughout the body with blood circulation, resulting in a local blockage.

Thrombosis is a multifactorial process in which a set of genetic and environmental factors interact and influence each other. The most common clinical characteristics of patients with thrombosis are familial hereditary, reversible, symptomatic severity, abnormal thrombosis site, and younger onset.

Cerebral infarction is a cerebrovascular disease caused by cerebral atherosclerosis, vascular intimal damage that narrows the lumen of the cerebral artery, and then causes local thrombosis due to a variety of factors, which aggravates or completely occludes the arterial stenosis, resulting in ischemia, hypoxia, necrosis of brain tissue, and neurological dysfunction.

Arteries and blood vessels are found throughout the body, so arteriosclerosis affects many organs. For example, cerebral artery stiffness can lead to cerebral infarction or cerebral hemorrhage, as well as vascular Alzheimer's disease; Carotid arteriosclerosis can lead to acute cerebrovascular accidents; Aortic sclerosis can lead to aortic dissection, abdominal aneurysm; Coronary arteriosclerosis can lead to coronary heart disease, and in severe cases, myocardial infarction; Renal arteriosclerosis can cause kidney failure and even uremia; Peripheral arteriosclerosis can cause ischemic pain, intermittent claudication, and severe necrosis amputation; Fundus arteriosclerosis can cause bleeding and vision loss in the fundus.

Thrombosis is formed slowly at the site of the thrombosis, and embolism is caused by the flow of thrombus from other parts here, causing blockage, like the common cerebral thrombosis is the thrombosis formed by cerebral arteriosclerosis, and cerebral embolism is due to the thrombus that has been formed in the heart, which flows out of the heart to the aorta after falling off, and then flows through the cerebrovascular site, causing embolism, if the vascular lining of the veins of the lower limbs is damaged. Or the blood viscosity increases, and venous thrombosis can also form. However, once the venous thrombosis falls off, it will flow into the pulmonary artery with the blood circulation, thereby causing embolism of the pulmonary artery, and the second thrombosis has a process, which is generally relatively slow, from the damage of the blood vessel to the formation of thrombosis, it takes a long time, even several years, and the embolism is very fast, and it can occur instantly.

The three major factors of deep vein thrombosis: slow venous blood flow, venous wall damage and blood hypercoagulability, are still recognized by scholars from all over the world. However, any single factor of the above three factors is often not enough to cause the disease, and it is often a combination of two or three factors that causes deep vein thrombosis.

Slow blood flow is the primary factor.

For example, blood vessels are like sewers, if the walls of the pipes are smooth, the water flowing down is fast, and there is very little garbage in the water, then the pipes are not easy to clog. On the contrary, the pipe wall is full of potholes, the water flow speed is very slow, and there are garbage such as rice grains and vegetable leaves in the water, and the sewer is easy to block. In the same way, if the lining of the blood vessels is very rough, the blood flow is very slow, and there is a lot of "garbage" in the blood, the blood vessels are easy to be blocked, and blood clots in the blood vessels are formed.

1.Thrombosis concept.

In the cardiovascular system of living organisms, the process by which blood is analyzed, aggregated, and coagulated into a solid mass is called thrombosis, and the solid mass formed is called thrombosis.

Wild rough 2Thrombotic conditions.

1) Cardiovascular intimal damage. First, due to the burst of subendothelial collagen fibers, the state dew activates factor 1, which initiates the endogenous coagulation system. Second, the damaged intima can release tissue factors that activate the exogenous coagulation system.

In addition, the intima of the injury is rough and uneven, which favors platelet sticking and can also lead to thrombosis.

2) Changes in blood flow status.

Slow blood flow. When the blood flow is slow, the platelets in the axial flow enter the side flow and are easy to be deposited in contact with the blood vessel wall. In addition, when the blood flow is slow, the locally adhered platelets and the coagulation factor formed are not easily diluted and washed away, which is conducive to thrombosis.

Eddy current formation. The impact of the vortex can cause the damaged endothelial cells to slough, expose the subendothelial collagen fibers, and cause platelets to side and accumulate due to centrifugal force, resulting in thrombus.

3) Increased blood coagulability. Refers to an increase in platelets, coagulation factors, or decreased activity of the fibrinolytic system. For example, extensive burns, excessive water loss, etc., make blood concentrate; Major surgery and trauma can cause compensatory thrombocytosis, and these new, naïve platelets are more viscous and easy to stick; In addition, tumor necrosis and early placental detachment can release tissue factors into the blood, resulting in increased blood coagulability.