Please help analyze this pathology report

What kind of pathology report is this, the cell diagnosis is not mentioned, and the atrophy is directly reported? It's too.,If my hospital comes out like this.,Immediately call ** to his director to learn from the new furnace.。。。 Still focal?

Still a film? it. This is by no means a report from a tertiary hospital, it is very rubbish.

May be an active phase of atrophic gastritis. Check for Helicobacter pylori infection.

The gastric horn is bracketed to indicate that the material sent for clinical examination is taken from the gastric horn. As a result, first of all, there is atrophy of the glands in chronic gastritis. Erosions and necrosis indicate that you should have ulcers. See if the gastroscopy report has any ulcers. It should not be too serious, but it should be actively symptomatic**.

It is not serious, it is a benign lesion, and it can be better if you take medicine.

Summary. The effect is fast, the effect is certain, the effect on rectal cancer is very good, the clinical application of rectal cancer patients is the first, its effect can shrink the lump in a short period of time, control.

This is the pathological diagnosis in the hospital's pathology ** report: (straight.

The effect is fast, the effect is certain, the effect on rectal cancer is very good, the clinical application of rectal cancer patients is the first, its effect can shrink the lump in a short period of time, control.

But other doctors told me that the report turned out to be cancer.

In fact, even cancer is not terrible, as long as you have a good attitude. It's okay for science to be so advanced now.

In fact, she is not sure of this list at all, and she must complete the pathology to know whether it is or not. If it's a primitive rent, it's good. It is very good to cure the lead of the socks.

They don't even need chemotherapy at all. No problem. Now you can only be suspicious.

It doesn't mean anything.

Possible lesions of cervical lymphadenopathy are:

1 chronic lymphadenitis, 2 lymphatic hyperplasia, 3 cervical lymph node tuberculosis, 4 malignant lymphoma, 5 lymph node metastases.

Biopsy or aspiration can be used to confirm this.

Hello! This pathological diagnosis report suggests that your stomach disease belongs to the period of chronic inflammatory activity. Edition.

Lymphoid tissue is a reticular connective tissue containing a large number of lymphocytes, which is mainly located in parts with important defense functions, such as the pharynx, digestive tract, and mucous membranes of the respiratory tract, forming the first line of defense against foreign pathogens.

All inflammations have three basic pathological changes: metamorphosis, exudation, and hyperplasia. Lymphoid hyperplasia can be seen in the lesional tissue of the gastric mucosa, indicating chronic inflammation, which is the result of long-term struggle between local anti-inflammatory factors and inflammatory factors, and is not lymphoma, so there is no need to worry too much.

If the pathology report is "gastric mucosal epithelial dysplasia" and "intestinal metaplasia", then you should be vigilant, because these two conditions are precancerous.

Chronic superficial gastritis, rational use of drugs for 4-6 weeks, most of them can be significantly improved or clinical, please receive the guidance of specialists for specific medication, in order to ensure the safety and effectiveness of medication.

Chronic stomach disease, pay attention to "three points **, seven points of recuperation". Excessive dependence on drugs does not solve the underlying problem, improves and consolidates the efficacy, prevents stomach problems**, and requires special dietary conditioning. Good luck soon**.

According to the pathological description, it is not too easy to judge, it is suspected that it is an early symptom of lymphoma, but it is still necessary to see the blood test report to confirm the diagnosis, you can send your blood test report to have a look.

Proliferative gastritis, which has the potential to become cancerous.

Hypertrophic gastritis. One of the most refractory stomach diseases, commonly used surgery**, more than 95% of stomach cancer is caused by hypertrophic gastritis.

It is only gastric mucosal hyperplasia, and the gastric mucosa that has not yet metaplasia, is only chronic gastritis now, and beware of chronic atrophic gastritis and gastric cancer in the future.

Chronic gastritis!Recommendations: Medications**, regular gastroscopy!Done!

Hello!Because the history is not detailed, the pathological findings (right wall pleural biopsy) are interpreted as follows

The goal of this pathological examination is to rule out the possibility of a tumor. The first is a routine pathologic finding, which suggests a small number of morphologically abnormal cells (large nuclei, different from normal cells), but further investigation is needed to find more evidence to support or rule out malignancy.

Immunohistochemical staining and genetic testing are molecular pathological examinations that are increasingly relied on in pathology. In this case, immunohistochemical staining and fluorescence in situ hybridization were performed. Among them, immunohistochemical staining results showed that only a few mesothelial cell markers were positive (CK, D2-40), while other mesothelial cell markers such as WT-1, CK5 and 6 were negative, and other immunohistochemical staining results excluded the possibility of lung epithelial ** tumors (MOC31, TTF-1 and CEA negative).

At present, combined with morphological and immunohistochemical staining results, the evidence of malignancy is insufficient.

Since molecular pathological examination (fluorescence in situ hybridization) showed that the p16 gene was deleted, considering that the deletion of the p16 gene may be related to the occurrence of tumors, although the evidence of the diagnosis of malignant tumors was insufficient in this pathological examination, based on the results of molecular pathological examination, it was recommended that clinicians further examine or follow up the changes in the patient's condition according to the patient's condition.

Although the above complex pathological examinations have been completed, no definitive conclusions have been drawn due to the complexity of the pathological diagnosis and the complexity of the disease. The pathological diagnosis of malignancy must be based on sufficient and clear evidence, and the changes in the condition are sometimes atypical, and inflammation and benign and malignant tumors may not be clearly distinguished. In addition, there are limitations and difficulties in obtaining clinical materials.

Therefore, when the pathology report is uncertain, it is a wise choice to closely observe the changes in the patient's condition, and then send the materials for pathological examination if necessary.

One. Main diagnoses: lobar pneumonia, septic shock. It is mainly caused by Streptococcus pneumoniae. There are often triggers such as cold, fatigue, alcoholism, and upper respiratory tract infection.

Two. Lobar pneumonia is mainly a fibrinous exudative inflammation of the alveoli. It usually affects only one lung, and is more common in the lower lobes, but can also occur in more than two lobes sequentially or simultaneously. The typical natural development process can be roughly divided into four stages:

1 Hyperemia and edema phase.

It is mainly seen 1 to 2 days after the onset of the disease. To the naked eye, the lobes of the lungs are swollen, congested, and dark red, and pale red serous overflow can be seen on the extruded section. Microscopically, the capillaries of the alveolar wall are dilated and congested, and serous exudate can be seen in the alveolar cavity, among which a small number of red blood cells, neutrophils, and alveolar macrophages are seen.

Streptococcus pneumoniae can be detected in the exudate, which allows the bacteria to multiply rapidly in protein-rich exudates.

2 Red hepatic stage.

This period generally begins 3 to 4 days after the onset of the disease. To the naked eye, the affected lobes are further enlarged, the texture becomes firm, and the section surface is grayish-red and rough. Fibrinous exudates may be present on the pleural surface.

Microscopically, the capillaries of the alveolar wall are still dilated and congested, and the alveolar cavity is filled with exudate containing a large number of red blood cells, a certain amount of cellulose, a small amount of neutrophils and macrophages, and the cellulose can pass through the alveolar interforamen to connect with the cellulose network in the adjacent alveoli, which is conducive to alveolar macrophages to engulf bacteria and prevent further spread of bacteria.

3 Gray hepatic stage.

Occurs on days 5 to 6 after onset. To the naked eye, the lung lobes are swollen, solid as liver, the section surface is dry and rough, due to the compression of the capillary pressure of the alveolar wall at this stage, the congestion subsides, most of the red blood cells in the alveolar cavity dissolve and disappear, and the cellulose exudation increases significantly, so the consolidation area is grayish-white. Microscopically, the alveolar exudate is mainly cellulose, and a large number of neutrophils and few red blood cells are seen in the cellulose reticulum.

The capillaries of the alveolar wall are compressed and the anemia appears. Most of the exudate has been eliminated and is not easy to detect.

4. Dissolution and dissipation period.

About 1 week after the onset of the disease, with the gradual enhancement of the body's immune function, the pathogenic bacteria are engulfed and dissolved by macrophages, neutrophils degenerate and die, and a large number of proteolytic enzymes are released, so that the exuded cellulose is gradually dissolved, and the macrophages in the alveolar cavity increase. The lysate is partially coughed up through the airways, or absorbed through the lymphatic vessels, and partially engulfed by macrophages. To the naked eye, the texture of the consolidated lung tissue becomes softer, the lesion disappears, and the yellow* color is asymptically yellow, and a small amount of pus-like turbid fluid can be seen spilling out of the squeezed section.

The lung tissue of the lesion is gradually purified, the alveoli are re-inflated, and the lung tissue can finally completely return to normal structure and function because the inflammation does not destroy the alveolar wall structure and there is no tissue necrosis.

1. (i.e., negative, here it indicates "not found", no infiltration.)

2. Invasive ductal carcinoma, that is, the cancer tissue breaks through the outer membrane of the breast duct and invades the surrounding tissues; The tissues listed above are only some of the surrounding tissues that have a significant impact on diagnosis and prognosis, but not all of them.

3. The classification of cancer tissue is graded by electron microscope, grade 3, indicating poor differentiation, high malignancy and strong metastatic ability of cancer cells;

4. 90% positive for KI-67 indicates that cancer cells are active in proliferation and sensitive to chemotherapy, and high-intensity chemotherapy may improve prognosis;

5. P53 (mutation) is 90% positive, and postoperative combined gene ** (now again) can be considered, which may improve the prognosis, and is currently in clinical trials.

Hello, if it's too complicated, I won't tell you Simply put, you are breast cancer, it is clear, but fortunately, the tumor has not invaded and the lymph nodes have not metastasized, which is a good phenomenon As for the grade 3, it means that the degree of malignancy of breast cancer is still high **The rate is high, and it will be rechecked more frequently in the future There may be radiotherapy or chemotherapy It depends on how your doctor decides P53 (90%+) K167 (90%+) These are graded by doctors, so you don't have to worry about it It's too difficult for you to understand.