Help analyze the thyroid function examination report

Hello; Chronic lymphocytic thyroiditis (Hashimoto's disease). Anti-TG antibody Thyroid peroxidase autoantibody TPO, most of the reasons are that the thyroid gland is damaged by inflammation. Traditional Chinese medicine can be antiviral, can clean up the release of thyroid hormones into the blood, can start from the patient's local lesions and systemic symptoms, from the overall regulation of clinical symptoms to improve or even disappear, re-examination of thyroid function TG TPO within the normal range, then the dose of traditional Chinese medicine can be reduced and stopped. In order to **.

In general, Hashimoto's disease is typically divided into three stages. (1) Early stage, hyperthyroidism. (2) In the medium term, hyperthyroidism and hypothyroidism coexist.

According to your examination report, high FT3 and FT4 and low TSH indicate that there is clinical hyperthyroidism, and high antibodies indicate inflammation and Hashimoto's hyperthyroidism.

According to your examination report, it is an indicator of Hashimoto's hyperthyroidism, which can be cured in time, and whether the condition is serious or not needs to be based on your actual situation, and the examination report can only see which thyroid disease you have.

Elevated FT3, elevated FT4, decreased TSH indicates hyperthyroidism, and elevated antibodies indicate inflammation.

The test sheet shows that the FT3 FT4 is too high and the TSH is too low.

High levels of anti-thyroglobulin antibodies and anti-thyroid peroxidase antibodies.

It should be Hashimoto's hyperthyroidism (Hashimoto's hyperthyroidism).

Hashimoto hyperthyroidism. Antibodies higher than three times can confirm the diagnosis of Hashimoto's thyroiditis, which is divided into three stages, hyperthyroidism, normal stage, hypothyroidism, and you are hyperthyroidism.

Hashimoto's disease causes hyperthyroidism and hypothyroidism to change all the time, and eventually the thyroid tissue will be destroyed, becoming hypothyroidism, and it will take medication for the rest of your life.

According to the examination report, it is Hashimoto's hyperthyroidism, and the condition is relatively serious, so I should consider going to a regular hospital as soon as possible.

What is the unit of your results, it seems that TSH is a bit low.

The following is for reference.

Total triiodothyronine (T3) is Xianqing Ren, Department of Pediatrics, First Affiliated Hospital of Henan University of Traditional Chinese Medicine.

Reference value] nanomoliters (NMO L).

Clinical significance] 1. Heightened:

Hyperthyroidism is increased in all cases, especially in T4, often 4 times higher than in normal people.

Elevated only, common in T3 hyperthyroidism, more common in iodine-deficient areas, and also in high-functioning thyroid adenoma, multinodular goiter, and hyperthyroidism and hypothyroxine tablets**.

In the early stage of hyperthyroidism or in the early stage of hyperthyroidism, T3 may be elevated.

Pregnancy, hepatitis, hyperthyroglobulinemia, or the use of contraceptives and estrogen can also cause T3 and T4 to increase, but the magnitude is small.

2. Reduction: Nephrotic syndrome, hypoproteinemia, etc., can reduce T3.

T3 may also be lowered with drugs such as receptor blockers, corticosteroids, phenytoin (Darentine), sulfopyrone (besulfonbutazone), etc.

Hypothyroidism, but the diagnosis of hypothyroidism is not as valuable as that of T4.

T3 is low in cirrhosis, early myocardial infarction, and poorly controlled diabetes.

Hunger can also lower T3.

Total thyroxine (T4).

Clinical significance] 1. Heightened:

T4 hyperthyroidism. Excessive intake of iodine-containing foods.

Pseudo-T4 hyperthyroidism. It can be seen in myocardial infarction, cor pulmonale, acute cerebrovascular disease, asthma, tumor, acute liver disease, etc. May decrease with improvement of the underlying disease.

Subacute thyroiditis.

May indicate hyperthyroidism, but early or mild hyperthyroidism is not as pronounced as elevated.

A small number of older people have hyperthyroidism.

2. Reduction: The diagnosis of hypothyroidism is more sensitive than that of T3.

Iodine-deficient goiter with decreased T4, normal T3, and elevated TSH.

Use glucocorticoids, estrogen, salicylic acid and other drugs and hyperthyroidism**.

Decreased T4 in critically ill patients indicates a poor prognosis.

Free triiodothyronine (FT3) and free thyroxine (FT4).

Clinical significance] Refer to T3 and T4.

Thyroid-stimulating hormone (TSH).

Reference value] 2-10 milliunit liter (mu l);

Clinical significance] TSH is an important indicator for the diagnosis of primary and secondary hypothyroidism.

Results may vary from lab to lab.

Your nail gong results sell the base dust as:

1. TSH decrease: It can be seen in subclinical hyperthyroidism, Hashimoto's hyperthyroidism, secondary hypothyroidism, etc.

2. TPO-AB: elevated, this is the thyroid antibody, which is more common in autoimmune thyroid diseases, such as Hashimoto's disease.

Based on the above results, there is a high possibility of Hashimoto's disease, and it is recommended to consult the instructing physician in combination with the clinical diagnosis.

The thyroid gland is normal but autoimmune is impaired and autoantibodies are high.

Although the level of T3 triiodothyronine is low, the level of thyroid-stimulating hormone TSH is normal, and the physiologically functional free triiodothyronine (FT3) and FT4 are normal, so hypothyroidism is not present.

The body is an organic whole, and if you are deficient in thyroxine, the body itself compensates, and the pituitary gland, the superior center of the thyroid gland, automatically increases the release of thyroid-stimulating hormone (TSH), but your TSH does not rise in any way. Therefore, there is no primary hypothyroidism.

Secondary hypothyroidism is also excluded. Secondary hypothyroidism is caused by a decrease in thyroid-stimulating hormone secretion caused by the pituitary gland itself, and then hypothyroidism, but your TSH is not too low, and the main thing is that the level of free thyroxine FT4 with physiological functions is above the midline of the reference range, and the free triiodothyronine is also normal. It is important to know that the thyroxine T3 and T4 in the blood are bound to globulin or albumin, and only a few are free, but these free have physiological functions, so you do not have hypothyroidism at all.

As for TMAB, the high level of TGAB itself only indicates that there is thyroid autoimmune damage, but it is not easy to judge whether it has reached the level of thyroiditis.

It must be combined with clinical practice, such as whether your thyroid gland is enlarged, whether you have nodules, etc.

Such a result does not need to be special in itself, only observation and follow-up are required.

Laboratory tests suggest that thyroiditis may be due to hypothyroidism.

The three --- of the first function are normal

There ---is a lack of hypermetabolism syndrome and endocrine descriptions in the description of the condition, such as: easy hunger, endocrine.

Excessive eating, thirst, sweating, irritability, irritability, menstrual irregularities...

There are many clinical types of hyperthyroidism--- such as: T3 hyperthyroidism, T4 hyperthyroidism...

Hyperthyroidism should also have a clinical differential diagnosis--- such as: myocarditis, cardiomyopathy, diabetes, methyleneitis...

In addition, in the preclinical stage of hyperthyroidism, laboratory tests can be normal.

Therefore, it is recommended that you re-examine at intervals, and during the re-examination, you should check eight items of thyroid function, blood lipids [low blood lipids in hyperthyroidism], cardiac enzymes, [differentiated from myocardial diseases], blood sugar [hyperthyroidism and diabetes are sometimes concomitant diseases], and blood routine [blood picture changes during hyperthyroidism].

The above is for your reference.

If you really suffer from hyperthyroidism, it's very simple, modern routine**, and it's good at home or in the outpatient clinic.

Did this Chinese medicine doctor read the report of the first three?

How is hyperthyroidism diagnosed?

Do you need to take Chinese medicine for hyperthyroidism?