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Anonymous users2024-02-09It is suggested that more coenzyme Q10 can be supplemented, coenzyme Q10, also known as decenquinone, is an activator of cell metabolism and cellular respiration, which can improve mitochondrial respiration function and promote oxidative phosphorylation. It is also a natural oxidant produced by the cell itself, which can inhibit the peroxidation of mitochondria and protect the structural integrity of biofilms. It has a non-specific enhancement effect on immunity, can increase the phagocytosis rate of phagocytic cells, increase the production of antibodies, and improve T cell function.
It can enhance the function of mitochondria very well, thereby improving mitochondrial aging, and it is very good to use Boaozhen Pyrovid Pro before, the ingredients contain PQQ, Q10, folic acid, vitamin B1, B6, B12 purple tea extract, olive leaf extract, Siberian larch extract extract The effect is very good, and it can also improve inflammatory aging, which is more comprehensive.
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Anonymous users2024-02-08First of all, as you get older, your mitochondria will also age and die. **When the production capacity of the mitochondria decreases and the number decreases, it will cause a series of aging feedback caused by insufficient energy You can try this product, which is specially designed to help mitochondria reverse aging, and can amplify the number of mitochondria and improve the anti-aging of the product.
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Anonymous users2024-02-07Because mitochondria are 95% of the energy production of living organisms, it can basically be said that there is no energy without mitochondria, and there is no way to survive, and as we age, the mitochondria in our body are gradually decreasing.
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Anonymous users2024-02-06The relationship between mitochondria and cellular senescence.
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Anonymous users2024-02-05a. Due to adaptation to specific functions, cell differentiation can change the type and number of organelles in the cell, a correct;
b. Senescent cells will have mitochondrial reduction, enzyme activity reduction and nucleus enlargement, etc., b correct;
c. The clearance of pathogen-infected cells is accomplished by apoptosis, c error;
d. The decrease of glycoprotein after cell carcinogenesis leads to the reduction of cell adhesion and easy to disperse and metastasize
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Anonymous users2024-02-04Human senescence arises from the senescence of cells, senescence of cells arises from senescence of mitochondria, and senescence of mitochondria derives from stained telomeres in mitochondria.
Telomeres are the longest at birth, and as we age, telomeres become progressively shorter. The longer the telomere, the longer the life expectancy of the person in theory, the different lifestyles of people, the different living habits, and the different lengths of the telomeres themselves at birth, will cause changes in the length of the telomeres. Telomeres are gradually shortening, but the rate of telomere shortening is different depending on lifestyle.
For example, some people seem to be 20 years old after 10 years, and some people are not much older after 10 years, but the telomeres are working. When the telomeres are short enough to make the cell unable to replicate properly, the cell becomes uncontrollable, and cancer occurs. As a result, cancer generally occurs in older people.
In short, a good lifestyle can prolong life. With a bad lifestyle, people's aging will be accelerated and their life expectancy will be shortened.
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Anonymous users2024-02-03The relationship between mitochondria and cellular senescence.
Regarding the origin of mitochondria, scientists have 2 theories, namely endosymbiotic origin and non-symbiotic origin. There has been a debate between the two theories, and according to the different timing of the symbiosis within the mitochondria, two schools have emerged. But all in all, it's the main place where the cells do aerobic respiration, it's the energy manufacturing factory in the cells, so we also call it the "power factory" of the cells. >>>More
The main role of mitochondria is function, and mitochondrial cells are able to provide the body with the energy needed for activity, which is very important for the respiratory system. Mitochondrial dysfunction occurs naturally as we age, and it is one of the hallmarks of aging. At present, the anti-aging product I know is Palovite, which targets mitochondrial anti-aging, which can increase the number of mitochondria in the human body and enhance the function of mitochondria. >>>More
Reductive molecules such as NADH and FADH2 (the reducing equivalent in the cytoplasmic matrix can enter the electron transport chain from the malic acid-aspartate shuttle system composed of retrotransporters or through the phosphoglycerol shuttle) undergo several reactions in the electron transport chain to finally reduce oxygen and release energy, part of which is used to generate ATP, and the rest is lost as heat energy. **Enzyme complexes on the inner membrane of the mitochondria (NADH-ubiquinone reductase, ubiquinone-cytochrome C reductase, cytochrome C oxidase) use the energy released during the process to pump protons into the mitochondrial membrane space in reverse concentration. Although this process is efficient, there are still a small number of electrons that prematurely reduce oxygen to form reactive oxygen species (ROS) such as superoxide, which can cause oxidative stress and degrade mitochondrial performance. >>>More
1. Like nuclear genes, they are expressed through transcription and translation, and these organelles have corresponding enzymes and amino acids, and the proteins expressed are generally used in and within the inner membrane. >>>More
The biggest impact of mitochondrial damage is human aging, Parkinson's disease, Alzheyers and other diseases are caused by mitochondrial damage >>>More