How is cerebellar ataxia diagnosed?

Let's go to the hospital and see if you are sure of the situation**.

It is generally manifested as imbalance and unsteady walking. There may be tumors, infarctions, and hemorrhages in the cerebellum.

Are there any other symptoms now?

Are there any symptoms of weakness in the limbs?

Spinocerebellar ataxia is the main type of inherited ataxia. In addition to cerebellar ataxia, manifestations may be accompanied by ocular movement disorders, slow eye movements, optic nerve atrophy, retinitis pigmentosa, pyramidal tract signs, extrapyramidal signs, muscle atrophy, peripheral neuropathy and dementia. According to the clinical examination of the cerebral nervous system, to determine whether the patient has cerebellar and spinal nerve chain disorder, Xiao Nasun and then through his family history, through magnetic resonance and genetic testing, to determine whether the patient has cerebellar atrophy.

According to the typical common symptoms such as ataxia, dysarthria, pyramidal tract signs, and poor finch skin, as well as the manifestations of ophthalmoplegia, extrapyramidal symptoms, and retinitis pigmentosa cherry cover, combined with MRI examination to find cerebellar and brainstem atrophy, the clinical diagnosis can be confirmed by excluding other cerebellar and brainstem degenerative diseases. However, it is still inaccurate to confirm the diagnosis based on the characteristic symptoms and signs of each subtype (except SCA7), and the PCR method can be used to accurately determine the continent type and the number of CAG amplifications for genetic diagnosis.

Cerebellar ataxia, which is characterized by irregularities in the strength, speed, amplitude, and rhythm of voluntary movements, is known as co-grip dyskinesias. May be associated with hypotonia, eye movement disorders, and speech impairments. 1. Abnormal posture and gait.

Cerebellar vermis lesions can cause ataxia of the head and trunk, leading to impaired balance, abnormal posture and gait. The patient is unsteady on his feet, has a staggered gait, and walks with his feet apart into an ataxia gait. When sitting, the patient spreads their hands and feet apart to maintain balance.

In the case of an upper vermisite lesion, the patient leans forward; In inferior vermisite lesions, the patient falls backwards. The cerebellar hemisphere controls the coordinated movement of the ipsilateral limb and maintains normal muscle tone. A damaged segment of the cerebellar hemisphere on the missing finger side is attacked, and the patient collapses to the affected side while walking.

2. It can also cause ataxia, speech disorder, eye movement disorder, hypotonia, and tendon reflex of ipsilateral limbs.

The symptoms of cerebellar atrophy ataxia are mainly manifested in patients who have unsteady walking, and even some patients are unsteady on their feet and unable to sit firmly when sitting.

Patients with cerebellar atrophy can take drugs to prevent arteriosclerosis, such as aspirin and statins, and oral drugs to nourish brain cells, which can prevent the progression of arteriosclerosis.

You use the name of Western medicine to seek diagnosis results, why can't you go to the Chinese medicine people who want to ask for money?

You can go to the hospital for a check-up.

After the patient has ataxia, if Hongjing is due to cerebellar ataxia, it is mainly not necessary to have some damage to the balance ability of the trunk, and for example, there is some unstable standing, etc., if the patient is due to vestibular ataxia, it may lead to some vestibular system defense coincidentally damaged, such as some balance disorders. If the patient is caused by a hereditary attack disorder, for example, it will be common to have some poor distance and other phenomena, and the key carrying of ataxia is still very harmful.

1. The diagnosis is mainly based on typical clinical manifestations, and the following characteristics are helpful for diagnosis:1History of prodromal infection.

2.Acute onset 3The main manifestation is acute cerebellar ataxia.

4.Systemic symptoms and other neurological symptoms are not obvious except for other neurological diseases. 2. Differential diagnosis 1

Specific neurologic infections such as encephalitis and meningitis can be confirmed by examination of cerebrospinal fluid (CSF) pathogens. 2.Drug poisoning: Ataxia caused by drug poisoning is seen in an overdose of antiepileptic drugs such as phenytoin.

Diagnosis is aided by history and blood concentration, and symptoms disappear when the drug is stopped. 3.Inborn metabolic abnormalities: ataxia caused by congenital metabolic abnormalities, such as hyperammonemia, tryptophan transport abnormalities, etc.

Diagnosis can be based on family history, metabolic characteristics, mental retardation, etc. Acquired metabolic abnormalities, such as hypoglycemia and hyposodemia, can also cause acute cerebellar ataxia. 4.

Posterior foveal mass lesions such as tumors, abscesses, and hematomas sometimes present with acute cerebellar symptoms, which can be distinguished by symptoms such as imaging and increased intracranial pressure. 5.Hereditary dominant ataxia can also be reversed to produce acute symptoms, which can be distinguished according to family history and course of illness.

6.Polyradiculitis or multiple sclerosis can present as acute or transient ataxia. 7.

Other hypoglycemia, hypoxia, head injury, labyrinthine diseases, etc. should also be differentiated. As for cerebellar degeneration or ataxia caused by cerebellar hypoplasia, it is chronic or progressive and can be easily distinguished from this disease.

If the patient has cerebellar ataxia, daily dressing, buttoning, holding water, writing, walking, etc. will appear inflexible, walking as if drunk, showing a "Z" shaped forward deviation state, and standing can also maintain the upright state of the body.

<> 13 manifestations of cerebellar ataxia!

Cerebellar ataxia, i.e., motor coordination disorders and speech ataxia, etc. Cerebellar ataxia is generally manifested as unsteady standing, drunken gait, poor distance argumentation, easy falling, eye rotation disorder, incoherent speech, difficulty writing, choking on drinking water, difficulty swallowing, clumsy movements, positive finger-nose test, unconscious tremors, and inability to take care of themselves.

1. Unsteady standing: patients with ataxia will shake back and forth when standing still.

2. Drunken gait: the patient's legs are very wide and extremely unstable when walking, and in order to maintain body balance, he will even use his upper limbs to coordinate, as if he has a drunken gait.

3. Poor distance recognition: It is difficult for patients to estimate the distance and speed of movement, which often leads to excessive movement.

4. Easy to fall: the patient is unable to maintain his body balance and is prone to wrestling without support.

5. Eye rotation disorder: patients with ataxia will also manifest as eye movement disorder, with both eyes swinging back and forth, and sometimes ** nystagmus.

6. Incoherent speech: like chanting poetry, the tongue is knotted and vague.

7. Difficulty in writing: it will tremble when writing, and the font will be unrecognizable.

8. Choking on drinking water: When the diseased part compresses the pharyngeal nerve, it will cause choking to a large extent.

9. Dysphagia: due to the damage of the brainstem nerve nucleus, ataxia will also lead to dysphagia in some patients.

10. Clumsy movements: patients have clumsy movements due to ataxia and trembling.

11. Positive finger nose test: ask the patient to touch the tip of his nose from different directions, and patients with ataxia often finger the nose are inaccurate, which is a positive finger nose test.

12. Involuntary tremors: Cerebellar ataxia often trembles unconsciously.

13. Inability to take care of oneself: due to movement disorders, eating disorders and involuntary tremors, patients with Gongqin Peiji disorder are often unable to take care of themselves.

Patients with ataxia should avoid going out as much as possible, sit up and walk as slowly as possible, and eat more high-protein, high-vitamin, and high-nutrient foods.

Manifestations of cerebellar ataxia, including abnormal postural gait, presence of tremor, and speech disturbances, eye movement disorders, and hypotonia, are as follows:

Clause. 1. Cerebellar ataxia can lead to abnormal posture and gait, and cerebellar vermis can lead to ataxia of the head and trunk, resulting in impaired balance of the patient, the patient is unsteady on standing, has a staggering gait, and walks with the legs apart, showing a wide basal gait. Lesions of the cerebellar hemisphere can lead to poor coordinated movement of the ipsilateral limb, and one cerebellar hemisphere may be damaged and may fall toward the affected side when walking.

Clause. 2. Patients may have intention tremor and poor distance discrimination, which means that the closer the action is to the target, the more obvious the tremor is, and the action is easy to exceed the target. Coordination of fine motor skills, such as writing larger and larger, and uneven strokes.

Clause. 3. It can cause speech disorders, and typical cerebellar ataxia language is called fulminant or poetic language, which is manifested as slow speech, slurred pronunciation, intermittent sound, or frustration and fulmination.

Clause. 4. Cerebellar ataxia, which can cause eye movement disorders, which can be manifested as coarse nystagmus in both eyes.

Clause. 5. Cerebellar ataxia, which can cause hypotonia, tendon reflexes may be weakened or disappeared. While percussion of the patient's knee reflex, the calf can swing incessantly, like a pendulum.

Abnormal posture and gait, unsteady standing, staggering gait, ataxic gait with legs apart when walking, and hands and legs spread out to maintain body balance when sitting.

First, if cerebellar atrophy ataxia caused by cerebrovascular disease is mainly given antithrombotic drugs**, including the application of some antiplatelet aggregation drugs, and some drugs to prevent atherosclerosis**, while giving some drugs to protect brain cells and active training, cerebellar lesions caused by cerebrovascular disease can usually be better recovered.

Second, there are some neurodegenerative diseases that can cause these symptoms, such as multiple system atrophy, which currently lacks effective means, mainly to give training.

Third, there are also some hereditary diseases, such as spinocerebellar ataxia, the patient's cerebellar atrophy is very obvious, and some **** should be given, and some coenzymes and some antioxidant stress drugs should be applied.

Cerebellar ataxia, most patients are hereditary cerebellar ataxia, this disease can be diagnosed by genetic classification when diagnosing, this method is also the basis for diagnosis. And in the later stage, the baby also needs to do head CT and electromyography examination. Once diagnosed, the baby needs to be given hormones**, which can be used for acute cerebellar ataxia.

The most common causes of cerebellar ataxia are, cerebral hemorrhage, cerebral infarction, cerebral flow, etc., cranial nerve damage caused by a variety of reasons, resulting in coordination disorders, and balance disorders, such as unsteady standing, unsteady walking, and staggering gait.

1. Through daily observation, this method is relatively simple and intuitive, if the patient has cerebellar ataxia, daily dressing, buckle, water, writing, walking, etc. will appear inflexible, walking as if drunk, showing a "Z" shaped forward deviation state, and standing can also maintain the body upright.

2. Ask the patient's genetic history to see if the patient's family suffers from the same disease, conduct a first-class monitoring for the patient, use genetic examination to check whether the patient has a genetic history of cerebellar ataxia, eliminate it from the root, and understand what kind of genetic disease it is.

3. If it is ruled out that it is caused by genetic factors, the patient can rely on the hospital's diagnosis and treatment equipment, such as electromyography, CT and other imaging, to understand the patient's condition and location, and observe the lesions of the cerebellum. And locate and diagnose the location of the damage to the cerebellar hemisphere, so as to cooperate with the medical **, so that the patient's condition can have a good **, so as to reduce the degree of the patient's uncoordinated movements.

How is cerebellar ataxia diagnosed? You go to the hospital and ask the doctor to check you, and she will diagnose it for you, don't guess at home.

Cerebellar ataxia is caused by lesions of the cerebellum itself or neurostructures related to the cerebellum, which are mainly manifested by abnormal posture and gait, and impaired coordination of movements. You need to go to the hospital for some relevant tests. Physical examination, antibody testing, pathogen culture, genetic testing, thyroid function testing, CT, magnetic resonance imaging, histopathological examination, etc.

When cerebellar ataxia is diagnosed, most of it is hereditary. Genetic classification can be used to confirm the diagnosis, and there is also a certain basis for diagnosis. In the early stages of diagnosis, you can wait until the child is born to do a CT or electromyography examination of the child's skull.

Through these diagnostic methods, the diagnosis can be made as soon as possible. Other than that. After the diagnosis of cerebellar ataxia, the patient needs to be given hormones**, which can be targeted**cerebellar ataxia.