Necrotizing enteritis in chickens How to treat necrotizing enteritis in broilers?

It looks very serious, so go to the hospital immediately**.

Reovic virus, adenovirus, parvovirus, coronavirus, rotavirus can become the cause of intestinal enterotoxic syndrome, these viruses cause intestinal inflammation, damage the absorption function of the intestine, and the typical symptoms of broiler chickens after infection are stunted, growth arrest and diarrhea The main manifestations of enteritis caused by viruses are: 1. At the beginning, individual chickens have thin and unformed feces, and the feces contain undigested feed 2. Over time, generally 3-5 days, most of the whole flock begins to have diarrhea, and the feces become unformed. Some chickens have watery diarrhea, there is more undigested feed in the feces, the feces are slightly light yellow in color, and the black loose stools appear severely, and the chickens in the middle and late stages of the disease will have frequent nodding or tremors and other neurological symptoms, some chickens rush forward, some retreat, and then paralyzed, screaming, crazy running and dying. Major pathological changes:

1. The main lesion is in the intestine. Hemorrhagic spots or hemorrhagic spots can be seen in the serous membrane of the intestine, the intestinal tube is distended, the intestinal wall is cut open and automatically turned outward, and the intestinal mucosa is thickened and wheat bran-like. 2. Some intestinal walls become thin due to the shedding of mucous membranes, the intestinal wall is brittle and fragile, the front segment is full of undigested feed fragments, showing water-like contents, and the back segment is full of orange mucus-like substances and undigested feed, showing fish intestines, tomatoes, carrot silks, egg whites of intestinal contents in the middle and late stages, and occasionally glandular stomach swelling.

The use of enteritis drug neomycin sulfate to control chicken enteritis, overfeed and diarrhea, improve the symptoms of indigestion of chicken feed, sauce color, white or yellow-green loose stools, increase chicken feed intake, and promote growth.

Combined with the use of enterotoxic syndrome drug colistin sulfate, it can effectively prevent and treat enterotoxicity syndrome or enteritis in laying hens, effectively control intestinal infections caused by gram-negative bacteria, increase feed intake of chickens, and improve feed conversion rate.

This disease is a contact infectious disease caused by clostridium septicais, Clostridium perfringens type A and Staphylococcus aureus alone, or in combination, causing acute high mortality in broilers or commercial laying hens.

Clostridium septica can be cultured anaerobically on blood agar or on egg yolk agar, and the spores are oval and located at one end of the thallus.

1) Popular features.

The disease often affects broilers at 4 to 8 weeks of age, commercial layers at 6 to 20 weeks of age, and broiler breeders at 20 weeks of age.

Pathogenic bacteria are widely present in soil, manure, dust, contaminated bedding, beverages and intestinal contents, and often settle on the ** and mucous membranes of chickens. **Mucosal wounds are often the breakthrough point for the invasion of pathogenic bacteria, and the decline of immune function is often the internal cause of the occurrence of this disease.

2) Clinical symptoms.

Sick birds often show depression, loss of appetite, leg weakness, and unsteady gait. Sick chickens are black and their feathers fall off, and lesions are common on both wings, chest, abdomen and legs. Sick chickens often die acutely, the course of the disease is generally not more than 24 hours, and the mortality rate is 1% to 60%.

3) Pathological changes.

A large amount of scarlet edema fluid can be seen under the skin on autopsy, the muscle color of the injured ** is pale or black-brown, there is edema fluid or gas between the muscles, and in some cases**, it is intact. However, edema or scarlet pulp exudate may be seen under the skin. Occasionally, a small number of white necrotic foci may be seen in the liver.

4) Laboratory diagnosis.

The diagnosis is made based on typical clinical symptoms and autopsy lesions, and pathogenic bacteria are often isolated from exudates, subcutaneous tissues, and muscles in the case.

5) Differential diagnosis.

There are many ** mycosis that must be distinguished from gangrenous dermatitis, such as gum red yeast, Aspergillus fumigatus, etc. The fungus can be found by tissue compression, and can be identified by combining the fungal isolation and culture.

6) Key points of prevention and control.

Adding chlortetracycline, erythromycin, oxytetracycline, penicillin, copper sulfate, etc. to feed or drinking water is effective for this disease.

Preventive measures include injecting 1-day-old chickens with multivalent inactivated Clostridium seedlings in poultry farms with high incidence of disease, which can reduce the incidence of disease. Strengthen feeding management, including the selection of high-quality bedding or net bedding before use, keeping the poultry house dry, regular disinfection, reducing the trauma of poultry and other measures, reducing the causes of disease.

1 Diagnosis of necrotizing enteritis.

Typical cases of late-stage necrotizing enteritis can be easily diagnosed.

Early cases can easily be misdiagnosed as (1) coccidiosis, (2) dysbiosis, (3) non-specific enteritis, and (4) infectious enteritis, all of which can be easily confused with necrotizing enteritis.

However, a Gram-stained smear of intestinal contents can be seen with many bacilli; Bacterial anaerobic culture, typical Clostridium weichsoni communities can be seen.

2 Prevention of necrotizing enteritis and**.

It is reassuring to know that Clostridium weisseri is most susceptible to enramycin, such as growth promoter products (EnlamycinTM enramycin premix);

5 Prevention and treatment techniques for necrotizing enteritis.

To prevent outbreaks of necrotizing enteritis, the goal is to maintain a healthy, stable intestinal environment. To achieve this, prevention of subclinical symptoms of coccidiosis should be one of the most important aspects. Enlamycin can reduce the clinical symptoms of coccidiosis, probably due to the strong inhibitory effect of enramycin on anaerobic bacteria that are secondary to infection.

According to the life history law of a 7-day growth and proliferation cycle of coccidia, and the previous pathogenesis pattern, it is necessary to increase the prophylactic coccidiosis drug before the onset of the disease to avoid the occurrence of necrotizing enteritis. The timing and skills of coccidiosis should be very particular: (1) before the obvious appearance of coccidiosis, (2) the effectiveness of coccidiosis drugs, (3) the dosage of coccidiosis drugs, and (4) the safety of coccidiosis drugs on food.

The use of high content of yellow corn in the feed will help to balance nutrients, easily absorb and homogenize nutrients. Immunization with live coccidiosis vaccine in broilers will still cause mild or short-term small changes in intestinal mucosal epithelial cells that do not enhance significant proliferation of Clostridium weisseri;However, the overproduction of coccidiosis after inoculation must be controlled to avoid damage to the mucous membranes. Severe outbreaks of necrotizing enteritis necromancer require the use of specific drugs**.

You've found the right platform, and I happened to be professional when I ran into your problem, which is really your honor.

I'm mainly a dog, and I don't know the most about chickens. Only a little bit understood. If you don't have the conditions to do laboratory analysis, you can try some broad-spectrum antibiotics, such as norfloxacin powder, mix some into chicken food.

Chicken enteritis can be treated with neomycin sulfate:

Indications] 1. The main treatment of viral enteritis, necrotizing enteritis, enteritis caused by protozoa and diarrhea have good effects;

2. Improve the symptoms of indigestion, anorexia, white or yellow-green loose stools in poultry feed, and autopsy symptoms intestinal diffuse bleeding, intestinal mucosa shedding, intestinal wall thinning, glandular and gastric edema, etc.;

3. While improving symptoms, increase feed intake, promote growth, and improve feed conversion rate.

Product advantages] 1. This product is formulated for hemorrhagic, ulcerative enteritis, diarrhea and other pathogenic characteristics

2. Neomycin sulfate is rarely absorbed when taken internally, and it is mainly a disease of intestinal diarrhea

3. Stubborn diarrhea in summer, intestinal Duqing with traditional Chinese medicine bacillus Kang, astringent intestine and dysentery.

Hope it helps.

Chicken enteritis ** protocol:

1.Do a good job in environmental sanitation, and at the same time do a good job in the cleanliness and sanitation of the trough and drinking water system, and often use pipeline cleaning agents to clean the water storage tank and drinking water line to ensure that the chicken drinking water is clean and pollution-free.

2.Prebiotics, enzyme preparations, mold adsorbents and high-quality sodium humate are added to the feed.

3.Bacitracin zinc, lincomycin, penicillin and other drugs mixed in the material can effectively prevent and control the disease.

4.Recommended**drug--intestinal toxinqing, which can be seen in one day to regulate the intestines and improve feces, **reasonable.

Enterovirus, adenovirus, parvovirus, some chickens have watery diarrhea, there are more undigested feed in the feces, the feces are slightly light yellow, and the serious black loose stools, growth arrest and diarrhea and other viruses caused by enteritis are mainly manifested as: 1. The beginning, coronavirus, rotavirus can become the cause of intestinal enterotoxic syndrome, these viruses cause intestinal inflammation, damage the absorption function of the intestine, and the typical symptoms of broiler chickens after infection are underdeveloped, and then paralyzed and screaming, The posterior segment is filled with orange-yellow mucus-like material and undigested feed, with occasional swelling of the glandular stomach and a wheat bran-like appearance. 2. Some intestinal walls become thin due to the shedding of mucous membranes, the intestinal wall is brittle and fragile, most of the whole flock begins to have diarrhea, individual chickens have feces become thin and unformed, and the feces contain undigested feed 2, over time, generally 3-5 days, fish intestines, feces become unformed, the middle and late stages of the disease will appear in chickens with frequent nodding or tremors and other neurological symptoms, some chickens rush forward, some retreat, intestinal mucosa thickening, crazy running death.

The main pathological changes: 1. The main lesions are in the intestine. Hemorrhagic spots or hemorrhagic spots can be seen in the serous membrane of the intestine, the intestinal tube is distended, the intestinal wall is cut open and the intestinal wall is automatically turned outward, and the anterior segment is filled with undigested feed fragments, showing watery contents, tomato-like, carrot-like shreds, and egg whites in the middle and late intestinal contents.

The symptoms of necrotizing enteritis in chickens are: yellow-white thin feces, sometimes yellow-brown pasty stinky feces, sometimes red or even black-brown coal tar-like feces, and some feces are mixed with blood and intestinal straw tissue; Severe loss of appetite, food reduction can reach more than 50% **: Commonly used antibiotics are penicillin, new tylosin (phosphate), vitamin nimycin, tylosin, ampicillin, bacillus, regomycin, lincolnmycin, kanamycin, gentamicin, etc.

1. Reduce feed protein in the early stage of growth.

2. Diclizurid, totrizuli, sulfonamides, acemequine, ampicillin, amoxicillin, metronidazole, antiviral drugs, cod liver oil, mucosal repair factors are used in drinking water and feed. The main ingredient of "Qiu Yanxiao" in the studio, totrizulil VK3 and intestinal Shu is a sulfonamide metronidazole component.

3. Increase the dosage of vitamin AD3 or cod liver oil to repair the intestinal mucosa, prevent stress, and improve the resistance of chickens. Because of the intestinal toxicity syndrome caused by the decline in growth rate, the ratio of feed to meat is high, but some of the mortality caused by is not very large, so it is often ignored by people, I have found that there are many farmers do not know about the disease, and not in time, resulting in the growth rate of chickens is very slow, and the ratio of feed to meat is very high, some as high as: 1 seriously affects the economic benefits, and even losses.

Moreover, this disease is easy to cause other epidemic diseases, so its economic loss is also very large, should cause the majority of farmers to pay enough attention.

4 We often see that many mucous membranes bleed or shed in the intestines, forming a slurry in the intestines. In particular, intestinal toxins are fine if they are mild, and only a thin layer is left in the intestines if they are severe. There is a mucopurulent discharge in it, and the intestinal absorption and utilization of the drug is not very good at this time, and the absorption rate of the drug is very low at this time.

My experience is that before taking medicine, drink water with potassium permanganate for 4 hours, clean up the intestines, clean up the contents of the intestines, and then use 5% glucose + VC for 5 hours, which can increase the energy of the chicken, and also facilitate the absorption of drugs when taking medicine. Then use the drug, which can have the effect of doing more with less.