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Nystagmus can cause dizziness.
Nystagmus and dizziness are generally hereditary congenital nystagmus. It is very likely that because of vestibular nystagmus, there is a phenomenon of vertigo and nystagmus, nystagmus will cause dizziness and headache and many other symptoms, and in serious cases, it will also cause your retina to detach, or your vision will decline, so if you have nystagmus, you should go to the hospital as soon as possible for diagnosis**, so as not to cause your vision to decline Many patients with vertigo can see various types of nystagmus in the acute stage, and some patients with vertigo or dizziness have no nystagmus on physical examination. Sometimes, people have nystagmus but no symptoms of vertigo or dizziness, but only a balance disorder.
So, what is the relationship between vertigo and nystagmus? As one of the most common positive signs in patients with vertigo and dizziness, nystagmus has important localization and diagnostic significance. Proper understanding and analysis of nystagmus plays an important role in better understanding vertigo and neurological disorders.
Nystagmus, referred to as nystagmus, refers to the inability to continuously fix the target, the eye slowly moves to one side away from the fixation target, followed by a rapid corrective nystagmus, which is an involuntary, biphasic, rhythmic, reciprocating eye movement, which can be physiological or pathological.
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When nystagmus and vertigo occur, cerebellar lesions, such as hematomas or tumors, are first suspected. It can be a tumor of both benign and malignant tumors, causing the patient to experience significant vertigo, accompanied by nausea, vomiting, and horizontal or vertical nystagmus. When such a situation occurs, you should choose to go to a local hospital in time.
Cranial CT is performed, and if necessary, the diagnosis can be confirmed by MRI scan of the brain, and if the lesion of the cerebellum is identified, it is recommended that surgery can be performed if surgery is indicated**.
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Nystagmus can cause dizziness. 1. Clinical manifestations of nystagmus 1. No self-conscious symptoms: congenital nystagmus 2. Self-conscious symptoms:
Acquired nystagmus, feeling obvious discomfort, uncontrollable nystagmus, obvious shaking sensation when looking at objects, seeing stationary objects as non-stop back and forth movement, varying degrees of vision loss, diplopia, dizziness and nausea after a long time 3. Compensatory symptoms: In order to see objects clearly, patients are accustomed to some abnormal movements to overcome discomfort. For example:
Compensatory head position, including head tilt, side face, involuntary head shaking, nodding head, etc. 4. Accompanying symptoms: ophthogenic nystagmus, which can be accompanied by visual impairment, refractive error, diplopia, etc.
Otogenic nystagmus includes vertigo, tinnitus, deafness, earache, etc. Central neurogenic nystagmus can manifest as headache, nausea, and ataxia. 2. Classification of nystagmus 1.
Oculogenic nystagmus: most of them are horizontal oscillating tremors, unable to distinguish between fast and slow phases, and the rhythm is small and persistent or permanent. Rarely vertical or rotational, without vertigo, but occasionally with a sense of wobble, symptoms disappear after the eyes are closed, and occur in miners or when there is partial extraocular muscle paralysis II.
Vestibular nystagmus: It can also be divided into peripheral nystagmus and vestibular central nystagmus. Peripheral patients are mostly horizontal or horizontally rotated, but not vertical.
In addition to nystagmus, it is often accompanied by dumping, and vertigo and vomiting are significant, which does not lessen when the eyes are closed, and can last for several weeks. Common in Meniere's disease, otitis media, labyrinthitis, acute vestibular nerve injury, etc.; Central disease refers to damage to the intracranial part of the vestibular nerve, vestibular nucleus, medial longitudinal tract, and the structure of the vestibule, cerebellum, and brain. It can be horizontal, rotary, or vertical.
Long-lasting, dizziness and vomiting, depending on the onset of the disease, patients with slow onset may not have vertigo. It is often accompanied by tipping and displacement. Usually due to peripheral vestibular, brainstem, and cerebellar lesions.
Its properties are more common in tumors, inflammation, vascular lesions, trauma, unsheathing diseases, and degenerative diseases.
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nystagmus and vertigo should be due to vestibular nystagmus, which generally causes symptoms such as vertigo and nystagmus, or can be separated or appear separately; Nystagmus alone cannot be called a disease and cannot be diagnosed by nystagmus alone, but must be diagnosed in conjunction with other symptoms, signs, and neuro-otology investigations.
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Nystagmus can be divided into congenital nystagmus and acquired nystagmus. Most patients have congenital nystagmus, and in congenital nystagmus, they are divided into perceptual nystagmus and motor deficit nystagmus.
Perceptual nystagmus, often accompanied by other eye disorders, such as physical deprivation, congenital glaucoma, congenital cataracts, and even fundus diseases with congenital optic dysplasia, can affect the child's visual function.
There is also a type of kinesthetic deficit nystagmus, which can also be called congenital infantile nystagmus syndrome, which is usually accompanied by fast and slow phases of tremor.
Nystagmus is an involuntary, rhythmic, pendulum-like or beating-like oscillation of the eyeball. The main causes of nystagmus are early eye motor dysplasia, such as congenital cataracts, which cause nystagmus, as well as family genetic history, Down syndrome, brain damage, or lesions, such as inflammation of the brain, tumors, degeneration, trauma, and vascular diseases, which may cause nystagmus. In addition, nystagmus can be caused by eye disease or eye dysfunction, degenerative changes of the optic nerve, severe astigmatism, high myopia, physical diseases, multi-site sclerosis, stroke, and severe ear inflammation. >>>More
The first type of nystagmus should be understood through the eye tracker and the two indicators of movement and sensation should be used to determine whether to operate or not, exclude acquired nystagmus, exclude the acquired nystagmus that cannot be operated, and evaluate whether the operation has room to improve vision through eye trackers and various professional examinations. For example, if the concave fixation time is too long, surgery is not recommended, and surgery is not recommended if the sensory function is too low. >>>More
There are 5 types of nystagmus in clinical practice: 1. Ophthalmogenic tremor Most of them have severe damage to central vision and some kind of eye disease, such as congenital cataract, albinism, corneal leukoplakia or staphyloma, etc., and most of them are pendulum-shaped horizontal tremor. 2. Labyrinthine nystagmus is mainly caused by middle and inner ear diseases, which is horizontal, but there are fast and slow phases, accompanied by vertigo, hearing loss, and balance disorders. >>>More
1. Lesions in the retina or refractive medium may lead to nystagmus. If the patient has been born with cataract and choroiditis and central cornea, then the patient will have opaque spots or refractive errors that cause the patient to develop amblyopia, in which case the patient still retains part of the vision, but the eyeball is always unable to be fixed, which will also cause nystagmus. >>>More
1. ****: symptomatic**.
2. Improve vision: ophthalmogenic nystagmus, the focus is to improve vision and prevent primary lesions that lead to random vision. Appropriate prisms can also be formulated to eliminate the compensatory head position and improve vision. >>>More