How to treat urinary construction caused by kidney failure?

Clause. 1. Diseases of the kidneys themselves. When patients develop acute and chronic glomerulonephritis or kidney stones and kidney cysts, this will lead to damage to kidney function, and if not treated, kidney function will become worse and worse, and eventually develop into chronic renal failure.

Clause. 2. Primary factors. In general, chronic kidney failure is related to chronic diseases, such as high blood pressure or high blood sugar.

After the patient develops hypertension, if it is well controlled, it will not cause any sequelae and complications, but the patient will not be cured for a long time, and the blood pressure will continue to rise, which will not only lead to chronic renal failure, but also induce cardiovascular and cerebrovascular diseases.

Clause. 3. Pharmacogenic factors. We all know that if patients take certain drugs for a long time, such as antibiotics or antipyretic and analgesic drugs, this will lead to the phenomenon of chronic renal failure.

To prevent this, patients must take medication under the guidance of a doctor.

Occlusion refers to a person with a urine output of less than 100ml in 24 hours or no urine in 12 hours, which can lead to urinary aconstruction in severe cases. Urinary atrophy is a disease characterized by difficulty in urination, and in severe cases, there is no urine excretion, and there is a lack of urination. It is more common in patients with severe shock and acute renal failure.

When the kidney function is abnormal, the urine cannot be released in time, and the toxins will accumulate in the body, causing poisoning symptoms. Kidney failure is not enough to rely on medication** alone, but also to pay attention to diet in daily life.

1. Sodium salt intake: edema should be eaten in a low-salt diet, so as not to aggravate edema, generally the daily salt amount should not exceed 2g, it is forbidden to preserved food, less monosodium glutamate and alkali, edema subsided, plasma protein is close to normal, can resume ordinary diet.

2. Protein intake: when renal failure syndrome, a large amount of plasma protein is excreted from the urine, the human body protein is reduced and in a state of protein malnutrition, hypoproteinemia reduces the plasma colloidal osmotic pressure, resulting in stubborn edema, and the body's resistance is also reduced, so in the absence of renal failure, it should be given a higher high-quality protein diet (1, such as fish and meat, etc.) in the early and extreme stages. This can help alleviate hypoproteinemia and some of its attendant comorbidities.

However, a high-protein diet can increase renal blood flow and glomerular filtration rate, keep glomerular capillaries in a state of high pressure, and intake of a large amount of protein can also increase proteinuria, which can accelerate glomerular sclerosis. Therefore, patients with chronic, non-polar renal failure syndrome should consume a small amount of high-quality protein (and in the case of chronic renal impairment, a low-protein diet ().

3. Fat intake: patients with renal failure syndrome often have hyperlipidemia, which can cause arteriosclerosis and glomerular damage and sclerosis, so the intake of cholesterol and fat-rich foods such as animal offal, fatty meat, and some seafood should be limited.

4. Supplementation of trace elements: due to the increase in the permeability of the glomerular basement membrane in patients with renal failure syndrome, in addition to the loss of a large amount of protein in the urine, it also loses some trace elements and hormones bound to protein, resulting in the lack of calcium, magnesium, zinc, iron and other elements in the human body, which should be appropriately supplemented. Generally, you can eat vegetables, fruits, grains, seafood, etc., which are rich in vitamins and trace elements.