What are the common causes of pulmonary edema?

Pulmonary edema is common** as follows:1Increased pulmonary capillary hydrostatic pressure:

It is more common in heart failure. 2.Increased pulmonary capillary permeability:

It is more common in severe lung injury disorders, such as hypoproteinemia due to viral infection: decreased capillary colloid osmotic pressure in the lungs, leading to the formation of pulmonary edema. Pulmonary edema caused by heart failure and hypoproteinemia has a good effect and a fast onset of action.

The most common** are: left ventricular insufficiency, hypoproteinemia, and infectious pulmonary edema.

Search up"pulmonary edema" will come out.

Excessive fluid accumulation in the interstitium of the lungs and/or overflow into the alveolar space is called pulmonary edema.

1) Cause: Fluid exchange and operation between blood vessels and tissues in the lungs.

There is a disorder that causes fluid and protein to leak out of the blood vessels, and.

Excess fluid accumulates when lymphatic tissue is cleared by the ability to clear it.

in lung tissue, it becomes pulmonary edema. 1.Hemodynamic pulmonary edema is the most important cause of pulmonary edema due to elevated pulmonary capillary hydrostatic pressure, which can be seen in the following conditions: (1).

Acute pulmonary edema is one of the cardiology emergencies, and its main clinical manifestations are: sudden severe dyspnea, orthopnea, accompanied by cough, often coughing up pink foamy sputum, the patient is irritable, cyanotic lips, sweating, rapid heart rate, both lungs are covered with crackles and wheezing, and in severe cases, it can cause syncope and cardiac arrest.

Typical acute pulmonary edema can be divided into four stages according to the pathological process, and the clinical symptoms and signs of each stage are described as follows.

1) Interstitial edema stage: the main manifestations are nocturnal onset dyspnea, forced to sit upright accompanied by cold sweat and restlessness, cyanosis of the lips, dry rales or wheezing can be heard in both lungs, tachycardia and increased blood pressure, at this time, due to pulmonary interstitial edema, increased pressure, narrowing of the bronchial tubes, and bronchospasm caused by hypoxia.

2) Alveolar edema stage: the main manifestations are severe dyspnea, orthopnea with fear of suffocation, bluish gray complexion** and obvious cyanosis of the lips, sweating, coughing, coughing up a large amount of pink foamy sputum, and incontinence. Both lungs are filled with sudden wet rales.

In the case of cardiogenic, the heart rate is rapid, arrhythmias, and the first heart sound at the apex is diminished, and pathological third and fourth heart sounds can be heard.

3) Shock phase: a large amount of plasma extravasation in a short period of time leads to a rapid decrease in blood volume in a short period of time, hypovolemic shock, and at the same time, due to the obvious weakening of myocardial contractility, cardiogenic shock causes shortness of breath, blood pressure drops, clammy cold, oliguria or anuria and other shock manifestations, accompanied by changes in consciousness.

4) End-stage: In a coma state, he often dies due to cardiopulmonary failure.

1) Symptoms: The patient presents with sudden onset of dyspnea, orthopnea, increased frequency, cyanosis of the lips, sweating, frequent coughing, and coughing up a large amount of white or pink foamy sputum.

2) Signs: Auscultation of both lungs full of wheezing and wet, rapid heart rate, hyperactivity of the second heart sound in the pulmonary valve area, muffled first heart sound at the apex, systolic murmur and diastolic gallop rhythm can be heard.

Hello: 1. Plasma colloidal osmotic pressure decreases.

Experiments in dogs have shown that pulmonary edema can occur when plasma colloidal osmolality drops; In patients without left-sided heart failure, a moderate fluid load (large infusions) is sufficient to cause pulmonary edema in the presence of reduced plasma proteins; Clinically, infusion of a large amount of crystalloid solution into patients in shock can easily cause pulmonary edema, which is very related to the dilution of plasma proteins.

2. Lymphatic insufficiency.

Pulmonary lymphangiopathy leads to incomplete drainage, which can lead to an imbalance in pulmonary fluid exchange. Mild mitral regurgitation or stenosis in dogs, and pulmonary edema may occur without pulmonary edema when the left atrial pressure rises, but edema occurs after incomplete ligation of pulmonary lymphatic vessels; Total ligation of pulmonary lymphatic vessels, although there is no valvular heart disease, can cause pulmonary edema. It is known that pulmonary lymphatic vessels can compensate for increased return by up to 3 10-fold (up to 25 100-fold in chronic interstitial pulmonary edema) when pulmonary capillary filtration increases.

Therefore, if the capillary hydrostatic pressure does not rise rapidly, pulmonary edema is not easy to occur. However, when pulmonary lymphangient insufficiency, this compensation is limited. For example, in silicosis, chronic lymphangitis obliterans obstruct lymphatic return, and in this condition, a rise in pulmonary capillary pressure is sufficient to cause pulmonary edema.

There are many reasons, and pulmonary edema is only a manifestation of lung lesions, so it cannot be said whether pulmonary edema will be contagious It mainly depends on whether the ** that causes pulmonary edema is infectious For example, tuberculous pulmonary edema may be contagious (or not), this also depends on the period of tuberculosis Traumatic pulmonary edema is not contagious, and pulmonary edema caused by heart disease is also not contagious So whether it is contagious or not depends on ** I wish you good health.

The early symptoms of pulmonary tuberculosis, after contact with tuberculosis patients, you should observe a few points for yourself. Some people will have low-grade fever, which is called low-grade fever in the afternoon, night sweats, fatigue, and then weight loss will occur in the later stage. Then the respiratory symptoms will be cough, sputum production, and blood in the sputum.

Therefore, if we come into contact with tuberculosis patients, we must monitor whether there is a cough or not, and whether the body temperature is normal or not. So what is a low-grade fever in the afternoon? Low-grade fever in the afternoon means that the body temperature begins to rise at 2 o'clock in the afternoon every day, and by 2 o'clock in the evening, the body temperature naturally returns to normal.

And this heat is low-grade, between 37 degrees 5 and 38 degrees 5, so we have to observe our own body temperature, and we must take more body temperatures.

And then what are night sweats? Night sweats are called night sweats when you secretly sweat every night after going to bed, and when you get up in the morning and sweat all over your body and the quilt is wet, this kind of patient is called night sweats. Therefore, anyone who has a low-grade fever, night sweats, fatigue, cough, and bloodshot sputum must pay attention to whether there is tuberculosis infection.