Distress Primary tuberculosis, caused by primary tuberculosis is

I am kidney tuberculosis neither sell medicine, nor recommend drugs, I am afraid of delaying you, how to take medicine after tuberculosis diagnosis is very important, most of them are eaten in the morning on an empty stomach, I have been eating for 8 months This is the 3rd group, you refer to: 1Anti-tuberculosis drugs:

Rifapentine capsules 3 capsules daily, one hour in the morning before meals, 2 times a week.

Ethambutol hydrochloride tablets 3 capsules daily, one hour before meals in the morning, once a day.

Isoniazid tablets 3 capsules daily, one hour before meals in the morning, once a day.

Prothiomethalide enteric-coated tablets 6 capsules daily, taken after meals, three times daily. 2 tablets each time.

Clarithromycin: Take one tablet twice daily.

2.Hepatoprotective drugs (because my biochemical test potassium is low, the doctor recommended to change it):

Silymarin meglumine tablets 3 times a day. Take 3 tablets after meals.

Hello, you asked me about the situation of taking medicine, now tell you, we exchange experience, you can refer to it. The medicine was prescribed from the Tongxian Chest Hospital in Beijing, and I think they are quite experienced in prescribing tuberculosis drugs, and I have seen a lot of people from all over the country who go there to prescribe tuberculosis drugs. In addition, I remember that I seem to have seen from **, that pyrazinamide is not good for the kidneys, it can cause high uric acid, you are consulting a doctor.

See if you have high uric acid because of the use of that drug.

Asked me how I reacted to taking the medicine, just took this set of medicines, nausea, dizziness, I didn't want to eat for three days, and now it's basically better.

I hope these are useful to you, you don't have too much psychological pressure, we will defeat the disease together, and we will all be able to **!

This is a patient's, written on my blog, it doesn't matter if you give it or not, I hope it will be useful to you.

It's been so long, you should have been contagious to people on the street.

Who is infected with tuberculosis? It's hard to be sure, it's possible that your neighbors and uncles are all infected, or it may be caused by those unknown bacteria patients in the society.

Answer]: A Primary tuberculosis refers to the tuberculosis caused by the patient's first infection with Mycobacterium tuberculosis. The pathological changes of paradisorder are the primary syndrome of primary lesions, tuberculous lymphangitis and hilar lymph node tuberculosis.

The vast majority of patients heal spontaneously, and only a very small number of patients worsen with enlargement of the primary lesion or lymph node lesion, causing airway, lymph tract, and hematogenous spread. airway spread causes lobular caseating pneumonia; Lymphatic spread causes tracheal bifurcation, paratracheal, mediastinum, and subclavian lymph node tuberculosis; Bloodborne spread causes bloodborne tuberculosis, such as extrapulmonary organ tuberculosis, acute systemic miliary tuberculosis, and acute pulmonary miliary tuberculosis. Secondary tuberculosis includes focal tuberculosis, infiltrative tuberculosis, caseating pneumonia, chronic cavitary tuberculosis, and tuberculoma.

Secondary pulmonary tuberculosis rarely causes acute miliary tuberculosis and usually occurs in immunocompromised patients.

Answer]: The pathological feature of primary tuberculosis is the formation of a primary syndrome. After the formation of the primary syndrome, although the bacteria spread to other organs in the body through the bloodstream or lymphatic tract in the first few weeks, due to the establishment of cellular immunity, about 95% of cases no longer develop, and the lesions progressively take Chang to do fibrosis and calcification, and achieve healing.

Sometimes hilar lymph node lesions continue to develop and spread through the lymphatic tract, resulting in bronchial lymph node tuberculosis and cervical lymph node tuberculosis. A small number of children who are malnourished or have other infectious diseases at the same time have enlarged lesions, caseous necrosis and cavitation, and some even spread intrapulmonary to form miliary tuberculosis or rapid systemic spread to form systemic miliary tuberculosis.

Lesions: First, the lower part of the upper lobe or the upper part of the lower lobe near the lung membrane with good lung ventilation is formed.

It begins as exudative, followed by cheese-like silver-tan necrosis.

It quickly invades the lymphatic vessels and reaches the hilar lymph nodes, causing tuberculous lymphangitis and hilar lymphadenopathy and caseous necrosis.

Return: 1The lesion is completely resorbent, and calcification or induration (latent or healed) is most common, and the presence of calcification indicates that the lesion has been present for at least 6 to 12 months.

2.Enlargement of the primary lesion, creating cavitation, Peribroncholynodalitis, formation of lymph node bronchial fistula, leading to endobronchial tuberculosis or caseating pneumonia, Bronchial lymphadenopathy, causing atelectasis or obstructive emphysema, Tuberculous pleurisy.

3. Deterioration and hematogenous spread, resulting in acute miliary tuberculosis or systemic miliary tuberculosis.

Children with primary tuberculosis have irregular fever, usually low-grade fever, accompanied by mild cough, loss of appetite, excessive sweating, slight emaciation, and a history of close exposure to tuberculosis. Physical examination of the lungs is often unremarkable, with hilar lymphadenopathy on x-ray, sometimes initial opacities in the lungs, and a positive "OT" test.

The prognosis of primary tuberculosis is generally good, and most children gradually recover after 6 months to 1 year, but if they are not timely and thoroughly**, especially when they are young, poorly nourished, or at the same time suffer from infectious diseases such as measles and whooping cough, tuberculosis lesions may spread throughout the body.