Is weakness in the limbs, sweating in the hands and feet a symptom of motor neurons?

It's hard to say, don't ask here, go to the hospital immediately for a detailed examination, don't delay the condition.

Caused by the disease, viral infection and immunity, MND is a disease with an increasing incidence and a younger age of onset.

There are many subtypes of this disease, some of which are more critical, develop rapidly and quickly involve the medulla oblongata, causing respiratory failure and death, while some subtypes tend to affect only some limbs in the first few years, and develop slowly for many years, before getting worse or progressing to another limb.

Problem analysis: multiple peripheral nerve damage is a demyelinating disease, ** many, the early symptoms are unknown, is an acute or chronic cannibalization of nerve damage pathological changes, it is mainly damage to the body's autonomic nerves and nerve roots, and lead to paresthesia in the limb discourse segments, the onset of the disease for too long will lead to low immune function in the human body, and the disease will gradually worsen after being invaded by virus or inflammation. **Improper and delayed** can easily lead to serious clinical symptoms, and in severe cases, incomplete paralysis occurs.

Opinions and suggestions: **plan: hormones have no effect on the disease, ** must be treated with Chinese and Western complex syndromes, enhance the body's immune function, and improve the body's ability to resist diseases.

Nourish the nerve, expand the microcirculation so that the damaged residual nerve can get sufficient blood supply, and prevent the disease from progressing. And use nerve regeneration drugs to stimulate nerves, activate paralyzed and shocking nerves and regenerate and repair nerves to get an early **.

MND does not cause numbness in the hands and feet. Motor neuron disease (MND) is a series of chronic progressive neurodegenerative diseases characterized by upper and lower motor neuron damage. The clinical manifestations are a different combination of upper and lower motor neuron lesions, characterized by muscle weakness and muscle atrophy, bulbar palsy, and pyramidal tract signs.

Sensory system and sphincter function are usually not affected. Because the sensory system is not affected, MND does not cause numbness in the limbs. There are various hypotheses for the pathogenesis and ** of motor neuron disease, and the exact pathogenesis is currently unknown.

Some data suggest that older men, history of trauma, and excessive physical exertion may be risk factors for the disease.

Motor neuron degenerative disease is a chronic neuronal cannibalization disease, which is difficult to heal in the later stage of the disease, and its severe onset can invade the anterior horn cells of the spinal cord and the nerve nucleus of the brainstem and the pyramidal cells of the motor cortex of the brain, resulting in dyspnea and life-threatening, about 5% and 7% of patients and genetic immunity abnormalities or viral infections. The remaining patients** are unknown, but have been reported to be related to heavy metals, chemical poisoning, and the surrounding environment. The efficacy of simple drugs ** is not good, the condition is difficult to control, and the disease leads to neurotrophic disorder and gradually aggravates the secondary deficiency neurological impairment, so that the neurological symptoms are further aggravated.

Finally, it leads to paralysis of the cervical spine and threatens life-type motor neuron disease, which is a group of chronic progressive degenerative diseases that mainly damage the anterior horn of the spinal cord, the motor nucleus and pyramidal tract of the cranial nerve. Symptoms such as muscle atrophy and muscle weakness are the most common. Such as:

Slurred speech, difficulty swallowing, difficulty moving, difficulty breathing, etc. Eventually, the patient died of inability to breathe while he was conscious. Therefore, this kind of patient is also called "ALS".

The main manifestations of this disease, the earliest symptoms are mostly seen in the early part of the hand brightness and rise, the patient feels weakness, stiffness, clumsiness of finger movements, gradual atrophy of hand muscles, and fasciculations can be seen. The distal limbs show progressive muscle atrophy, and more than half of the cases show atrophy of the thenar muscle of one upper limb and hand in the early stage, and then extend to the forearm muscles, even the pectoralis major muscle, the back muscles can also be atrophied, the calf muscles can also be atrophied, the muscles are atrophied, the limbs are weak, the muscle tone is high (stretching sensation), the muscle is tremor, the difficulty of moving, breathing and swallowing disorders and other symptoms. In the case of early pathological bilateral pyramidal tracts, spastic paraplegia of both lower extremities may be present.

Motor neurons, or externally conductive neurons, are neurons responsible for transmitting messages from the spinal cord and brain to the muscles and endocrine glands, innervating the activity of effector organs. It usually begins with small muscle weakness and gradual muscle atrophy in the hand, which can affect one or both sides, or start on one side and then spread to the other side.

The palms of the hands are flat due to the atrophy of the thenar muscles, and the claw-like hands are due to the atrophy of the interosseous muscles. Muscle atrophy expands upwards and gradually encroaches on the forearm, upper arm, and shoulder girdle. Muscle strength is weakened, muscle tone is reduced, and tendon reflexes are diminished or absent.

Weakness and soreness in the lower limbs may indeed be a manifestation of motor neurons, but many patients will have weakness and soreness in the lower limbs after the disease.

Night sweats have nothing to do with this disease, night sweats at night have the phenomenon of yin deficiency, or the lack of yang qi ignores the table, this has to be dialectical, you can go to Chinese medicine to check it.

Amyotrophic lateral sclerosis (ALS) is also called motor neuron disease.

Clinical presentation. Early symptoms are mild and can be easily confused with other diseases. Patients may only feel some symptoms such as weakness, flesh jumping, and fatigue easily, and gradually progress to generalized muscle atrophy and difficulty swallowing. Respiratory failure develops.

According to the clinical symptoms, it can be roughly divided into two types:

1.Limb-onset type.

Symptoms begin with progressive atrophy and weakness of the muscles in the limbs, and finally respiratory failure.

2.Bulbar onset.

Difficulty swallowing and speaking begins with early progression, and respiratory failure soon progresses.

In order to diagnose amyotrophic lateral sclerosis at an early stage, in addition to the neurological clinical examination, electromyography, nerve conduction velocity testing, serum special antibody testing, lumbar puncture cerebrospinal fluid examination, imaging examination, and even muscle biopsy are also required.

Motor neuron disease includes amyotrophic lateral sclerosis, progressive spinal muscular atrophy, primary lateral sclerosis, and progressive bulbar palsy. The course of the lesion is mostly the same for all types of motor neuron disease, and the main difference is the location of the lesion. Amyotrophic lateral sclerosis can be thought of as a representative of this group of diseases, while other types are its variants.

Amyotrophic lateral sclerosis (ALS) was synonymous with motor neuron disease in the early stages, specifically referring to a separate disease in which lower motor neuron damage followed by upper motor neuron damage. However, it was later discovered that there were two other variants in which only upper motor neurons were always involved during the course of the disease, the former being called primary lateral sclerosis, and the latter being called spinal muscular atrophy. However, some literature still uses MND to refer specifically to amyotrophic lateral sclerosis.

Most scholars are accustomed to divide motor neuron disease into three types: amyotrophic lateral sclerosis, primary lateral sclerosis, and spinal muscular atrophy, according to the different combinations of upper and lower motor neuron involvement. Amyotrophic lateral sclerosis shares a pathological basis with a variety of related diseases, including primary lateral sclerosis, ALS-dementia, ALS-associated frontal dementia, progressive spinal muscular atrophy, multiple system atrophy, and Lewy corpopathy. Pathological examination revealed that these diseases also contained ubiquitin-positive inclusions and transparent mass inclusions, but with a variety of clinical combinations due to injury to different anatomical sites.

Motor neurons feel much better when they take the Recovery Formula.