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The cause of pulmonary hypoplasia or hypoplasia is that at some stage of the embryonic development process, a disorder of lung development occurs, resulting in pulmonary hypoplasia and pulmonary hypoplasia. Most of the causes are born defects, with 50% to 80% of infants with congenital diaphragm clinically proven to die from pulmonary insufficiency.
Pulmonary hypoplasia or pulmonary hypoplasia can be divided into the following pathological types. 1.Pulmonary dysplasia, i.e., parenchymal dysplasia, is commonly characterized by underdeveloped lung tissue with congenital diaphragm, absence of pulmonary arteries, and spherical lung with bronchial malformations.
2.The lobes of the lungs are absent as well as one or more lobes, usually missing the right middle lobe and the right lower lobe. The mediastinum moves laterally, and the lateral lungs are associated with other malformations.
3.The absence of a lung may be due to a lung dysplasia leading to bronchial atresia, with no lung tissue and pulmonary vessels in the distance. The other part is bronchial stenosis and emphysema in distal lung tissue.
This is because when the body passes through the lateral bronchi into the lung tissue, which has insufficient blood vessels in the lungs, the main bronchi is insufficient, and the accessory lungs are insufficient. There are many more on the left than on the right.
Recurrent respiratory tract infections are the main cause of diagnosis and treatment. Patients with unilateral pulmonary hypoplasia often have mild dyspnea, decreased physical strength and endurance, and some patients may have hemoptysis due to circulation, collateral branches, systemic circulation, and respiratory tract infections that cause dyspnea, coarse hair, and breath sounds, and slow growth and development. If there is a malformation of the heart, bones, or other organs, there may be symptoms.
The patient's thoracic cavity is always deformed, nearly bilaterally symmetrical or symmetrical, with weak lateral breathing movements, weak or absent breath sounds, and percussion may be unaphonic, soft, or specific. Chest deformity is often the most obvious sign.
This symptom of pulmonary hypoplasia gradually develops lung function with age, and the specific time required varies from person to person. Recurrent respiratory tract infections are common during the course of the disease, and attention should be paid to prevention and promptly**. Keep the airway open, use antibiotic aerosol inhalation at any time and assist the child to pass sputum.
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Pulmonary hypoplasia is a congenital defect caused by a disorder in the development of the bronchi and lungs during the embryonic period, sometimes complicated by malformations of other organs.
In the case of unilateral lung hypoplasia, patients may have mild dyspnea and poor endurance and physical strength, while others may have a concomitant respiratory infection that worsens dyspnea, delays in growth and development, and is accompanied by deformities of the heart, bones, or other organs.
If the patient has no obvious clinical symptoms of pulmonary hypoplasia, it can not be done, if there are symptoms such as developmental delay or coughing up blood, lung infection, etc., it is necessary to intervene effectively under the guidance of a doctor, such as lobectomy.
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It is caused by physical causes, genetic causes, genetic causes, mental causes, and work reasons. So when we have this situation, we must go to the hospital for a physical examination.
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The reason for growth, the reason for the body, is related to genetics, is related to the development of the fetus, and is caused by illness.
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The main reason for Hu Bitian's pulmonary hypoplasia may be the reduction of the effective capacity of intrathoracic lung growth and development, and the most common reason is that the diaphragm on one side of the diaphragmatic hernia cannot be closed, and the abdominal viscera herniated into the chest cavity, thus affecting the development of the lungs.
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Congenital pulmonary hypoplasiaLung congenital hypoplasia can be divided into 3 categories according to the degree of occurrence: absence of one or both lungs in the lung; The undeveloped bronchial primordium of the lungs showed a terminal blind sac, and no pulmonary vessels and lung parenchyma were seen; Pulmonary hypoplasia may be seen in bronchial, vascular, and alveolar tissue, but decreased in number and/or volume. Patients may have concomitant pulmonary vascular and other malformations.
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In severe cases, death occurs immediately after birth. The main manifestations are dyspnea, even respiratory distress, and long-term recurrent respiratory tract infections, physical examination can show chest collapse on the affected side, reduced mobility, dullness on percussion, decreased or absent breath sounds on auscultation, and patients may be accompanied by other clinical manifestations of congenital malformations, such as renal insufficiency. Mild cases may have no obvious clinical symptoms and are only detected on a routine chest x-ray.
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1.Laboratory tests.
Arterial blood gases show hypoxemia while arterial carbon dioxide partial pressure is in the normal range.
2.Computed tomography (CT) scan of the chest.
In particular, 3D imaging scans can clearly show tracheobronchial malformations, and angiography and color Doppler can help visualize vascular (pulmonary arteries, venous azygous) malformations to distinguish other causes of atelectasis and bronchiectasis.
3.Flexible bronchoscopy.
Examination is helpful to visualize the bronchial lumen and obstruction.
4.Pulmonary function tests.
Pulmonary function is characterized by restrictive ventilatory dysfunction, decreased lung compliance, and increased airway resistance.
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