Inflammatory demyelination and infection What are the dangers of myelitis?

Inflammatory demyelinating disease is an acute attack or subacute damage to the central white matter of the disease, the acute phase is generally the use of hormones, while with anti-inflammatory drugs for symptomatic, generally speaking, is more complex, and the effect of the patient has a lot to do with the patient's physique, it is recommended that the patient is active.

1. Acute transverse myelitis is an acute progressive inflammatory demyelinating lesion of the spinal cord (demyelinating lesions: the nerve myelin sheath of acute demyelinating disease can be regenerated at a relatively rapid rate and to a more complete degree, although the regenerated myelin sheath is thinner, it generally has little impact on functional recovery. In chronic demyelinating neuropathy, the nerve may become thicker and there is axon loss due to the marked proliferation of regenerated Schwann cells with repeated demyelination and myelination. , most of which have been confirmed to be MS manifestations, are monophasic or chronic multiphasic **, clinical common disseminated myelitis, signs are asymmetrical and incomplete, rapid (hours or days) progression of paraplegia, truncal sensory impairment plane and sphincter dysfunction, paroxysmal tonic spasm and radicular pain can be seen in about 1 3 ** patients, but uniphasic course patients usually rarely occur.

Second, the age of onset is 5 60 years old, 21 41 years old, there are many children, both men and women can have the disease, acute transverse or disseminated myelitis and bilateral simultaneous or successive optic neuritis is the characteristic manifestation of the disease, in a short period of time continuous appearance, resulting in paraplegia and blindness, the disease progresses rapidly, can be relieved**.

3. Most patients with NOM have a uniphasic course of disease, 70 cases have paraplegia within a few days, about half of the patients have total blindness in the affected eye, and a few patients have a ** course of disease, of which about 1 3 have paraplegia. About 1 to 4 vision is affected, and the interval between clinical events is months to six months, and there can be multiple **isolated onions and myelitis within the next 3 years.

Only when the lesion is regenerated and repaired correctly, can the body produce viral antibodies no more**. Otherwise, there is a possibility that the virus is latent at any time. The cause of demyelinating myelitis is caused by immune-specific viruses invading nerves, which can be ** (**the sooner the recovery, the better), but non-hormonal healing, **inappropriate and prone** and delayed multiple sclerosis.

The reason is that the hormone and the original affected neurological insufficiency lead to low immunity in the human body and the virus is latent. In the early stage, most of them are hormones and nutrition, but the efficacy is difficult to control, because the disease leads to myelin loss and nerve re-damage, so that the neurological symptoms are further aggravated, and the damaged nerve is delayed for too long, and the late ischemic degeneration and multiple sclerosis are delayed, so the recovery is more difficult, and spastic paralysis and secondary complications are life-threatening in severe cases. No information can only provide you with a theoretical ** plan:

After the normal hormone is controlled, it can be replaced with natural hormones while the hormone is reduced, which is non-toxic to the human body, and the combination of traditional Chinese and Western medicine can enhance the body's immune function and improve the body's ability to resist diseases. Nourish the nerve, expand the microcirculation so that the damaged residual nerve can get sufficient blood supply, and prevent the disease from progressing. At the same time, the nerves are excited, and the nerves of paralysis and shock are activated so that the body produces viral antibodies and no longer has the disease, so as to achieve the regeneration and repair of the lesion and obtain the recovery of nearly normal function.

In terms of maintenance, you need a complete plan, and if you need help, please send magnetic resonance ** for your guidance.

Demyelination occurs when the myelin sheath of the axons of the cerebrospinal cord is lost. There are two categories: myelin destruction; Disorders of myelination. Spinal inflammatory demyelinating diseases are currently considered to be immune-mediated diseases, and their clinical features are:

The patients were children and young adults;

Acute onset, common history of cold, fever, infection, rash, vaccination, cold, childbirth or surgery 1 month before the illness;

A thorough neurological examination often reveals evidence of other central nervous system involvement in addition to spinal cord symptoms and signs;

cerebrospinal fluid examination is positive for oligoclonal bands;

Electrophysiology and MRI can detect some subclinical lesions in the brain; MRI reveals abnormal signals of white matter in the brain. Some myelittic demyelinating lesions present:"Pseudoneoplasia"Manifestations, the MRI shows a mild mass effect, mild edema around it, and there may be patchy hemorrhagic signals, which can easily be misdiagnosed as spinal cord tumors. At present, the effect of internal medicine drugs** is not good.

Therefore, neurosurgical intervention is inevitable and necessary.

Demyelinating myelitis is usually acute multiple sclerosis (MS) spinal type, and the clinical presentation is similar to that of postinfectious myelitis, but the progression is slow, and the disease usually peaks within 1 to 3 weeks. The prodromal infection may be subtle, usually incomplete transverse lesions, with weakness or paralysis of one or both lower extremities, with numbness, the level of sensory impairment is inconspicuous or two-plane, and urinary and fecal disorders are present. Evoked potentials and MRI may reveal lesions elsewhere in the CNS.

Myelitis will cause spinal cord edema and cause spinal cord demyelination and nerve dysfunction and paraplegia or incomplete paraplegia, Western medicine to hormone Tongyi and protein ** controllable disease secondary to serious spinal cord damage but can not restore the nerves, the recovery of its nerve function is to rely on its own repair, the other is to rely on the drug to activate the nutrition and excitement of the nerves, such as the onset time is too long, the spinal cord will be due to ischemia for too long secondary ischemic softening and atrophy, the recovery of the disease is more difficult, and will lead to permanent spastic paraplegia.

I don't provide information, I can only provide you with a theoretical ** plan, congratulations: traditional Chinese medicine nutrition, nerve enhancement, improve the blood circulation of spinal cord microcirculation, so that the affected paralyzed nerves can get adequate blood supply, and prevent secondary ischemic involvement of neurodegeneration. At the same time, the nerve cells of paralytic shock can be activated by the stimulation of Nerve Regeneration Pill in order to obtain the best recovery of various functions such as movement and stool, and reasonable functional exercise is required to help the recovery and improvement of various functions.

Guidance should be sent at the time of onset and the most recent magnetic resonance**.

The first point is that in the acute stage, relatively large doses of glucocorticoids are usually used**, and the application of glucocorticoids can inhibit abnormal inflammatory responses and reduce spinal cord injury, which is still important for controlling the disease. When using high-dose glucocorticoids, it is necessary to pay attention to adverse reactions, such as the disorder of fat metabolism in the body, leading to osteoporosis and even necrosis of the femoral head. Some patients may be treated with gamma globulin in the acute phase.

Second, if the patient has spinal cord demyelination due to neuromyelitis optica, a small amount of glucocorticoids is usually needed to maintain it during remission, in combination with some other immunosuppressants.

The third point is to pay attention to the ****, ** training still plays a certain role in the recovery of spinal cord injury.

The symptoms of demyelinating myelitis ** are similar to the symptoms of the first episode, you now feel numb in your hands after catching a cold, there is a possibility of **, it is recommended to go to the hospital for a follow-up examination, in time**.

1. Medical history and symptoms:

Most young adults have onset of disease, and there are symptoms of respiratory infection within two weeks before the illness, or a history of epidemic or vaccination vaccination. There are triggers such as cold, overwork, and trauma. The first symptoms are numbness and weakness in both lower extremities, back pain and banding sensation in the corresponding part of the lesion, urinary retention, and fecal or fecal incontinence.

2. Physical examination found signs of spinal cord transverse injury:

1.Early causes"Spinal shock phase"Manifests as flaccid paralysis, in which the limb or body innervated below the lesion site presents with paralysis of the upper motor or meridian element after the shock period (3-4 weeks).

2.Sensory depth below the level of the lesion is absent, and some may have hyperesthesia zones in the plane of the lesion.

3.Vegetal or menstrual disorders: manifested as slag and urine retention, large residual urine and filling urinary incontinence, and fecal or fecal incontinence. The shock phase is followed by reflex bladder or bladder, large or constipated knots, and priapism or erection of the pubic or shaft.

3. Auxiliary inspection or inspection

1.In the acute phase, peripheral white blood cell counts are normal or slightly elevated.

2.Cerebrospinal fluid pressure is normal, and some patients have mildly elevated white blood cells and protein, and normal levels of sugar and chloride.

3.MRI of the spinal cord shows hypersolusion or thickening of the spinal cord at the site of the lesion, and the signal is abnormal.

4. If it is differentiated, it needs to be distinguished from acute epidural abscess, spinal tuberculosis, spinal metastatic tumor or tumor, visual or transmyelitis, spinal cord or blood.

It is the protective layer of the nerve that is infected by inflammation. Also known as demyelinating disease. Demyelinating disease is an acute onset or subacute damage to the white matter of the central nervous system, the sooner the recovery, the better, Chinese medicine called impotence syndrome, rheumatism, prickly heat, dizziness, bones, dizziness, blue blindness, paralysis is called body inertia, spinal cord symptoms are severe, the brain is seen in chronic subacute lesions damage nerves resulting in dysfunction after magnetic resonance can be found, if the second ischemic degeneration of the damaged nerve is delayed, multiple sclerosis will occur, When the disease is severe, it can invade the anterior horn cells of the spinal cord and the nerve nucleus of the brainstem, as well as the pyramidal cells of the motor cortex of the brain, which is life-threatening, and is mostly caused by abnormal genetic immunity or viral infection.

In the early stage, most of them are hormones and nutrition, but the curative effect is difficult to control, and the disease leads to neurological damage caused by myelin detachment, and when the neurological damage is severe, the axonal damage is further aggravated. Secondary exacerbation of the disease.

**Solution:**In addition to normal hormones**, it can be gradually replaced with natural hormones and is non-toxic to people***. It should enhance the body's immune function and improve the body's ability to resist diseases.

Nutrition of the nerves, the combination of traditional Chinese and Western medicine to expand the microcirculation so that the damaged residual nerves can get sufficient blood supply, to prevent the disease from further developing. At the same time, the nerve regeneration drug is used to stimulate the nerves, activate the nerve cells that are paralyzed and shocked, so that the body can produce viral antibodies and no longer achieve the purpose of regenerating and repairing damaged nerves to obtain the best recoveryIf you need help, please send magnetic resonance for your guidance.

Tips to see the must-read and successful cases of nerve injury in my space will help you.

Most of the nutritional neurological drugs (methylcobalamin, methylprednisolone, potassium chloride, citicoline, purine tablets), hormonal drugs (prednisone, prednisone), gam-globulin shock, etc., reduce the condition but limit the ability of the immune system to improve itself and actively resist the molecular structure of the cause of the invasion of the body.

Neurological disorders require TCM**.

Therefore, according to the corresponding **, if the patient is ischemia and hypoxia caused by atherosclerosis.

It is the protective layer of the nerve that is infected by inflammation.

If the secondary ischemic transformation of the nervous system is delayed, multiple sclerosis will be produced, and when the disease is more serious, it can invade the anterior horn organ of the spinal cord and the nerve nucleus of the brainstem and the cortical cone mechanism of the human brain, which seriously endangers the patient's life, and produces a heavy psychological state and economic development work pressure on the patient and the patient's home.

The main manifestations, such as sluggishness or resolution, are called demyelinating disorders. Structure of neuronal cells.

Multiple demyelinating lesions are mostly caused by the white matter of the brain, so people also call it multiple demyelinating lesions of the white matter.