How does bilirubin encephalopathy develop? What are the after effects of bilirubin encephalopathy?

Bilirubin encephalopathy is caused by a greater production of bilirubin than the amount of bilirubin excreted. The sequelae of bilirubin encephalopathy include the inability of the eyes to move properly, hearing loss, bleeding teeth, cerebral palsy, and frequent drooling.

It is likely to be caused by intrauterine asphyxia, infection, or hemolytic factors. It can cause cerebral palsy, it can also cause twitching of the hands and feet, and then it can also lead to epilepsy, which can affect the baby's mental development.

It is likely to be hemolytic disease, it may be an infection, and it is also possible that it is dehydrated. It can cause cerebral palsy, it can also lead to mental retardation, it can cause epilepsy, and it can also cause cramps in the hands and legs.

Some newborns have jaundice with high blood bilirubin concentration, which may lead to jaundice in the central nervous system, causing irreversible damage to central nervous system cells and causing sequelae such as visual and hearing impairment.

Causes of bilirubin encephalopathy: The value of bilirubin in the blood is related to the degree of development of the blood-brain barrier. If the concentration of unconjugated bilirubin in the blood is high, or the child is a premature infant, the blood-brain barrier is not well developed, or the fetus is infected and hypoxic in the mother's womb, the unconjugated bilirubin in the blood may pass through the undeveloped blood-brain barrier and enter the cranial nerves to cause a series of damage.

Because the prognosis of kernicterus is relatively poor and there are many sequelae, prevention is emphasized. In the jaundice stage, it is irradiated with blue light as soon as possible or the primary disease is yellowed.

It is because of neonatal jaundice, that is, the concentration of bilirubin in the serum is too high, and after accumulating to a certain extent in the body, it will cause most of the xanthrorubin to be based on unconjugated bilirubin, which will cause bilirubin encephalopathy, bilirubin encephalopathy, also known as coxanthropathy persimmon hyperunconjugated bilirubinemia, free bilirubin is deposited in the basal nerve nucleus, thalamus, thalamus, mandibular and cerebellum through the blood-brain barrier, inhibiting the use of oxygen in brain tissue, resulting in brain damage.

Newborns with severe jaundice have a high concentration of bilirubin in the blood, which can damage central nervous system cells, and affect hearing and vision. Bilirubin encephalopathy is a condition in which bilirubin levels are severely higher than normal, impeding nerve function.

a.Drowsiness, hypotonia.

b.Convulsions, increased muscle tone.

c.The muscle family history has normal tension and normal body temperature.

d.The spasms are reduced and the breathing is better.

e.Cerebral palsy, mental retardation.

Correct Answer: a

Consider cerebral palsy in the context of what you describe. Symptoms of cerebral palsy: crying, increased muscle tone, difficulty feeding. **Bilirubin encephalopathy, asphyxia and hypoxia, and infections are common.