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The cancer cells spread quickly, and it will affect their normal life, and the impact on their life is relatively large, and there will be a lot of painful symptoms, and their mood is not particularly good. Of course, there is ***, *** is that it may cause hair loss.
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The condition is more severe, basically all patients in the middle and advanced stages, and the body cannot tolerate the pain; Targeted drugs have a certain amount of energy, which can cause hair loss and damage gastrointestinal function.
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The chance of cancer patients taking targeted drugs** is very small, but for cancer, the effect of targeted drugs** is still relatively good, and patients with advanced cancer can use it, which can prolong the survival time of patients; Targeted drugs are not ***, and they will make some patients achieve the best effect.
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Surgery** is notCancerThe ultimate killer,ChemoradiotherapyIt's not the ultimate killer of cancer, and chemoradiotherapy isn't the ultimate killer of cancerInterventional surgeryIt is not the ultimate killer of cancer, and endocrine ** is not the ultimate killer of cancerTargeted drugs.
It's not the ultimate killer of cancer, and now it's the ultimate killer of cancer, and it's not the ultimate killer ......of cancerThere is no one way to diagnose and treat cancer! Since cancer is a complex group of diseases, it is far more complex than everyone thinks, and there is no need to expect a single treatment to solve all cancers. Each method of diagnosis and treatment has its advantages and disadvantages, only some of them deal with part of the problem of cancer, and it is unlikely to completely eliminate all the problems, so it is unlikely to be the ultimate killer of cancer.
Targeted drugs have not yet been able to completely eradicate malignant tumors.
After a period of treatment, it is accompanied by drug resistance to malignancy.
In the end, the treatment was unsuccessful. Drug resistance of malignant tumors is also one of the highest key points in the pharmaceutical industry at this stage, and the reproduction system of cancer cells that produce drug resistance will be more complicated, and finally there will be no drugs to use. As it stands, targeted drugs** can still be called sarcomas.
The ultimate killer is the diversity of the drug resistance system of malignant tumors, which is constantly evolving with the promotion of medical treatment, and the efficiency of people's drug development has not kept up with the rate of drug resistance of malignant tumors.
Targeted drugs**, are in the somatic cells.
At the level of molecular structure, for the domains of carcinogens that have been identified.
(This site can be a protein molecule in the internal structure of cancer cells, or it can be a gene fragment). The drug will specially select the carcinogen domain to closely combine to produce efficacy, so that the cancer cells will die non-specifically, and will not spread to the normal cell tissues around the malignant tumor, so molecular targeting** is also called "biological missile".
After treatment with targeted drugs, patients can sometimes resolve all the solid tumors in the lungs, thereby improving the survival rate of patients. After the application of targeted drugs suitable for patients, the 3-year survival rate exceeds 50%, which is also chemotherapy and radiotherapy.
and other practical effects that cannot be achieved by traditional healing.
At this stage, targeted drugs are far from the relative height of "the ultimate killer of cancer treatment". Because cancer, like the epidemic, is in a changing law from beginning to end. With the interaction of cancer cells and drugs, through the survival of the fittest, cancer cells that meet the conditions will be solved and disappeared by targeted drugs, but malignant tumors will grow new gene mutation domains, and new loci can not be greatly harmed by targeted drugs, and then sweep growth and developmentand resistance to the initial targeted drugs, and then the targeted drugs become dispensable, so that cancer cells can survive and progress in the fight against the growth and decline of targeted drugs.
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Targeting** is more precise. Targeting** can also have a rapid onset of action, and the immune** effect is relatively long-lasting; Genetic testing is usually followed by genetic testing before deciding on a specific chemotherapy treatment.
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I think it is more efficient to use targeted drugs**, because targeted drugs are targeted drugs that can kill cancer cells very well.
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I think targeted drugs are better. Immunomedicine can only improve immunity, but targeted drugs can fight off those cancer cells.
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It is necessary to choose different ways according to different situations, and the suitable methods for each cancer patient are different, and cannot be said one-sidedly.
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Drug dose: The maximum tolerable dose of cytotoxic drugs has been achieved before the inhibition of large malignant tumors, and the straight slope of the dose-utility curve before the larger tolerated dose is relatively largeTherefore, clinical medicine should choose its maximum safe dose. However, the toxicity of targeted ** drugs is relatively light, and the inhibitor saturation state has been achieved before the maximum dose is achieved, giving full play to the highest inhibitory effect.
At this time, if the dose is increased again, the efficacy is not significantly enhanced, but the excess toxicity is increased. Therefore, the dosage of targeted ** drugs should be the best molecular biology dose.
Timeline of drug application: Inhibitor inhibition is reversible in most cases, and malignant tumors have a regenerative and reusable system. Therefore, in order to achieve continuous manipulation of tumor cells, the targeted drugs used in medicine at this stage are generally used continuously until the malignant tumor progresses or the patient can no longer bear it.
Therefore, a comprehensive and high priority should be given to the effectiveness of the patient, noting that the constant occurrence of *** even if the level is too high will cause confusion in the patient's daily life and jeopardize the patient's quality of life. Continuity should be proactively addressedBefore the application of targeted drugs, it is necessary to have a clear distinction between the diagnosis of the patient and the installment payment, and then distinguish whether the patient meets the requirements of the application of molecular structure targeted drugs, and at the same time, it is necessary to select the type of targeted drugs, the time and cost of treatment and related common problems.
.During the period of application of targeted drugs, it is indispensable to prevent and alleviate the adverse reactions related to targeted ** drugs, such as rashes, which can be cleaned with honeysuckle water gargle and mouth rinse. If numbness occurs during the course of taking the medicine, you should go to the hospital for medical treatment as soon as possible.
Differences between targeted drugs** and basic chemoradiotherapy. The big difference between the targeted ** drug and the basic anti-tumor drug lies in its mechanism of action: the basic anti-tumor drug gives full play to the growth and development of active somatic cells according to the destructive force, but it cannot accurately identify cancer cells, so it destroys cancer cells and will also affect normal somatic cells, so it forms a great ***.
The targeted drug is developed and designed for the abnormal molecular structure and genetic genes of malignant tumors, which can be fused with abnormal inhibitors unique to cancer cells or institutions, and then destroy cancer cells or block their growth and development. When antineoplastic drugs enter the body, they can interfere with normal somatic cells and harm cancer cells. Although both somatic cells can die due to irreversible damage to somatic cells caused by chemotherapy drugs, normal somatic cells have a strong ability to repair and survive again if the damage is small.
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Targeted ** mainly has the advantages of significant efficacy, small and easy to use, and it is necessary to pay attention to whether the drug is in line with the patient's condition, and prevent and treat the drug ***.
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This method is more targeted, and will be fixed to a certain point, and the response is very small. It is important to observe and treat in a timely manner, and also pay attention to the binding site.
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kind of pertinence, fixed to a certain point, with few side works. Be sure to observe the treatment in time, and also pay attention to the binding part.
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