What are the symptoms of spinocerebellar degeneration?

Since cervical myelopathy is not as common as other types of cervical spondylosis, patients naturally take it lightly. But in fact, this idea is very dangerous, because the disease is not inferior to other types of cervical spondylosis. Next, I will tell you about the symptoms of this disease, and I hope you will prevent it as soon as possible.

What are the symptoms of spinal cord lesions.

Symptom 1: In the early stage, the patient will feel tightness and weakness in the lower limbs, the pace is chaotic when walking, and the legs are very heavy as if they have been filled with lead. If the disease lasts too long, the patient may also have a limp gait and walk more difficult.

Soreness and weakness may also occur in the upper limbs, and sometimes objects may slip out of the hands. The limbs feel like an electric shock, and accompanied by headache, dizziness and other discomforts, patients may sometimes have frequent urination and urgency, incomplete urination, and weak defecation.

Symptom 2: After the advanced stage, patients may develop severe symptoms such as fecal incontinence and limb paralysis. There is an obstacle to the movement of the neck, especially when it is extended backwards.

There is varying degrees of tenderness in the neck, and there is sensory deficit below the compressed spinal cord segment.

Symptom 3: X-ray of the cervical spine shows the disappearance or reverse arch of the cervical spine, narrowing of the intervertebral space, anterior slippage of the vertebral body and articular process, and labial hyperplasia at the edge of the vertebral body. CT or MRI of the cervical spine shows a herniated cervical disc that is posteriorly protruding and compressing the spinal cord.

Precautions. Don't relax your vigilance because cervical myelopathy is uncommon, because the disease is very harmful to patients, once the symptoms appear, it is necessary to actively carry out **, the sooner ** the effect will be better, and the probability will be greater.

Weakness of the body's limbs may occur.

There are many causes of spinal cord lesions, such as myelitis, spinal cord compression, spinal cord vascular disease, including ischemic vascular disease, hemorrhagic vascular disease, etc., each spinal cord lesion will cause three main symptoms: movement disorders, sensory disorders, and autonomic disorders.

The severity of symptoms varies depending on the degree of injury. It may have the following manifestations. During spinal cord shock, it is manifested as flaccid paralysis below the level of injury, loss of movement, reflexes and sphincter function, loss of sensory plane and inability to speak orally, and gradually evolves into spastic paralysis after 2-4 weeks, manifested by increased muscle tone, hyperreflexia of tendons, and pathological vertebral tract signs, thoracic spinal cord injury is paraplegia, cervical spinal cord injury is quadriplegia, and quadriplegia of upper cervical spine injury is spastic paralysis, Quadriplegia with lower cervical spine injury is due to the destruction of the swollen part of the spinal cord and neck, and the upper limbs are flaccid paralysis, and the lower limbs are still spasmodic paralysis.

Inflammation, after the virus invades the spinal cord, the delay is improper, and the qualitative changes of the spinal cord are called spinal cord degeneration, and the necrosis of the spinal cord that continues to deteriorate and degeneration will lead to spastic high paraplegia, and whether it can be recovered must be correct.

Due to the lack of understanding of the condition, it is necessary to explain in detail the age of onset, the time of onset, the exact location of the onset, the results of the examination (original magnetic resonance**), the detailed symptoms of the current condition, and the more detailed the better, which is of great help to the qualitative analysis, evaluation and ** of the condition, to see if it can help you. Inflammation, viruses, edema, trauma, many causes can cause spinal cord damage, spinal cord degeneration is the onset of ** delay after the onset of ischemic spinal cord degeneration or spinal cord atrophy scarring necrosis. If you need to send an MRI** to guide you.

It is suggested that the continued deterioration of inappropriately delayed ** can lead to spastic painful high paraplegia. Myelitis is an inflammatory disease that destroys the function of the spinal cord, and can be divided into acute myelitis and subacute myelitis. The lesion usually occurs in the thoracic spinal cord, and if the lesion invades only a few segments of the gray and white matter of the spinal cord, it is called transverse myelitis.

The exact ** is not well understood, but it may be caused by a viral infection or due to the body's autoimmune response after a viral infection. Trauma, cold, and overwork can be predisposing factors. It must be differentiated from Guillain-Barré syndrome, anterior spinal artery occlusion, and acute spinal cord compression due to epidural abscess, haematoma, or tumor.

Take the medicine regularly and quantitatively according to the doctor's instructions, do not stop or reduce the dose by yourself, and report nausea, abdominal distention, and black stool during the medication. It is also necessary to pay attention to strengthening nutrition, enhancing the body's immunity, preventing colds, and avoiding the recurrence of diseases caused by colds. Strengthen the active or passive functional exercise of the limbs, so that the paraplegic limbs can maintain the functional position of the limbs and obtain the functions of the limbs through the cells.

It's very important to remember the early days. Here are some recommendations from experts for patients** [General**] 1 Strengthen nutrition and eat foods that are high in protein and vitamins. 2 Keep warm and avoid cold.

3 Keep ** clean and dry, keep the sheets dry, soft and flat; Turning over frequently; The paralyzed limb remains in a functional position and passive movements are performed as soon as possible; Place air rings or soft pads on the sacrum, ankles, shoulder blades, etc., and massage frequently, followed by safflower alcohol. 4. Keep the airway open and remove respiratory secretions in time. For people with dyspnea, oxygen should be given as soon as possible; If the cough is weak and cannot discharge sputum, the tracheotomy should be made in time, and artificial ventilator should be used if necessary.

For patients who have been on artificial ventilators, sputum suction should be timely, not more than 15 seconds, and the interval between the two should be 3 5 minutes; Humidified solution should be dripped into the trachea, 2 5ml times, 1 time every 15 minutes; The inner cannula is replaced 2 times a day, the outer cannula is replaced once every half a month, and the gauze pad is replaced 2 times a day. 5 For patients with urinary retention, the urinary catheter should be kept and opened once every 3 to 5 hours. And rinse the bladder with O 1 furacillin or 3 boric acid solution, 1 2 times a day, 250ml times, keep for half an hour and then release.

There are several reasons why spinal cord lesions can lead to spinal cord lesions

1. Spinal cord diseases: spinal cord lesions can be caused by vertebral fractures, intervertebral disc herniation, spinal stenosis, spinal tuberculosis, spinal primary tumors and metastases.

2. Extraspinal lesions in the neuraxial canal, such as extramedullary tumors such as neurofibromas and meningiomas, spinal cord vascular malformations, epidural abscesses, etc.

3. Intraspinal cord lesions, such as spinal cord tumors, tuberculoma, hemorrhage, etc.

Spinal cord lesions mainly cause symptoms of spinal cord compression, which are mainly manifested by loss of pain and temperature sensation, while the sensation of touch, vibration, and position is normally preserved. Most patients present with decreased muscle strength of the limbs, often manifested as loss of head strength in one or both muscle malfunctions, and some patients also have muscle atrophy.

Syringomyelia: 1. Congenital developmental abnormalities. Because syringomyelia is often complicated by other congenital anomalies such as skull base depression, cerebellar tonsillar herniation, spina bifida, scoliosis deformity, etc., and often has a tendency to develop in families, it is believed that syringomyelia is related to genetic factors.

2. Cerebrospinal fluid dynamic abnormality. There is no unified understanding of the myelomyelia and its pathogenesis, and congenital malformations of the craniocervical junction combined with syringomyelia are common in clinical work, and syringomyelia is mostly found in the cervical and upper thoracic spinal cords. It is thought that cerebellar tonsillar herniation impairs the circulation of cerebrospinal fluid in this area, resulting in spinal cord damage and the formation of syrinx.

In addition, spinal cord injuries and tumors can cause syringoma to form. 3. Abnormal blood circulation. Due to the abnormal blood circulation of the spinal cord, such as compression of the anterior spinal artery or obstruction of spinal venous return, it causes ischemia, necrosis, liquefaction of the tissues in the spinal cord, and finally forms a syrinx.

Syringomyelia is a chronic, progressive condition of the spinal cord. **It is not well understood, but the lesion is characterized by the formation of a tubular cavity in the spinal cord (mainly gray matter) and hyperplasia of glia (non-nerve cells). It usually occurs in the cervical spinal cord.

When the lesion involves the medulla oblongata, it is called syringomyelia.

Hello! **: Poliomyelitisvirus is a genus of enteroviruses in the family Pesteviridae.

Under an electron microscope, the virus was observed to be a small spherical shape, with a diameter of 24 30 nm, and a round granular shape. It contains a single strand of ribonucleic acid, and the nucleic acid content is 20% to 30%. The core shell of the virus is composed of 32 captogranules, each of which contains four structural proteins, namely VP1, which has a special affinity for human cell membrane receptors and is related to the pathogenicity and virulence of the virus.