Can you die if Raynaud s disease is severe, and what happens when Raynaud s disease is severe?

Generally, it does not induce death, and the disease is easy to cause paroxysmal strong contraction of acral arterioles to cause acral ischemia, and long-term changes in the condition are easy to induce ischemia.

Raynaud's syndrome"It can be associated with many diseases, and severe Raynaud's syndrome can have serious consequences such as gangrene and loss of finger gangrene, which can lead to impaired limb function. In most cases, patients have long-term insufficient blood supply to the extremities, resulting in acromegaly and dysfunction, which seriously affects the work and quality of life of patients, and brings a lot of inconvenience and pain. At the same time, if it coexists with other diseases, it also affects the ** of other diseases to a certain extent.

Therefore, for early Raynaud's syndrome, enough attention should be paid to early detection and early detection to avoid adverse consequences.

Raynaud's syndrome is a group of syndromes in which the fingers (toes) turn pale, purple and then flush due to cold or emotional agitation. If there is no specific cause, it is called idiopathic Raynaud's syndrome; Secondary to other disorders is called secondary Raynaud syndrome.

In severe cases of Raynaud's disease, there are ulcers or gangrene on the fingertips, mild atrophy of muscles and bones, and if there is no timely **, it will also cause other more serious cardiovascular and cerebrovascular complications.

In severe cases, there will be symptoms of pale and purple fingers, and there will be long-term continuous arterial spasm, resulting in organic narrowing of the arteries, and some irreversible lesions, such as severe acral ischemia, and even open ulcers or necrosis.

When this disease is severe, it will cause gangrene of the fingers or toes, that is, slowly necrosis and blackening, and then gradually fall off, and the fingers will be shortened, which is really terrifying, and it will also cause heart failure, shortness of breath, and breathing will become more and more difficult.

It will show tissue necrosis, and there will also be limb pain, which can also lead to anemia, and at the same time, it will also reduce the body's immunity, and it will also lack oxygen, which will also cause slow growth of toenails.

Raynaud's syndrome occurs more often in women than men between the ages of 20 and 40. The onset is slow, starting in winter, and the duration is short, and the onset can be triggered by cold or emotional agitation. It is usually symmetrical in both hands and fingers, but also in the toes.

The hands and feet are cold, numb, and occasionally painful during the attack. Typical seizures are bounded by metacarpophalangeal joints, and the fingers become cold, pale, purple, and then flushed. In the later stages of the disease, hair on the back of the fingers disappears, nails grow slowly, roughly, and deformed, atrophied, thinned, and tightened (scleroderma fingers), sores form around the fingertips or nail beds, and infections can occur.

The main symptom is discoloration of the fingers or other affected areas. Normal skin color changes from white to blue and red, and the rate of change varies depending on the temperature of the affected area. There is usually no pain, but there is numbness and a "pins and needles" sensation in the affected area.

Raynaud's disease progressively worsens at a very slow rate. The Raynaud's phenomenon worsens rapidly. In the advanced stages of the disease, the skin and flesh of the affected part of the patient atrophy, and when the tissue is damaged, small ulcers can form due to the tissue not getting enough blood**.

1. Microcirculation changes: Raynaud's disease generally does not change significantly in the early and middle stages, and the vast majority of severe cases show telangiectasia, ring-shaped, hair-like or irregular flexion.

2. Changes in the digital artery: In the late stage of Raynaud's disease, due to nutritional disorders, the intimama of the digital artery proliferates, the elastic membrane breaks and the muscle layer thickens, forming thrombosis, which is not easy to distinguish from occlusive vascular disease.

3. Avascular necrosis of the fingers (toes): the small arterioles at the end of Raynaud's disease are occluded, resulting in the avascular necrosis of the fingers, and in severe cases, the fingers (toes) can be flattened and gangrene, and the distal phalanx can be necrosis due to ischemia, absorbed and dissolved, and the phenomenon of shortening or amputation of the fingers may occur.

If Raynaud's syndrome is reversed and the disease progresses gradually, it will bring some harm. For example, it can lead to the gradual atrophy of the subcutaneous tissue at the end of the fingers, the gradual disappearance of the fingers, the dryness of the fingers, and the repeated reversal of the author, and the affected parts can be dystrophied. In addition, there may be severe manifestations, such as ulcers on the fingers, gangrene, or shortening of the fingers.

The author may also have some organ damage, especially in some patients secondary to connective tissue disease, who often have severe ischemic symptoms. In addition, if the patient does the opposite, there may also be lung lesions, heart damage, kidney lesions, etc. So pay attention to the disease.