What are receptor blockers? What are the receptor blockers?

Receptor blockers can selectively bind to adrenal receptors, do not excite or weaken the activation of adrenergic receptors, but can block the corresponding neurotransmitters and drugs from binding to the receptors, thereby producing anti-adrenergic effects.

1 Receptors are mainly distributed in vascular smooth muscle (such as **, mucosal blood vessels, and some splanchnic blood vessels), causing vasoconstriction when excited; 1 receptors are also distributed in the pupillary maximus muscle, and when excited, the pupil expands and the pupil dilates.

Receptor blockers are a kind of antihypertensive drugs, including betalux, bisoprolol fumarate, etc., which have the effect of slowing down the heart rate and reducing myocardial contractility. Hurry up and click ****.

Receptor blockers can be divided into 3 categories:

1. Non-selective receptor blockers, blocking 1 and 2 receptors at the same time, such as propranolol, etc.;

2. Selective receptor blockers, with little or no effect on 2 receptors, such as bisoprolol, etc.;

3. Block 1 and receptors, such as carvedilol.

The 2015 Guidelines for the Diagnosis and Diagnosis of Acute ST-Elevation Myocardial Infarction state that if there are no contraindications, receptor blockers should be routinely used within 24 hours and used for a long time ( , b). The 2012 US AHA Guidelines for Stable Coronary Artery Disease recommend that receptor blockers should be used in patients with heart failure ( , A), after myocardial infarction and angina pectoris ( , B), and for patients with hypertension and coronary heart disease, ACE inhibitors or receptor blockers can be preferred.

Receptor blockers can be divided into 3 categories:

1. Non-selective receptor blockers, blocking 1 and 2 receptors at the same time, such as propranolol, etc.;

2. Selective receptor blockers, with little or no effect on 2 receptors, such as bisoprolol, etc.;

3. Block 1 and receptors, such as carvedilol.

The 2015 Guidelines for the Diagnosis and Diagnosis of Acute ST-Elevation Myocardial Infarction state that if there are no contraindications, receptor blockers should be routinely used within 24 hours and used for a long time ( , b). The 2012 US AHA Guidelines for Stable Coronary Artery Disease recommend that receptor blockers should be used in patients with heart failure ( , A), after myocardial infarction and angina pectoris ( , B), and for patients with hypertension and coronary heart disease, ACE inhibitors or receptor blockers can be preferred.

Receptor blockers can selectively bind to adrenal receptors and do not excite or weaken the agonistic adrenergic receptors, but can block the corresponding neurotransmitters and drugs from binding to the receptors, thereby producing anti-adrenergic effects.

Answer] :d receptor blockers can make bronchial smooth muscle contract and increase airway resistance, so in patients with bronchial asthma or chronic obstructive pulmonary disease, it can aggravate or induce an acute attack of asthma when there is lead touching, but this effect has little impact on normal people. Selective <>

Receptor blockers have a weaker effect. However <>

Vascular smooth muscle contraction caused by receptor blockers can prevent and treat migraine attacks. Therefore, D is selected for this question.

Receptor blockers.

Mainly used for high blood pressure. The efficacy in heart failure and coronary heart disease has not been proven. Receptor blockers (such as clonine) that act on the center are rarely used due to their obvious characteristics.

Receptor blockers that mainly act on the periphery are commonly used pizosin, doxazosin, urapidil, etc.

Non-selective receptor blockers, such as phenolbenzamine;

Selective 1-receptor blockers, such as afuzosin, prazosin;

Selective long-acting 1-receptor blockers, such as terazosin, doxazosin;

Subtype-selective 1-blockers, such as the superselective 1a blocker, tamsulosin.

A new generation of 1A and 1D receptor dual blockers, nafoperadil.

Answer] :d receptor blockers can promote spasm of bronchial smooth muscle, which is mainly to induce asthma.

Summary. Receptor blockers (French: bêta-bloquants;English:

Beta blockers, also known as beta blockers, receptor blockers, receptor blockers, receptor antagonists, or blockers, are a class of drugs used to prevent arrhythmias, prevent a second heart attack after a heart attack (secondary prevention), and in some cases high blood pressure.

Receptor blockers (French: bêta-bloquants;English Mountains: Beta Blockers), also known as beta blockers, receptor blockers, receptor blockers, receptor antagonists or blockers, is a class of drugs used to prevent arrhythmias, prevent secondary heart attacks after the onset of cardiac disease (secondary prevention) and in some cases to improve high blood pressure.

Receptor blockers are a type of drug that selectively binds to adrenergic receptors and antagonizes the agonistic effects of neurotransmitters and catecholamines from the hood. Adrenergic receptors are distributed on the effector cell membranes innervated by most of the sympathetic nerve fibers after the ganglion, and their receptors are divided into three types, which can be stimulated to cause increased heart rate and cardiac muscle contractility, bronchiectasis, vasodilation, visceral smooth muscle relaxation, etc., and lipolysis. These effects can be blocked and antagonized by receptor blockers.