What is pre excitation syndrome type A, what are the harms, and is it necessary to treat it?

Pre-excitation syndrome (WPW syndrome) is an electrocardiographic manifestation of abnormal conduction of the heart tract, if the landlord does not have a tachycardia attack (that is, suddenly feels that his heart is beating so fast), or has occasional attacks but the symptoms are mild, there is no need for **, if the tachycardia episodes are frequent with obvious symptoms, it is necessary, **methods include drugs and catheter ablation. I still recommend that the landlord go to the cardiology department of a big hospital to have a look.

Preexcitation is an abnormal phenomenon of atrioventricular conduction in which impulses travel down through additional channels to prematurely excite part or all of the ventricles, causing premature activation of part of the ventricular muscle.

No special is required for the pre-excitation itself**. In the case of supraventricular tachycardia, ** is the same as general supraventricular tachycardia. In the case of atrial fibrillation or atrial flutter, if the ventricular rate is rapid and accompanied by circulatory disorders, synchronous direct current cardioversion should be used as soon as possible.

Lidocaine, procainamide, propafenone, and amiodarone slow conduction in the bypass pathway, which can slow the ventricular rate or revert atrial fibrillation and atrial flutter to sinus rhythm. Digitalis accelerates bypass conduction, and verapamil and propranolol slow intranodal conduction, both of which may significantly increase the ventricular rate and even develop ventricular fibrillation, so it should not be used. If supraventricular tachycardia, atrial fibrillation, and atrial flutter seizures are frequent, the above-mentioned antiarrhythmic drugs should be used for long-term oral prophylaxis of seizures.

If the drug cannot be controlled, the electrophysiological examination determines that the bypass refractory period is short, or the bypass refractory period is shortened by rapid atrial pacing, or the ventricular rate reaches about 200 minutes at the time of atrial fibrillation, there are indications for electrolysis, radiofrequency, laser or cryoablation after positioning, or surgical shut-off of the bypass to prevent seizures.

Categories: Healthcare.

Analysis: Pre-excitation syndrome is a congenital heart disease, which is an extra bypass in the normal conduction pathway of the heart. It is easy to cause tachyarrhythmias.

If you feel nervous, feel your pulse, if the pulse rate is below 160, you must not be worried. Radiofrequency ablation can also be used to cut the bypass, but it is not always necessary. Because some people don't have symptoms for the rest of their lives.

With increasing age, the incidence also decreasesPreexcitation is an abnormal phenomenon of atrioventricular conduction in which impulses travel down through additional channels and prematurely excite some or all of the ventricles, causing premature activation of part of the ventricular muscle. Those with pre-excitation phenomenon are called pre-excitation syndrome or syndrome, often combined with supraventricular paroxysmal tachycardia, and the diagnosis is mainly * electrocardiogram. Pre-excitation is the presence of congenital atrioventricular accessory channels (referred to as accessory pathways) other than the normal atrioventricular conduction system.

Most patients do not have structural heart disease. Preexcitation alone is asymptomatic, and concurrent supraventricular tachycardia is similar to that of general supraventricular tachycardia. The pre-excitation itself does not require special **.

Concurrent supraventricular tachycardia** is the same as general supraventricular tachycardia. If supraventricular tachycardia, atrial fibrillation, and frequent atrial flutter seizures, you should go to a qualified hospital for radiofrequency ablation**, and destroy the bypass to achieve **. If you don't have structural heart disease or an arrhythmia, don't worry.

Preexcitation syndrome is a congenital heart disease in which an additional pathway is added to the heart's normal conduction pathway (the presence of congenital atrioventricular accession channels (referred to as accessory pathways) in addition to the normal AV conduction system). Due to the presence of additional channels (referred to as bypasses), it is easy to cause tachyarrhythmias, causing supraventricular tachycardia. The incidence also decreases with age.

Most patients do not have structural heart disease.

Pre-excitation syndrome itself does not cause clinical symptoms, but symptoms such as chest tightness and palpitation may occur when supraventricular tachycardia is caused. Some people don't have symptoms for the rest of their lives.

The pre-excitation itself does not require special **. However, if rapid supraventricular tachycardia is present, urgent management is often required to stop the onset of supraventricular tachycardia. If supraventricular tachycardia or atrial fibrillation and atrial flutter seizures are frequent, you should go to a qualified hospital for radiofrequency ablation**, and destroy the bypass to achieve **.

This method is the best method for patients with high efficacy. If you don't have structural heart disease and haven't had an arrhythmia, you don't have to worry too much.

Hello, now there are two main problems, one is your heart rate variability. Generally speaking, the heart rate of normal people will increase significantly when they are exercising or excited, and it will slow down when they are resting or relaxing, if you measure your heart rate at about 60 times each time, you should be careful if there is a problem of low heart rate variability, which is mostly due to sinus node dysfunction, and 24h Holter ECG can evaluate heart rate variability.

Regarding type A preexcitation, most of them are due to abnormal "high-speed passages" in the atrioventricular ring of the left ventricle or the atrioventricular ring behind the right ventricle, such as rapid supraventricular arrhythmia, impulses can be directly transmitted down through such a "highway" without filtering through the atrioventricular node, making the heart beat very fast, typical manifestations will be chest tightness, palpitations, dizziness, blackness in front of the eyes, fainting, etc., and in more serious cases, it may even cause ventricular fibrillation and be fatal.

The best way for this disease is to do radiofrequency ablation, the trauma is minimal and the success rate of the operation is very high, you are so young, you should go for such an operation, and if there is a suspicion of sinus node function, you can be carefully examined at the same time during the operation.

Hello, I should say that there is nothing to pay attention to, because it is useless to pay attention, the pre-excitation is mainly to see if paroxysmal tachycardia occurs, if it does not occur, or rarely occurs, if the tachycardia occurs frequently, radiofrequency surgery should be carried out**.

The heart rate at the onset of pre-excitation syndrome is at least 160, and there is a sudden stop, your current symptoms are likely to be hypochondriac after learning of your condition, appropriate self-regulation, don't scare yourself, if you have a sudden attack, try to pick your throat, or massage the carotid sinus by yourself, the heart rate will come down.

Paroxysmal supraventricular tachycardia, atrial flutter and atrial fibrillation, ventricular tachycardia, and atrial tachycardia caused by pre-excitation syndrome and atrioventricular node dual meridians. Among them, the ** rate of paroxysmal supraventricular tachycardia can reach more than 90, and the ** rate of ventricular tachycardia is about 50. Radiofrequency ablation of atrial tachycardia, atrial flutter, and atrial fibrillation is in clinical trials.

The medical problem of pre-excitation syndrome can just look at the above, but it is a very professional thing, I will answer your question, if your situation is pre-excitation syndrome, generally speaking, it has nothing to do with your previous exercise, because this is a congenital disease, that is, there is a little problem with the conduction of your heart, but it is not a serious problem, first of all, you should not be nervous. Your current situation is occasional tachycardia, so there is no need for medication**, and in ordinary life, you should be careful not to be overworked and nervous, and there are generally no excessive requirements for diet. **The drug is mainly aimed at the absorption of substances and blood lipid metabolism in people, and does not have a great impact on your disease, and you can apply it under the guidance of a dietitian.

There is a good way to do this disease now, and that is to ablate**, and there is no great danger, but now you are not the best time, just observe it. If your tachycardia attacks frequently.

Tachycardia cardiomyopathy may occur, but it is not at that stage and is generally not life-threatening.

The pre-excitation itself does not cause symptoms, and there are no adverse manifestations of general pre-excitation. The average occurrence of the population is thousands, and of course, the pre-excitation also returns to bad performance. Atrial fibrillation, tachycardia, and generally do not**.

If severe, it can worsen into heart failure. If tachycardia episodes are frequent and semi-symptomatic, it should be **.

There are three differences:

1. The preshock and QRS complexes of type A are both upward in lead V1, while the preshock and main waves of the QRS complex in lead B V1 are downward.

2. Type A indicates myocardial preexcitation at the left ventricular or posterior base of the right ventricle, while type B indicates myocardial prestimulation of the anterior lateral wall of the right ventricle.

3. The bypass of type A is located around the left atrioventricular annulus, and the bypass of type B is located around the right atrioventricular annulus.

Type A indicates preexcitation of the left ventricular or posterior right ventricular basilar myocardium, and type B suggests the latter suggests myocardial prestimulation of the anterior lateral wall of the right ventricle. Although this classification method is limited by the variable QRS complexes caused by the bypass of different parts of the preexcitation, it helps to distinguish the ventricular end of the accessory route from left or right, anterior or posterior, and is still used today.

According to the morphology of the QRS complex in the precordial leads of the ECG, preexcitation syndrome was previously divided into two types: type A QRS main wave is upward, and preexcitation occurs in the left ventricular or right posterior ventricular bottom; In type B, the main QRS complex in V1 is down, and in leads V5 and V6 is up, and preexcitation occurs in the anterior lateral wall of the right ventricle.

Positivity is seen in the normal route of anterior transmission, and the parafascicle is retrograde. Most commonly, the classic manifestation is paroxysmal tachycardia with sudden stopping. Frequencies range from 150 to 250 per minute, the heart rhythm is regular, and retrograde P waves are retrograde in the ST segment and are retrograde through the parafascicular and normal routes.

The QRS presents a fully pre-excited pattern, P is within the QRS, which is not easily visible. This type is distinguished from room velocity. That's about it.

Type A: Activation of this type enters the ventricle from the posterior base of the left ventricle. The pre-shock mean vector points to the left, front, and down.

Schematic representation: the pre-shock wave in leads V1 - V6 is upward, and the main wave of the QRS complex is also upward. Lead V 1 is mostly R, RS, and RSR?

Lead V 6 is R or RS.

Type B: The excitation enters the ventricle from the anterior lateral wall of the right ventricle, depolarizes from anterior to posterior, and the δ vector points to the left posterior. ECG findings:

The preshock in leads V 1 and V 2 is downward, and the main wave of the QRS complex is also downward in the QS or RS pattern, while the preshock in leads V 5 and V 6 is upward, and the main wave is high in R.

The main cause of betalux is that the heart rate slows down, and the early or excessive dosage can cause heart failure or heart failure aggravation.