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Cerebellar atrophy is divided into physiological and pathological. Physiological cerebellar atrophy maintains normal lifespan; Pathologic cerebellar atrophy has a limited survival cycle.
1. Physiological cerebellar atrophy.
Physiological cerebellar atrophy has no other obvious symptoms, mainly caused by physiological factors, no lesions occur in local brain tissues, and the survival time of patients can be the same as that of normal people, and there is no difference.
2. Pathological cerebellar atrophy.
Pathological cerebellar atrophy is complicated with many symptoms, and most patients are accompanied by complications such as dysphagia, slurred speech, unsteady walking, and uncoordinated movements. The disease is irreversible, and the purpose of surgery**, radiotherapy, chemotherapy, nerve tissue repair** and other methods** is to delay the speed of local lesions and control a variety of symptoms. The survival time of those who are not timely** is generally 3 to 5 years, and the survival time of individual care can reach more than 10 years.
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Cerebellar atrophy ataxia is the main manifestation of central nervous system diseases, mostly from the prime of life, hereditary, according to statistics, the mortality rate of various brain atrophy diseases is getting higher and higher. Worldwide, about 15 million people die each year from various brain atrophy diseases, accounting for more than 50% of the total mortality rate. The mortality rate of brain atrophy disease is unimaginably high, and the complications caused by various brain atrophy diseases are even more unexpected.
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Patients with cerebellar atrophy will go through three stages: early, middle and late stages as the disease progresses. Normally, the course of the disease from cerebellar atrophy to death is about 3 7 years, but because the family did not find the initial symptoms of the patient in time, and did not timely **, many patients have entered the middle or even late stage of the disease when they are diagnosed with cerebral atrophy, so the question of how long patients with cerebral atrophy can live depends on the specific situation of the patient.
Cerebellar atrophy has nothing to do with the life expectancy of the human body, the main role of the cerebellum is to have an impact on the patient's ability to exercise, balance and other abilities, and does not have much impact on the life span of the human body, so the patient with cerebellar atrophy will have great obstacles in exercise ability and self-care ability in life, but as long as the nursing is in place, the survival time should be relatively long.
How long can patients with cerebellar atrophy live is mainly judged according to the severity of cerebellar atrophy and the rate of progression, and the survival time of cerebellar atrophy caused by different causes will be different, so the specific survival period is not particularly clear, and the average survival period is about 20 years.
Once cerebellar atrophy occurs, it is impossible to achieve the goal of complete reversal, and it is mainly necessary to actively deal with the goal, if the cerebellar atrophy can be removed, there will be no obvious progress in cerebellar atrophy. However, if the symptoms of cerebellar atrophy are caused by cerebrovascular disease or due to genetic factors, they may gradually worsen, causing symptoms such as gait instability, ataxia, severe memory impairment, mental retardation, and dementia. Once the patient is bedridden, it is prone to complications such as bedsores and pneumonia that threaten the patient's life, and if the complications can be prevented, the survival of the patient can be effectively prolonged.
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Cerebellar atrophy is often caused by symptomatic other **, not simple atrophy, and can generally live for several years according to the original disease, as follows: 1. Degenerative disease, multisystem atrophy, patients may only live for 1-2 years; 2. Olive, pontine cerebellar atrophy, patients can only live for 2-3 years; 3. Cerebellar ataxia or hereditary cerebellar atrophy can survive for a long time. Because cerebellar atrophy is often combined with some symptoms such as swallowing disorders and unsteady walking, it is easy to fall and affect life expectancy, such as pneumonia, which may cause life-threatening septic shock, and infection after falling and fracture in bed, all of which affect life expectancy.
Therefore, cerebellar atrophy can live for several years, and it should be judged according to the cause and complications.
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How long can you live with cerebellar atrophy? Regarding this problem, I don't think you have to worry, as long as the brain atrophy is standardized, it can still be controlled, and it can be controlled with some family conditioning. Patients with cerebral atrophy should pay attention to supplementing seafood, edible mushrooms, beans and their products, fish, milk, sesame paste, various vegetables and fruits and other foods in their daily diet, so that the body can obtain sufficient minerals.
1) When cooking various dishes, do not put too much MSG. Brain tissue produces glutamate to aminobutyric acid through the conversion of enzymes. When too much MSG is ingested, it can cause symptoms such as headaches and nausea.
2) Pay attention to a low-sugar diet. Because too much sugar, especially refined sugar, is easy to cause brain function to appear nervous or neurasthenic disorders. (3) Sufficient supply of essential fatty acids.
It is extremely important to provide sufficient essential fatty acids in the diet, which are indispensable nutrients for the normal function of the brain, walnuts, fish oil, safflower oil, and evening primrose oil have more essential fatty acid content, which can be increased in the diet. (4) Attention should be paid to supplementing vitamins in the diet. Such as E, vitamin C and - carotene-rich foods, such as wheat germ oil, cottonseed oil, corn oil, peanut oil, sesame oil, etc., these substances have antioxidant substances and can delay aging.
Geriatric studies have found that some minerals contained in the diet are closely related to the prevention and treatment of Alzheimer's disease, such as calcium can regulate neuromuscular excitability, maintain the normal activity of heart function, and improve the cognitive ability of the elderly. Magnesium is a cofactor for various enzyme reactions, and works with potassium, calcium and other elements to maintain myocardium and prevent arteriosclerosis, thereby enhancing cerebral blood flow, which is conducive to the prevention and treatment of Alzheimer's disease.
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1. The most severe cerebellar atrophy will cause dysfunction of swallowing function, difficulty swallowing, and eventually the need for nasogastric feeding for liquid diet, because the cerebellum will also affect language function, and eventually slurred speech. If the cerebellum is severely atrophied, there will eventually be changes in muscle tone, uncoordination of the body, disharmony of the limbs, affecting the activities of the limbs, and eventually bed rest.
2. Patients with cerebellar atrophy will also have a limb tremor in severe cases, showing Parkinson-like seizures, so the most serious cerebellar atrophy is to affect the function of language, and there will be a complete inability to speak. It can also affect swallowing function and requires nasogastric feeding for maintenance diet. Eventually, it affects our motor function, and there will be complete inability to move accompanied by tremors of the limbs.
The cerebellum is located behind the cranium, below the tentorial of the cerebellum, dorsal to the pons and medulla oblongata. The cerebellar vermis and the cerebellar hemispheres on both sides make up the cerebellum. Symptoms of cerebellar atrophy are ataxia and balance disorders.
When cerebellar vermis atrophy is severe, there may be trunk dysfunction, inability to maintain uprightness, unsteady standing, forward or backward leaning, difficulty walking (+) feet apart, shaking from side to side, and opening the eyes cannot change this ataxia. Limb ataxia occurs when the cerebellar hemispheres are severely atrophied, with the upper extremities heavier than the lower extremities, the distal segments heavier than the proximal segments, and the fine motor segments heavier than the gross motor segments. Patients with severe cerebellar atrophy are unable to walk and can only be used in wheelchairs or bed rest.
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It is difficult to conclude that patients with cerebellar atrophy live for several years, as each patient has a different degree of progression and a different condition. In most cases, patients with cerebellar atrophy are able to live relatively long, one or two decades should be no problem. Because patients with cerebellar atrophy tend to run in hereditary families, many of them will have this condition.
The disease often does not progress very quickly, and the main impact is on the body's ability to balance and control the limbs, and does not affect the patient's life. Only when the cerebellar atrophy reaches the end stage of severe inability to move and stay in bed, some complications will occur and death.
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If there is cerebellar atrophy, the main thing is to pay attention to the application of some drugs that nourish brain cells, and at the same time, eat more brain-replenishing foods in the diet, such as walnuts, jujubes, soy products, etc., which can play a role in protecting the brain, nourishing brain cells to avoid aggravation of brain atrophy, and usually using the brain regularly to avoid the appearance of high blood pressure.
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Cerebellar atrophy is often caused by genetic degenerative diseases, such as common multisystem atrophy and cerebellar pontine olive degeneration, and patients may have obvious ataxia, unsteady walking, dizziness and other symptoms. At the same time, patients will have dysfunction of the extravertebral system and autonomic nerves, and some patients will also have symptoms of vertebral tract damage such as increased muscle tone.
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The disease has the greatest impact on the brain, and it does not have a particularly large impact on the rest of the body.
If you are in good physical condition, it will not have a particularly big impact on your lifespan.
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Cerebellar ataxia is one of the more common diseases. The occurrence of the disease has brought a lot of impact to the patient. It can even make the patient lose the ability to take care of himself.
Changes in muscle tone can change from decreased lesion to spasticity, and the ataxic gait can change to spastic ataxia. Unsteady standing, leaning forward or swaying from side to side, more prominent when standing on tiptoe or heel, and easy to fall are often the most common symptoms in the early stage of the patient.
Name: Mr. Guan.
Age: 43 years old.
Course of disease: suffered from cerebellar ataxia for 4 years, main symptoms: dizziness, blurred vision, slurred speech, inaccurate hand grasping, small strength, unsteady legs, shaky walking, diarrhea, cold thighs and calves, poor sleep, dreaming, easy to wake up, sweating at night, hot hands and feet at night, constipation, burning sensation in urine, heart discomfort, palpitation, occasional tinnitus.
Often dry cough, insufficient gas, choking cough when drinking water, heavy salt taste like to drink water, heavy menstrual flow and blackness, easy temper tantrum, tongue with tooth marks, tongue coating cracks.
Previously**: After taking clonazepam, butylphthalide, methylcobalamin and other drugs, the condition was controlled, but *** was too large, but the condition never improved.
Now**: Brain Walking Soup**.
After a course of treatment: dizziness and cough improved slightly, sleep quality recovered, blurred vision, blurred ghosting improved.
After three courses of treatment: walking more steadily, insomnia and dreams better, urine and urine improved, and the feeling of cold legs and feet was better.
After six courses of treatment: walking steadily, drinking water and choking, difficulty in swallowing recovered, speaking clearly, dizziness was gone, the whole body muscle strength was basically restored, blurred vision was basically recovered, the legs and feet were cold, night sweats, heart, tinnitus improved, and then continued to consolidate a course of treatment, the symptoms were basically none, and now there is no recurrence in the return visit.
Suggestion: Patients with cerebellar ataxia should also maintain daily nutrition and calorie intake, and at the same time, patients with many sequelae of cerebral hemorrhage are prone to aphasia and inability to express their physical condition, and cerebellar ataxia occurs, resulting in a lack of nutrients in the patient's body and need to be ingested from food
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Patients with cerebellar atrophy do not have an impact on their survival, but only on their quality of life and ability to behave normally. Patients with cerebellar atrophy are mainly characterized by gait instability, abnormal balance and ataxia, and some patients are even bedridden for a long time or even unable to walk at all due to difficulty in walking. As a result, some patients develop lesions such as pneumonia, which will affect the survival time of patients.
And for patients with slower disease progression, the quality of life is poor, but the survival time can be more than decades.
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Cerebellar atrophy alone is not life-threatening. Most of the causes of death are due to other causes, such as multiple cerebral infarctions of the heart and brain, or failure of other organs, such as kidney failure. Or some patients have serious complications, such as an infection in the lungs. Some patients are malnourished or have hypoproteinemia.
Some cerebellar atrophy causes trauma, resulting in traumatic brain injury, which can also be life-threatening. Cerebellar atrophy alone does not kill people.
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Cerebellar atrophy can be treated with traditional Chinese medicine, and it can be conditioned, it is not a terminal disease, there is no need to worry.
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There is no exact number of years for survival in patients with cerebellar atrophy.
1. Causes of cerebellar atrophy.
1.Hereditary cerebellar ataxia, such as spinocerebellar ataxia and ataxia, can lead to cerebellar atrophy.
2.Hepatocerebral degeneration: Chronic acquired hepatocerebral degeneration and Wilson's disease can cause cerebellar degeneration.
3.Genetic phenomenon: Patients with familial amaurosis often have obvious genetic phenomena, with enlarged nerve cells, lipid-like cytoplasm, and small granules.
2. The best method of cerebellar atrophy.
1.Drugs**: Combination of buspirone, amantadine, and gabapentin can improve ataxia symptoms; Levodopa or dopamine agonists can relieve extrapyramidal symptoms such as rigidity.
2.Traditional Chinese Medicine**: Traditional Chinese medicine believes that cerebellar atrophy is caused by lack of righteous qi, qi deficiency and blood stasis, and obstruction of the veins. **It is mainly based on invigorating qi and blood circulation, filling essence and opening the body. Its commonly used dietary therapy methods include ginseng porridge, sesame porridge, walnut porridge, etc.
3.Psychological**: Family members should patiently listen to the patient's voice, understand the patient's psychological state, and choose the appropriate way to communicate with the patient. Only by treating patients with sincerity can their loneliness and anxiety be alleviated.
3. Precautions for patients with cerebellar atrophy.
1.Smoking and alcohol are prohibited to prevent the patient's condition from worsening.
2.Eat more easily digestible foods rich in high-quality protein and vitamins and give them the necessary nutritional support.
3.Patients with acute cerebral infarction should eat a low-salt and low-fat diet.
To sum up: cerebellar atrophy itself does not affect a person's lifespan, but if there are some complications of cerebellar atrophy, it will affect a person's lifespan.
How long you can live with cerebellar atrophy is related to **. Hereditary cerebellar ataxia and multiple system atrophy are neurodegenerative diseases with a survival period of 10 to 20 years after the onset of the disease, and the survival time of patients can reach about 20 years through active intervention, healthy lifestyle and good care of family members. In some patients, the lesions that cause the cerebellum are quiescent and do not develop, and their survival time is the same as that of normal people.
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