What is the basis for the medical diagnosis of Hashimoto s thyroiditis?

The early symptoms of Hashimoto's thyroiditis are highly hidden, and some patients do not know the ** and symptoms of the disease, and the probability of being detected early is very low. Only through medical examination and diagnosis can the type of disease be confirmed, and the severity of the disease can be selected, and the best way to control the disease is symptomatic. So, what are the medical diagnostic bases for the diagnosis of whether it belongs to this disease type?

The medical diagnosis of Hashimoto's thyroiditis is based on: 1. Thyroid function test is also one of the methods to check for thyroiditis. Toxic nodules that function autonomously are mostly hyperthyroidism, which can also occur in the early stages of subacute thyroiditis, and thyroid function in chronic lymphocytic thyroiditis can be normal, hyperactive, or reduced.

Thyroid nodules caused by the remaining lesions are mostly functional. 2. Most of Hashimoto's thyroiditis turns into hypothyroidism, and if there are symptoms of hypothyroidism, thyroid preparations are used to replace it**, Hashimoto's thyroiditis can recover naturally, but the progress is very slow. For patients with obvious goiter, particularly hard texture, noduular shape, even if the thyroid function is normal, thyroid preparations should be applied**, and after the thyroid gland shrinks, it should be maintained for about two years**.

This is also one of the diagnoses of Hashimoto's thyroiditis. 3. The titers of anti-thyroglobulin antibody (TGA) and anti-thyroid microsomal (peroxidase) antibody (TMA) in the blood of immunological examination are significantly increased, and both of them are greater than 50% (radiotherapy-free bispecific antibody method), which can last for several years or more than ten years. 4. Thyroid ultrasound examination can show that the nodule is solid, cystic or mixed lesions, a single solid nodule has a higher possibility of malignancy, mixed nodules also have the possibility of malignancy, and simple cystic nodules have less chance of malignancy.

5. A variety of direct antibodies can be detected in the patient's serum hyperthyroidism, the important ones are anti-thyroglobulin antibody (TGA), anti-thyroid microsomal antibody (MCA), and the other two glial antibodies and cell surface antibodies can be detected by fluorescent immunization in the patient, and there are T cells and B cells infiltrated in the thyroid tissue, and the disease is definitely related to genetic cause hypothyroidism. (Recommended reading: What are the triggers for the onset of Hashimoto's disease) Hashimoto's thyroiditis can be used"Targeted regulation—TSH balance**"**，"Targeted regulation—TSH balance**"**This is not toxic*** and very safe.

It can start from the root cause of thyroid secretion, combined with the principle of thyroid in traditional Chinese medicine, first change the internal environment of the human body, so that the abnormal secretion of thyroid disease is not suitable for survival, and then combined with modern medicine (Western medicine), the use of modern nanotechnology, will not hurt the patient's liver and stomach, and its best effect is also ideal, greatly shortening the first time. Tips: Be familiar with the key points of medical diagnosis of Hashimoto's thyroiditis to avoid misdiagnosis and endanger the health of patients.

The symptoms of Hashimoto's thyroiditis in the early stage are not obvious, and patients generally have symptoms such as heat intolerance, weight loss, and irritability in the middle stage, and there will be fundamental changes with the development of the disease.

Most patients with Hashimoto's thyroiditis present with hypothyroidism, general fatigue, non-concave edema, abdominal distention, less urine, slow movements, lack of speech, slow response to answers, heart rate below 60 minutes, hoarseness, thickness, and desquamation. Most patients often have pharyngeal discomfort, and a few may have cardiomegaly, pericardial effusion, or coronary artery disease.

Hashimoto's thyroiditis, also known as chronic lymphocytic thyroiditis, is an autoimmune thyroid disease that is likely to be cured even if cured. The clinical symptoms of the disease are not obvious in the early stage, and the manifestations of hypothyroidism appear in the late course of the disease. If thyroid function is normal, no special ** is needed.

It can also be properly conditioned with anti-inflammatory, analgesic, swelling and dispersing Prunella vulgaris preparations. Commonly used drugs are Rhizata, which is suitable for patients with thyroiditis. **During this period, regular follow-up visits.

Eliminate greasy, fried, high-sugar, high-salt foods and avoid extra energy intake. Pay attention to eat more fresh vegetables, fruits, etc., and supplement more vitamin B complex, trace elements, electrolytes and other auxiliary improvements.

The symptoms of Hashimoto's thyroiditis have a variety of manifestations:

1. Patients with Hashimoto's thyroiditis may be manifested by hyperthyroidism, and typical hyperthyroid symptoms include: hand tremors, heat intolerance, easy sweating, restlessness, palpitations, weight loss, etc.

2. Patients with Hashimoto's thyroiditis can also have hypothyroidism, and typical hypothyroidism has the following symptoms: sensitivity to cold, fatness, constipation, listlessness, irregular menstruation, etc.

Hello landlord, Hashimoto's thyroiditis is more common in women in their 30s and 50s, with insidious onset, slow development and long course of disease, mainly manifested as goiter, most of which are diffuse, a few can be localized, and some of them start with swelling of the face and limbs. The disease can be divided into eight types:

1) Hashimoto's hyperthyroidism: patients have typical symptoms of hyperthyroidism and positive laboratory test results, and hyperthyroidism and Hashimoto's disease can occur at the same time or successively, coexisting with each other, and transforming each other.

2) Pseudohyperthyroidism: a small number of symptoms of hyperthyroidism may be present, but there is no evidence of hyperthyroidism in thyroid function tests, and TGAB and TMAB are positive.

3) Exophthalmos: proptosis, thyroid function can be normal, hyperactive or reduced.

4) Subacute thyroiditis type: acute onset, goiter pain, fever, accelerated blood sedimentation rate, but the iodine intake rate of 131 is normal or increased, and the thyroid antibody titer is positive.

5) Juvenile type: accounting for about 40% of juvenile goiter, with normal thyroid function and low antibody titers.

6) Fibrotic type: the course of the disease is long, and extensive or partial fibrosis of the thyroid gland, thyroid atrophy, and hypothyroidism may occur.

7) Accompanied by thyroid adenoma or cancer: often solitary nodules, with high TGAB and TMAB titers, and (8) accompanied by other autoimmune diseases. Pro-Phecy hopes to help you.

Irritable mood and bad temper.

This disease is more common in middle-aged women, manifested as goiter, slow onset, often found inadvertently, the volume is about 2 3 times that of the normal thyroid gland, the surface is smooth, the texture is tough and elastic like rubber, obvious nodules are rare, no tenderness, no adhesions around it, and can move with swallowing. In the late stages, a small number of symptoms of mild local compression may occur.

The disease progresses slowly, and sometimes the goiter does not seem to change significantly for several years. Initially, the thyroid gland is functioning normally. In the course of the disease, sometimes hyperthyroidism can also occur, followed by normal function, hypothyroidism, and then normal, the process is similar to subacute thyroiditis, but without pain, fever, etc., so this state is called ** thyroiditis, and the postpartum onset is called postpartum thyroiditis.

However, when the thyroid gland is destroyed to a certain extent, many patients gradually develop hypothyroidism, and a few have myxedema. The disease is sometimes associated with pernicious anemia due to the presence of autoantibodies to the parietal cells of the stomach. Hashimoto's thyroiditis is initially variable and can easily cause mistaken symptoms.

Hashimoto's thyroiditis can be divided into the following basic types:

1) Hashimoto hyperthyroidism: patients are accompanied by hyperthyroidism, and some cases may also have infiltrative exophthalmos, myxedema, etc. Typical hyperthyroidism may be present.

Such patients may have hyperthyroidism for several years and often require antithyroid drugs** or the use of traditional Chinese medicine (TCM)** to prevent hypothyroidism from developing. Surgical excision or radionuclides** are not appropriate and are prone to permanent hypothyroidism.

2) Pseudohyperthyroidism: A small number of patients may have clinical manifestations of hyperthyroidism, such as palpitations, hyperhidrosis, nervousness, etc., but there is no evidence of hyperthyroidism in thyroid function tests, and TGAB and TMAB are positive. Such patients do not need antithyroid drugs** and symptoms go away on their own.

3) Ophthalmos: Infiltrative exophthalmos can occur in this disease, and its thyroid function can be normal, hyperactive or hypothyroid. The retroorbital muscles have lymphocytic infiltrates and edema. Serum TGAB and TMAB were positive. This article is from the Thyroid Diagnosis and Treatment Center of Beijing Canal Hospital of Traditional Chinese Medicine.

1. Hand tremors, heat intolerance, easy sweating, restlessness, palpitations, weight loss, abnormal menstruation, at this time, anti-thyroid drugs must be used**. Patients with Hashimoto's thyroiditis can also have hypothyroidism.

2. Sensitivity to cold, fatness, easy constipation, listlessness, irregular menstruation, etc., the way of Hashimoto's thyroiditis** is to supplement thyroxine hormone. will-ing hope it helps.