What kind of method is used to demyelination now?

What is the best method of demyelinating disease: muscle aches, feeling no strength, detecting demyelinating disease, what is the best method of demyelinating disease?

Demyelinating disease is symptomatic. For example, in the acute phase, high-dose corticosteroids** are predominantly used. This is followed by a stable phase, when disease modification is used to control disease progression and reduce**.

Finally, the drug is controlled, and carbamazepine is used to inhibit nerve firing and relieve pain symptoms. ****。The specific medication should be combined with the actual clinical practice, and the doctor's face-to-face consultation guidance shall prevail.

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Cancel. Zhou Zeng, Deputy Chief Physician.

Department of Neurology, Ma'anshan People's Hospital.

Top three. Disease analysis: Nutritional deficiency of nutritional supplementation in demyelinating disease.

Complications**Caused by poisoning, detoxification treatment is carried out; If carbon monoxide poisoning occurs, it should be separated from the toxic environment as soon as possible and oxygen should be administered**. , such as hereditary disorders, should be adjusted according to the specific cause of the genetic and metabolic disorders. Finally, for autoimmune dysfunction, drugs such as nourishing nerves and improving metabolism can also be added.

You can try Lee's Raw Myelin Solid Sheath Soup**.

What about demyelination? This method is simply amazing?

The prevalence of demyelinating encephalopathy is relatively low in life, but it is usually less effective in recovery. The damage caused to the patient's home work is great, and the key to demyelinating encephalopathy is a neurological disease characterized by the loss and damage of the myelin sheath of the white matter sympathetic nerve of the brain and spinal cord.

Demyelinating encephalopathy, hemiplegia, joint muscle stiffness, pressure ulcers, visual impairment.

The key symptoms are divided into two types: one is the myelin sheath destruction type, in which the myelin sheath is damaged by itself or other viruses; The other is the transformation of the myelin sheath into an obstruction, most of which is harmed by genetic abnormalities. Demyelinating encephalopathy is a key prevalent location of harm:

Locations of the optic nerve, spinal cord, thalamus, brainstem, etc. Symptoms such as visual impairment, impaired activity of the limbs, and poor ability to balance work will be exacerbated. MRI, cerebrospinal fluid, electrophysiology and other auxiliary tools can be used to help confirm the diagnosis.

Patients and their loved ones should be careful and attentive to get rid of depression and ensure the following categories as much as possible:

Healing and medical care at home:

1. If the disease is discovered, it should be treated immediately to avoid further malignant transformation of the disease and reduce the sequelae in the middle and late stages.

2. Avoid some very easy to cause symptoms, such as: excessive fatigue, excitement, some infectious diseases, etc. will make the symptoms attack or further aggravate the safety risk, pregnancy will also aggravate the disease, so young ladies suffering from demyelination should prepare for pregnancy under the specific guidance of the doctor.

3.During the recovery period of surgery, the hemiplegic body should be given moderate passive skill fitness exercises to prevent joint muscle stiffness and pain; Patients who are bedridden at the end of the stage should be given frequent risen-up and skin care to avoid pressure ulcers.

4.Patients should go to the hospital outpatient clinic for re-examination on time, pay attention to the development trend of the condition, properly handle the change of the condition, and avoid further malignant deterioration of the condition.

Herb soup.

Step 1: Relieve wind and stop spasms, clear away heat and scare, invigorate blood and disperse blood stasis, so as to dispel wind and channels, open the body and awaken the mind.

Step 2: Nourish blood and qi, clear away heat and detoxify, nourish liver and kidneys, so as to generate jin and invigorate qi, and benefit the eyes and minds.

Step 3: Dispelling wind and cold, regulating qi and strengthening the spleen, which can regulate qi and strengthen the spleen, nourish and nourish the spleen.

First, the most common is mediated by an immune-inflammatory response. These are caused by autoimmune dysfunction, which leads to the appearance of abnormal immune cells and abnormal antibodies in the body, such as multiple sclerosis, neuromyelitis optica spectrum and other related diseases.

Second cerebral ischemia is also a very common cause of white matter demyelination, high blood pressure leads to degeneration of arterioles, so deep white matter will also appear demyelinating-like lesions.

Third, some patients' specific infection, poisoning, and inflammatory reactions can also cause demyelinating diseases.

Demyelinating lesions can occur in the peripheral nervous system or in the central nervous system, where demyelinating lesions can be caused by congenital qualitative factors, such as genetic factors, resulting in deficiencies in certain enzymes, causing a disorder of nerve myelin phospholipid metabolism, called leukodystrophy. Common diseases such as adrenoleukodystrophy, metachromatic leukodystrophy, etc.; Acquired factors are demyelinating lesions caused by certain diseases, such as multiple sclerosis, neuromyelitis optica spectrum diseases, and carbon monoxide poisoning caused by ischemia and hypoxia, delayed encephalopathy, etc.; Diseases caused by nutrient deficiencies, such as subacute combined degeneration; Viral infectious diseases, such as subacute sclerosing panencephalitis caused by measles virus infection, have pathological manifestations of white matter demyelination. Demyelination in the peripheral nervous system, commonly known as Guillain-Barré syndrome, is caused by an autoimmune response.

For autoimmunogenic demyelinating diseases, glucocorticoids are commonly used to administer gamma globulin if necessary.

In the case of malyelination, Jianqingchan recommends that the patient be treated as follows:

In the first case, if this demyelination is caused by cerebrovascular disease, such as cerebral small vessel disease will cause multiple demyelination of white matter, the main way to deal with it is that the patient must pay attention to maintaining a stable mood, control blood pressure, achieve a low-salt and low-fat diet, can take anti-platelet aggregation drugs, and some drugs that reduce blood lipids and stabilize atherosclerosis**.

In the second case, if the demyelination is caused by multiple sclerosis and neuromyelitis optica, the first way is to use some gamma globulin, glucocorticoids, and long-term follow-up to adjust the dose of the drug.

If demyelination occurs, the following management is recommended

In the first case, if the demyelination is caused by cerebrovascular disease, such as cerebral small vessel disease will cause multiple demyelination of white matter, the main way to deal with it is that the patient must pay attention to maintaining a stable mood, control blood pressure, achieve a low-salt and low-fat diet, and can take anti-platelet aggregation drugs, as well as some drugs that reduce blood lipids and stabilize atherosclerosis.

In the second case, if the demyelination is caused by multiple sclerosis or neuromyelitis optica, the first way is to use some gamma globulin, glucocorticoids, and long-term follow-up to adjust the dose of the drug.

In the third case, if the peripheral nerve is demyelinated, such as acute Guillain-Barré, the modality is mainly gamma globulin and B vitamins.

Demyelination is a general term for a large group of diseases, and any disease that damages the myelin sheath can be called a demyelinating disease, such as multiple sclerosis, acute disseminated encephalomyelitis, and Barlow disease. These diseases are generally unable to **, and it is necessary to give symptomatic treatment as soon as possible to control the development of the disease and avoid life-threatening. When the myelin sheath is damaged, scar tissue forms around it.

The signals sent by the brain do not pass through the scar tissue, which ultimately causes the nerves to not function properly.

Demyelinating lesions are acute or subacute neurological impairments that can be relieved by:

1. In the early stage of the disease, hydrocortisone or dexamethasone can be used intravenously**, and the dosage can be gradually reduced with the remission of the disease.

2. Mannitol can be used for intravenous infusion treatment of vertical cavity therapy, which can reduce nerve swelling and improve early blood circulation of the local spinal cord.

3. Neurotrophic drugs such as high-dose vitamin B preparations, citicoline, and methylcobalamin can be used to alleviate local symptoms.

4. It is necessary to turn over and pat the back regularly to prevent bedsores, and at the same time strengthen the patient's limb function exercise to prevent the contracture and deformity of muscles and joints.

What is demyelination? (1) Demyelinating lesions are a general term for a class of diseases with demyelination as the main pathological manifestation, and the more common ones are: multiple sclerosis, neuromyelitis optica, and subacute diffuse demyelinating peripheral neuropathy.

2) The myelin sheath is a layer of chemicals that wrap the outer capsule of neuronal cells. Neuronal cells (Fig. 1) are composed of nerve cell bodies (i.e., key somatic cells) and elongated axons (including myelin sheath and axon) that emerge from nerve cells, and the surface of the axons is composed of a layer of outer skin cysts, which carries the outer skin of the sedan is the myelin sheath (the axons of the nervous system can be roughly compared to the copper wire in the middle of the cable, and the plastic skin on the surface is the myelin sheath).

Data signals are transmitted on the same neuronal cell in a current-flow method. The myelin sheath (plastic sheet) is used to maintain the axons of the nervous system (copper wire) without injury, and the other sail crack outside the speed of the transmission rate of various data signals in the human brain (it is often said that the role of the myelin sheath is very different from that of the cable plastic sheet, depending on not only the maintenance effect, but also the acceleration of electrical signal transmission). 3) Any reason, such as ischemia, abnormal immunity, insufficient nutritional value or harmful chemicals, will cause the loss of myelin, cause abnormal transmission of electrical signals, and cause relative damage, such as:

The main manifestations of activity inflexibility, feeling sluggish or regressing, etc., are called demyelinating disorders. Structure of neuronal cells.

Demyelination is a neurological disease that can directly threaten people's lives and health. After suffering from this disease, the condition will be reversed, and it may lead to severe symptoms such as paralysis and urine and fecal disorders, which greatly affect daily life and activities.

So, what exactly is demyelinating lesions? What should I do best after I get sick?

1. Demyelinating lesions.

The function of the myelin sheath is to protect the neurons and allow nerve impulses to be transmitted on the neurons, so the loss of the myelin sheath will affect the transmission of nerve impulses. There are two major types of acute and chronic diseases, acute demyelinating diseases can regenerate the nerve myelin sheath in acute demyelinating diseases, and the speed is relatively rapid and complete, although the regenerated myelin sheath is thinner, but generally has little impact on functional recovery. In chronic demyelinating neuropathy, the nerve may become thicker and there is axonal loss due to the marked proliferation of regenerated Schwann cells with repeated demyelination and myelination.

This disease can actually be ** in the early stages. However, it is more difficult to maintain the stability of the disease in the later stage of the disease, and can only be maintained by hormone shocks combined with nutritional nerve drugs and other methods. Most of them are caused by abnormal genetic immunity or viral infection, and if it is not well controlled in the later stage, it may lead to dementia, and in severe cases, it can lead to unsteady walking, numbness in the legs, and then paralysis.

2. Symptoms of demyelinating lesions.

1. Early symptoms are numbness and weakness of limbs in daily activities, accompanied by fatigue.

2. Mental state: excitement, strong crying, forced laughter, memory loss.

3. In language communication, you will feel weak to speak, dysarthria, or incoherent speech.

4. Vision loss, dizziness, eye pain.

5. Flaccid paralysis, urinary disorders, impotence, etc.

3. Demyelinating lesions.

Demyelinating lesions are not a disease that cannot be ***, as long as it is detected early, and it is detected and carried out in time** at an early stage, the condition will be well recovered. Western medicine commonly used hormones, immunosuppressants and various neurotrophic factors for demyelinating lesions, etc., its efficacy is limited to controlling symptoms, and the toxic adverse reactions are large, and the effect is not good.

At present, there is no special scientific targeted means and drugs for demyelinating lesions in Western medicine. Compared with Western medicine, TCM** demyelinating lesions have their own advantages, which have been widely recognized and accepted by the majority of patients.

Demyelination occurs when the myelin sheath of the axons of the cerebrospinal cord is lost. There are two categories: myelin destruction; Disorders of myelination. Spinal inflammatory demyelinating diseases are currently considered to be immune-mediated diseases, and their clinical features are:

The patients were children and young adults;

Acute onset, common history of cold, fever, infection, rash, vaccination, cold, childbirth or surgery 1 month before the illness;

A thorough neurological examination often reveals evidence of other central nervous system involvement in addition to spinal cord symptoms and signs;

cerebrospinal fluid examination is positive for oligoclonal bands;

Electrophysiology and MRI can detect some subclinical lesions in the brain; MRI reveals abnormal signals of white matter in the brain. Some myelittic demyelinating lesions present:"Pseudoneoplasia"Manifestations, the MRI shows a mild mass effect, mild edema around it, and there may be patchy hemorrhagic signals, which can easily be misdiagnosed as spinal cord tumors. At present, the effect of internal medicine drugs** is not good.

Therefore, neurosurgical intervention is inevitable and necessary.

Demyelinating myelitis is usually acute multiple sclerosis (MS) spinal type, and the clinical presentation is similar to that of postinfectious myelitis, but the progression is slow, and the disease usually peaks within 1 to 3 weeks. The prodromal infection may be subtle, usually incomplete transverse lesions, with weakness or paralysis of one or both lower extremities, with numbness, the level of sensory impairment is inconspicuous or two-plane, and urinary and fecal disorders are present. Evoked potentials and MRI may reveal lesions elsewhere in the CNS.