What is rickets in children 5, what is rickets in children

What are the causes of rickets in children?

Rickets is also known as "hypochondriasis"., mainly due to vitamin D insufficiency and calcium and phosphorus malabsorption, is a common chronic nutritional deficiency that causes bone growth disorders. The disease is rarely life-threatening, but it develops slowly and is not easy to detect. The earliest manifestations of rickets in children are mainly mental changes, such as irritability, crying, restless sleep, crying, malaise, lack of interest in anything, and profuse sweating at the slightest activity, and hair loss on the back of the head.

Later, the symptoms gradually became apparent, mainly skeletal changes. For example, 3-6 months old babies may have "skull malacia", and the skull can be felt with obvious elasticity when pressed with the hand; Babies aged 8 to 9 months will have a square head, late fontanelle closure, and late teething. In severe cases, the sternum protrudes forward or becomes inverted to form a "chicken breast" or "pectus excavatum", the spine is convex and forms a hunchback, and "X" or "O" shaped legs are easy to form after walking.

The specific reason is caused by the abnormal metabolism of calcium and phosphorus in the whole body caused by vitamin D deficiency, so as long as vitamin D is supplemented and exposed to the sun, rickets can be prevented, ** rickets take vitamin D at the same time as calcium to ensure calcium absorption, parathyroid compensatory hypercompensatory effect is terminated, will not lead to decalcification, rickets can be recovered.

It is a disease caused by vitamin D deficiency that causes calcium and phosphorus metabolism disorders in the body, resulting in poor calcification of bones.

**The key to rickets is to supplement vitamin D, not calcium (of course, calcium supplementation is also necessary and indispensable), to understand that rickets is also divided into degrees, according to different degrees, use different ** schemes. Normal and mild rickets are only fine with ixosine, and if it is moderate or higher, high doses of vitamin D3** will be considered. Vitamin D3 is preferred for the prevention of moderate or higher rickets, and vitamin D-deficient rickets are preferred.

It's better to go to the hospital and listen to what the doctor has to say.

Analysis: Hello, rickets, commonly known as calcium deficiency, is more common in infancy, is a disease caused by vitamin D deficiency caused by calcium and phosphorus metabolism disorders in the body, resulting in poor calcification of bones. Rickets has a slow onset and is not easy to take seriously.

Rickets reduces children's resistance, and is prone to pneumonia and diarrhea, which affects children's growth and development. Therefore, it is necessary to actively prevent and control it.

Suggestions: The current specific drugs mainly include vitamin D tablets, gel pills, injections, and emulsions, and vitamin D3 is absorbed faster than vitamin D2 and has a better effect. It needs to be used under the guidance of a doctor.

Rickets in children is mainly due to less sunlight, which can generally be cured, and can be ** if you are young. Clinical signs and symptoms of rickets at any period of time gradually lessen or disappear after exposure to ** or sunlight. Blood calcium and phosphorus gradually return to normal, and alkaline phosphatase drops to normal levels in about 1-2 months.

**After 2-3 weeks, the X-ray changes of the bones are improved, and irregular calcification lines appear, and then the calcification zone becomes dense and thickened, and gradually returns to normal.

1. Insufficient sunshine.

The natural sunlight ultraviolet wavelength in most regions is 296-310nm, which cannot pass through ordinary glass windows; lack of outdoor activities for infants and young children; Tall buildings in large cities block sunlight; Air pollution such as smog and dust can absorb some ultraviolet rays, which makes endogenous vitamin D production insufficient.

2. Insufficient sunshine for pregnant women.

For this reason, the diet is deficient in nutrients such as vitamin D, calcium, phosphorus, and protein, and the offspring born are susceptible to rickets.

The incidence of rickets is higher in cow's milk-fed children.

3. Insufficient or improper proportion of calcium and phosphorus in food.

The ratio of calcium and phosphorus in human milk is appropriate, 2:1, which is easy to absorb; Although the calcium and phosphorus content in milk is high, it is not easy to absorb because the phosphorus content is too high. Thus, milk-fed'The incidence of rickets is higher in children.

Fourth, the growth and development is too fast.

When children grow and develop too fast, the need for vitamin D is large, and rickets are prone to occur if the supply is insufficient. In addition, premature infants are prone to rickets due to insufficient calcium and phosphorus reserves in their bodies, coupled with rapid growth and development after birth.

5. Some other reasons.

1. Low blood phosphate antivitamin D rickets: it is a hereditary disease, and there are still active rickets after the age of 2-3 years. Serum calcium is normal, serum phosphorus is markedly reduced, and general doses of vitamin D** are ineffective.

2. Distal renal tubular acidosis: the patient is short stature, metabolic acidosis, polyuria, low blood calcium, low blood phosphorus, and low blood potassium.

3. Vitamin D-dependent rickets: It is a hereditary disease, which is caused by the body's inability to convert vitamin D into active vitamin D, or the body's failure to respond to active vitamin D.

4. Nephrogenic rickets: It is caused by calcium and phosphorus metabolism disorders caused by chronic renal dysfunction.

5. Hepatic rickets: caused by liver disease that impairs vitamin D absorption or active vitamin D production.

In summary, there are many causes of rickets in children, which should be analyzed specifically. If your child is still suffering from rickets at the age of 5, it is recommended that you take your child to the pediatric department of the hospital for a detailed examination to clarify the cause before proceeding**.

Canine rickets is a nutritional bone disease caused by vitamin D deficiency, resulting in calcium and phosphorus metabolism disorders, bone calcification insufficiency, and insufficient bone salt deposition. Manifested in osteogenic disorders during growth. The initial symptoms of the disease are loss of appetite, dyspepsia, heterophobia, dull coat, gradual emaciation, and slow growth and development.

Later, the limbs are plantar landing, intertoe opening, joint swelling, forelimb wrist joint deformation and pain, biarticular state, and severe claudication. The long bones are deformed and curved, with "X" or "0" shaped legs. Osteoporosis is loose, swollen, deformed, and the junction of the ribs and costochondral is bead-like.

**: Enhance outdoor activities and get more sun.

1) Add cod liver oil to food, 5 10 ml a day; Or add bone meal, shell powder, and calcium powder to food.

2) Intramuscular injection of vitamin D 330,000 50,000 units of kilogram body weight, once every 3 4 days.

3) Intravenous calcium gluconate 10 ml, 1 time a day, for 3 to 5 days.

4) Bone Enhancement Powder 1 2 sachets to be taken internally, 2 times a day.

The calves are bent, easy to wake up when sleeping, love to sweat (medically called night sweats), observe your description, the child is indeed caused by calcium deficiency, as for rickets, you had better take the child to the hospital for a serum test, see what the people there say first. **The earliest and most timely way to treat rickets is to supplement calcium and VD and exercise more. Eat calcium tablets, let your child eat some high-calcium tablets every day - calcium edge.

This is good for calcium deficiencies such as rickets, chicken breasts, and looped legs.

Rickets is a chronic calcium deficiency condition, ** be patient, in a word: calcium supplementation + exercise.

My child's night sweats are calcium, and I feel okay, but I have to eat a bottle for a long time.

Eat more foods that contain vitamin D, such as animal liver, eggs, milk, etc.

Analysis: Hello, there are two possibilities for the child's condition, one is that calcium deficiency causes rickets, and the other is that there are local lesions in the joints.

Guidance: You now need to check the child to confirm the specific situation, it is recommended to check the X-ray of the knee joint, and check the trace elements, so that you can confirm the specific situation, and then actively treat the symptoms** to return to normal as soon as possible.

Analysis: Hello, the early symptoms of rickets are hyperhidrosis, night terrors, occipital baldness, etc., and later there will be costal valgus.

Guidance: Late teething, O, X-shaped legs, etc., heavy chicken breasts, late closure of the breganelle, etc., vitamin D should be supplemented, as well as calcium, etc., and more sunlight.

After the occurrence of rickets, the early manifestations of the child are excessive sweating, crying, sleeplessness, and irritability, and the head is itchy due to excessive sweating on the head, and the child often shakes his head and causes baldness on the headrest.

The above manifestations can only suggest that parents and children may have rickets, and they need to take their children to the hospital for further examination, and do not give their children a large amount of vitamin D supplements to prevent vitamin D poisoning. If the disease progresses further, it can be seen that the child's muscles are relaxed and weak, especially the abdominal wall and intestinal wall muscles, which can cause the child's intestinal distention, and cause the abdomen to distangle like a frog's belly. The most predominant change in children with rickets is the symptoms that occur due to skeletal lesions, which are characteristic of rickets.

For children under 6 months old, gently press their occipital or parietal bones with their fingers, just like a table tennis ball**; 8 9-month-old children have a square head and a large anterior fontanelle, which cannot be closed by 18 months. In children around 1 year old, the junction between the ribs and the costal cartilage can be seen in the chest to bulge like beads, which is called rib beads; Thoracic deformities such as anterior sternal protrusion, "chicken breast" and valgus of the costal margin may occur. Due to the weakness of the limbs and back muscles, the child sits, stands, and walks later than a healthy child, and is prone to falling, and after the age of 1 year, the lower limbs can be bent inward or outward, and the deformity is "0" or "X" shaped legs.

In addition, teething is delayed and prone to tooth decay.

Vitamin D is an effective drug for rickets, generally children can be given oral vitamin D pills, and children who cannot take oral or have diarrhea can inject vitamin D under the guidance of a doctor, and add calcium at the same time. At the same time, let the child bask in the sun more, the baby continues to breastfeed, add complementary food in time, and feed reasonably; To prevent deformity, do not let your child stand and sit for long periods of time, and do not let your child walk too early.

Vitamin D deficiency rickets, referred to as rickets, is a systemic chronic nutritional disease characterized by bone lesions due to abnormal calcium and phosphorus metabolism caused by vitamin D deficiency, which makes the growing epiphyseal cartilage plate unable to calcify normally, resulting in bone lesions.

So vitamin D supplementation is key.

The child is now 5 years old and has missed the best period. In the early stage**, the disease is controlled, and skeletal deformities can be prevented to a certain extent. While the child is not very old now, go to the hospital again, after all, this is related to the child's life.

1. Outdoor exercise for a certain period of time every day, directly exposed to sunlight, to promote the production of endogenous vitamin D.

2. Supplement vitamin D. Give foods rich in vitamin D, calcium, phosphorus, and protein.