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Gout is a syndrome, not a disease. Gout is a chronic disease, and patient long-term medication is the only way to succeed. If you stop taking uric acid-lowering drugs for a period of time because gout no longer attacks, the uric acid in the blood will rise again soon after, and usually gout will recur from time to time!
Hyperuricemia that has never had an episode usually does not require medication!Most asymptomatic hyperuricemia does not require medication, but the underlying cause should be identified, and diet** can be used to lower uric acid and prevent gout attacks. If you've ever had a seizure, start positively**!
If there is an arthritis attack, it is necessary to start low-purine food control, and if there is another attack, it is necessary to start using uric acid-lowering drugs, so that the gout will not recur and the complications of gout can be avoided. Even if it is not painful now, it is still necessary to take medicine for a long time, because according to the observation of gout patients who do not have **, almost all of them will get tophi, although tophi will not be painful most of the time, but its destruction of the joints is continuous.
Guidance: ** The patient should first distinguish and understand which painkillers or uric acid-lowering drugs are used, and the timing of their use is completely different. Painkillers are used when there is sudden acute pain in the joint, and they should be stopped when there is no pain, while uric acid-lowering drugs generally have no pain-relieving effect, but they can reduce uric acid to the normal range and prevent gout attacks, so they should be taken daily.
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Gout is a crystal-related arthropathy caused by long-term hyperuricemia, which is directly related to hyperuricemia caused by purine metabolism disorders and decreased uric acid excretion, and it belongs to the category of metabolic rheumatism. Gout can be complicated by kidney disease, severe bone destruction, kidney function damage, lead often accompanied by hyperlipidemia, hypertension, diabetes, atherosclerosis, coronary heart disease and other diseases.
In recent years, epidemiological surveys of gout attacks have shown that the incidence of gout is gradually showing a trend of younger people, with a male-to-female ratio of 15:1, and more than 50% of gout patients are obese and overweight. The level of uric acid in the blood at the time of the first gout attack can be as high as 527 mol L in men and 516 mol L in women.
The main reason for patients with gout is joint pain, which is close to 40% of men and 30% of women. In addition, fatigue and fever are present.
The causes of gout vary greatly depending on the gender. The main triggers common in men include alcohol consumption, high-purine diet, and strenuous exercise, while in women, the main triggers are high-purine diet, sudden coldness, and strenuous exercise. For gout, the first thing is to lower uric acid, and the uric acid level is controlled to a normal and reasonable range, which has good benefits for the acute attack of gout, including the progression of gout.
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Gout is a metabolic disease caused by a disorder of uric acid metabolism in the body that causes uric acid to accumulate in the joints and soft tissues, causing symptoms such as pain, redness, swelling, and inflammation.
The main symptoms of gout include:
Acute gouty arthritis: manifested as sudden onset of joint redness, swelling, heat, pain and dysfunction of the radical balance, mostly in the thumb, metatarsophalangeal joint, wrist joint, etc.
Chronic tophi disease: manifested by the gradual appearance of tophi, stiffness and dysfunction of the joints, commonly found in the ear wheels, between the fingers, between the toes, elbows and knees.
Uric acid nephropathy: It is characterized by high uric acid concentration in urine examination, accompanied by renal impairment and changes in kidney structure, and is common in pyelonephritis, kidney stones, and kidney cancer.
Urate uric tract stones: manifested as high uric acid concentration in urine examination, accompanied by symptoms such as urinary tract stone formation and obstruction, common in kidney stones, ureteral stones, and bladder stones.
Joint deformity and dysfunction: The high uric acid state during the Changming period can lead to the destruction of articular cartilage, causing joint deformity and dysfunction.
In addition, gout may also cause other symptoms such as fever, anorexia, headache, nausea, etc.
The ** of gout mainly includes drugs ** and non-drugs **. Drugs** include drugs that inhibit uric acid production and drugs that promote uric acid excretion, and non-drugs** include diet control, exercise**, drug conditioning, etc. Timely control of high uric acid status can effectively prevent the onset of gout and**.
Focus on sharing knowledge about high uric acid and gout**.
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Gout attacks are usually in the joints, especially the toes and fingers, and the attacks are red, swollen, burning, swollen and accompanied by severe pain. During the acute phase of a gout attack, you should rest more, elevate the affected limb to help local blood return, and do not wear squeezing or uncomfortable shoes to reduce joint swelling and pain. When joint redness, swelling, heat and pain are severe, you can take some anti-inflammatory painkillers to relieve the symptoms.
After the pain is gone, you must regret going to the hospital for examination in time, and the doctor will give you a detailed judgment of the symptoms of gout and the best method.
If not treated in time, excessive uric acid in the body will make the joints of the hands and feet, the pinna and other places have stone-like substances, which is a manifestation of long-term repeated deposition of uric acid, and generally tophi will occur in patients with poor long-term gout control. The symptoms and methods of gout mainly start from the three aspects of medicine, diet, and lifestyle.
First of all, it is necessary to use drugs rationally according to the doctor's instructions, eat less high-purine foods in terms of diet, such as animal offal, seafood and fish, various meats, etc., and learn to combine work and rest and learn to relax oneself in terms of life and rest. Finally, regardless of whether the disease occurs or not, you should always measure your uric acid value to avoid a sudden attack of high uric acid. I bought myself a Sino uric acid meter, and I can often measure my urine finger to change acid at home, which is very convenient.
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Gout is a metabolic disease, which is caused by some metabolic abnormalities in the body, the synthesis of too much uric acid or the excretion function of the kidneys in the body, and some of the uric acid in the body cannot be fully excreted from the body, which leads to an increase in the level of uric acid in the blood.
Under normal circumstances, if you go for a blood test on an empty stomach and the blood uric acid exceeds 420 mol L, it is called hyperuricemia, and when the blood uric acid continues to increase in the blood, and when the blood uric acid is higher than the amount of blood uric acid that can be dissolved in the blood, a part of the uric acid will be deposited in the form of crystals into the joints, which will cause arthritis. If urate crystals are deposited in the kidneys, they can also cause gouty nephropathy and even cause some damage to kidney function. Gout is a metabolic disease, the main cause of which is the increase of blood uric acid, so after having gout, it is necessary to actively control blood uric acid, minimize damage to organs, and reduce the occurrence of complications.
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What is Gout?
In simple terms, gout is a joint disease caused by the deposition of monosodium urate in the joints, which is mainly related to the dysfunction of purine metabolism and the decrease of uric acid excretion, which can occur secondary to hyperuricemia. Its symptoms can be divided into several stages: acute onset arthritis, tophi chronic arthritis, and urate nephropathy. In severe cases, it can also induce a variety of complications such as renal insufficiency.
Gout**.
1.If primary gout is found to be primary gout, it is usually caused by congenital factors such as genetic inheritance. Moreover, it has been found that gout may be a polygenic genetic disease, mainly caused by a deficiency in purine metabolizing enzymes.
2.Secondary goutSecondary gout is usually caused by a pre-existing myeloproliferative disorder, such as leukemia and multiple myeloma, but can also be caused by kidney diseases such as tubular nephritis and polycystic kidney disease. In addition, misuse of drugs such as thiazide diuretics, furosemide or pyrazinamide can also increase the risk of gout.
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Dandelion and it soaked in water together, diuretic, excrete excess uric acid, and prevent gout!
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