Can otosclerosis cause neural hearing loss?

It can cause significant tinnitus in about 80% of people with otosclerosis. Tinnitus can occur before or at the same time as deafness. Tinnitus is generally bass-based, but some are high-pitched, continuous or intermittent.

When the cochlea is degenerated, the tinnitus is more severe and persistent, and patients often feel that tinnitus is more painful than deafness, because the ears are constantly screaming day and night, and the tinnitus is more frequent when they need to rest in a quiet environment, which makes many people with otosclerosis extremely annoying. Hazard 2: Cause deafness, affect hearing If the lesion of otosclerosis is limited to the stapes floor, the sound conduction is blocked, which is manifested as conductive hearing loss.

If the otosclerosis lesion invades the cochlea, the round window and vestibulocochlear nerve endings add the component of sensorineural suction disorder, which becomes mixed hearing loss, causing one of the greatest harms to patients with otosclerosis. In advanced stages, the lesion invades the inner ear, and both air conduction and bone conduction hearing are drastically reduced. Otosclerosis develops at different rates.

The fast development can be.

Total deafness within 2 or 3 years; If the development is slow, it can be more than ten years after the onset of the disease, and the hearing impairment is still limited to conductive deafness. The second harm of otosclerosis patients.

The main clinical manifestation of otosclerosis is to cause patients to develop slow progressive deafness, otosclerosis is a major lesion of unknown origin in the bone labyrinth, in the bone labyrinth of the envelope to form one or several limited attachment to the blood vessels of the new bone, and gradually harden, invade the normal bone, so it is also called otospongisis.

The typical manifestations of otosclerosis are slow progressive deafness, which is generally conductive in the early stage and mixed in the late stage, and bilateral onset is common, and patients also have prolonged tinnitus, which usually occurs at the same time. When the lesion is further exacerbated and invades the vestibular nerve, the patient experiences transient vertigo.

In the early stage of otosclerosis, sodium fluoride can be taken orally, and the effect is good. However, if conservatively** is ineffective and the patient has residual hearing, local surgery** can be performed.

If the otosclerosis is not properly obtained**, it can lead to neural hearing loss, a condition that arises.

1. Symptoms of nervous hearing loss.

After the patient develops neural deafness, the auditory nerve and auditory center of the inner ear will be damaged, which will cause hearing loss or even hearing loss. Patients with neural hearing loss may experience tinnitus. In addition, some patients may even have progressive hearing loss in both ears or one ear due to hearing loss, resulting in deafness and even a feeling of congestion in the ears.

Some patients will have dizziness, and even nausea and vomiting, which will affect their normal life and rest.

At the same time, the appearance of neural deafness is not only a problem with the nerves, but also more likely to be caused by the lesions of the cochlea, the central nervous system will have some lesions, not only referring to the problem of the auditory nerve, and different types of neural deafness will also have different degrees of manifestation, most patients will be accompanied by tinnitus, severe chiropractic poor hearing loss or even disappearance.

2. How to have neural deafness**.

1. Surgery**.

There are two ways for patients with neurological hearing loss**, the first is to perform surgery on the lesions in the ears to restore normal hearing. The second option is to place a cochlear implant in the ear so that the patient can regain the sound of the outside world.

2. Auditory language training.

Patients who have not completely lost their hearing need to use the maximum amount of residual hearing in the body to train and mobilize other sensory organs to train the ability to speak and speak, the two complement each other and cannot be neglected. Some younger children need to be exercised from an early age.

Due to the lesion of the cochlear nerve, the electrical activity of the auditory hair cells cannot cause the proper excitation of the cochlear nerve, or the process of transmitting nerve impulses caused by electrical activity to the brainstem is impaired. There are also deafness caused by cochlear neurological deafness caused by lesions of the cochlear Shenbi bradymenorrhea and auditory central pathway, which are collectively referred to as postcochlear deafness, and the resulting tinnitus is called neurogenic tinnitus. The reasons for it include:

1. Congenital hereditary inner ear dysplasia;

2. Deafness caused by ototoxic drugs**;

3. Spring deficiency otogenic labyrinthitis caused by bacterial or viral infection, such as meningitis, mumps, measles, labyrinthitis caused by epidemic ** and spirochetal syphilis infection;

4. Trauma includes knock injury, noise injury, rib fracture and surgical injury;

5. Tumors, such as acoustic neuroma, cerebellar pontine angle tumor, etc.;

6. Presbycusis deafness, caused by physiological aging and degeneration of hearing due to age;

7. Metabolic deafness, various diseases or factors that affect cochlear metabolism, such as diabetes, hypothyroidism, renal failure, hyperlipidemia, etc.;

8. Unexplained sudden deafness.

In the early stage of tinnitus, the 3rd to 6th month is an important period.

First, the primary disease that causes tinnitus.

Second, drugs**, vasodilators, calcium ion antagonists, tinnitus inhibitors, etc. can be used to reduce tinnitus, and neurotrophic drugs can be used.

Third, psychological counseling, many tinnitus patients have more or less psychological factors.

Fourth, concealment, which can be carried out with tinnitus detectors, tinnitus concealers, pure tone audiometers or hearing aids.

Fifth, the habit of tinnitus**.

Sixth, auditory arguments**, which can be used in traditional Chinese medicine**.

Mainly due to inner ear lesions.

It may be related to your rest, or it may be that you have a cold caused by rhinitis, pharyngitis, otitis media, and they are interconnected.

Neural hearing loss is generally caused by damage to the inner ear or auditory pathways, or the auditory center, resulting in hearing loss or loss. There are many causes of neural deafness, such as drug-induced deafness, deafness caused by ototoxic drugs, and all of them are neurological deafness. Shockwave injuries, such as those caused by detonation injuries, can damage the hair cells of the inner ear, leading to neural hearing loss.

There is also deafness caused by viral infections, etc., and it can also be neurological deafness. Neurological hearing loss can also occur due to injury to the inner ear or to the auditory cortex. Presbycusis is most likely caused by the decline of auditory organs such as the inner ear, and can also lead to neural hearing loss.

Basic congenital factors, such as genetic or chromosomal abnormalities, lead to defects in the development of hearing organs, resulting in hearing impairment. Fetal hearing impairment caused by maternal or childbirth factors during pregnancy. Common ototoxic drugs for drug poisoning include aminoglycoside antibiotics such as streptomycin and gentamicin, glycopeptide antibiotics such as vancomycin, and antitumor drugs such as nitrogen mustard, carboplatin, cisplatin, etc.

In addition to external factors such as drug dose, drug time and route, drug poisoning is also related to internal factors such as individual differences. The degenerative factors of aging are associated with aging (usually over 60 years old), and are mostly caused by atrophy of spiral ganglion cells or changes in the properties of the basement membrane of the cochlea. Autoimmune factorsSensorineural hearing loss caused by damage to inner ear tissue due to autoimmune disorders, which is progressive and fluctuating.

May be accompanied by tinnitus and vertigo. Acoustic neuropathy Acoustic neuropathy is a disease with special clinical manifestations, and the main audiological features include absence or severe abnormality of auditory brainstem responses, normal otoacoustic emissions, absent or elevated stapes reflex, and pure-tone audiogram with low-frequency hearing threshold loss. Viral or bacterial infections affect the auditory system and can cause unilateral or bilateral non-fluctuating hearing loss, and the most common deaf-causing infections are mumps, epidemics.

Usually, sensorineural hearing loss occurs in the inner ear or behind the cochlea, which will cause hearing loss, poor speech recognition and comprehension, inaudible or easy to hear clearly, inaudible in small voices, inaudible in loud voices, tinnitus and vertigo. Sensorineural hearing loss is congenital and includes vestibular aqueduct enlargement, as well as acquired and acquired hearing loss including senile, drug-induced, noise-infected, infectious, sudden, and Meniere's disease.

Neurological deafness may occur due to the following causes: Neurological deafness caused by drug poisoning caused by continuous use of antibiotics such as streptomycin, kanamycin, gentamicin, etc. Sudden deafness caused by a viral infection or embolism of the inner ear vessels.

Infectious deafness caused by infectious diseases such as meningitis, measles, typhoid, etc. Deafness caused by trauma or knocking, noise, etc. Traditional Chinese medicine believes that the ear is the orifices of the kidneys, which are perfused by the 12 meridians and pass through the brain.

Its pathogenesis can include wind and heat invasion, liver fire disturbance, phlegm and fire stagnation, kidney essence loss, spleen and stomach weakness and so on. The main clinical manifestations are hearing impairment, loss or even disappearance, and children often feel cicadas or other sounds in their ears, which is more obvious in a quiet environment. It can be accompanied by a variety of systemic symptoms such as fever, headache, irritability, abdominal distension, and backache and fatigue.

Neural hearing loss is caused by the lesions of the sound wave receptor and analysis pathway, the auditory nerve of the inner ear and the auditory center, which hinders the perception of sound or the transmission of sound information, and the resulting hearing loss or hearing loss, which is called neural hearing loss. Genetic factors account for about 55% of neural hearing loss, and 45% of cases are unknown, and the types of diseases are as follows:

1. Drug-induced deafness, sensorineural deafness caused by the use of antibiotics, salicylates, antineoplastic drugs, etc.

2. Sudden deafness, sensorineural hearing loss of unknown cause.

3. Infectious deafness mainly refers to hearing loss caused by the invasion of pathogenic microorganisms such as bacteria and viruses in the auditory nervous system.

4. Hereditary deafness, hearing impairment caused by abnormal development of hearing organs caused by genetic or chromosomal abnormalities and other genetic defects.

5. Presbycusis, deafness caused by degeneration of the auditory system due to aging.

6. Autoimmune deafness, which mostly occurs on both sides of young adults, occurs at the same time or successively appears asymmetrical, fluctuating, and neurological deafness, and 7. Auditory neuropathy is a kind of cochlear hair cells and auditory nerve protrusions, and auditory dysfunction caused by its own dysfunction, and patients often start the disease since childhood.