Do you know what are the characteristics of angina pectoris and myocardial infarction?

The pain of angina pectoris and myocardial infarction is very severe, and the pain is generally unbearable, and it lasts for a long time, and may be accompanied by shock, heart failure, and arrhythmia, which is very dangerous.

Frequent heartache, inability to do strenuous exercise, frequent palpitations, frequent heart palpitations, these are all characteristics.

Yes, colic can develop into myocardial infarction! But not all angina pectoris will develop into myocardial infarction, and there are many ways to prevent angina from developing into myocardial infarction! What is myocardial infarction?

It is the complete blockage of the blood vessels of the heart, leaving no blood flowing, causing myocardial necrosis in the most severe coronary heart disease. Simple understanding: this is the narrowing of the blood vessels that do not clog angina, and once blocked, it will develop into a heart attack.

Classification of angina: Angina pectoris is divided into exertional angina and unstable angina.

Exercise-induced angina: mostly stable angina, which can be induced only when angina occurs after activity, after going to work, or after overwork, which can increase the burden on the heart and lead to aggravation of myocardial ischemia. The vast majority of angina pectoris, which can be controlled with medication, is to control the heart rate at rest up to about 60 beats; Control blood sugar, blood sugar, blood lipid levels, and quit smoking.

By doing so, most excessive angina is controlled, little progresses to myocardial infarction, and coronary angiography is not required. But if you quit smoking, take your medication on time, your heart rate and blood pressure are up to standard, but you still have seizures, or your activity level decreases, then we call it worsening angina, and you should start with unstable angina**.

Unstable angina: All unstable angina pectoris, on the one hand, mostly occurs at rest and slightly worsens with activity. The most important plaque in the blood vessels of the heart is an unstable plaque that can rupture at any time, and once ruptured, blood clots will form and myocardial infarction can occur, that is, angina pectoris develops into myocardial infarction.

For unstable angina, in addition to improving routine, quitting smoking, and taking medications, for most angina that cannot be controlled by drugs, further coronary articulated lithography should be performed, and on this basis, revascularization (i.e., bypass grafting or stenting) should be determined. Can angina develop into myocardial infarction? How do doctors teach you about prevention?

Prevention of coronary heart disease: To prevent coronary heart disease, it is mainly a little bit of life. Start with a healthy diet with more fruits and vegetables, less red meat, less fatty meat, and more fish; a low-salt, low-sugar diet; Adhere to aerobic exercise, quit smoking and limit alcohol, control weight, and actively control the three highs.

Once the symptoms of crown coarse heart disease appear, it is necessary to see a doctor in time and make a plan to avoid myocardial infarction. Actively control hypertension, diabetes, hyperlipidemia, and add medication if necessary; A healthy lifestyle is the most fundamental way to prevent angina from progressing to myocardial infarction. Can colic develop into myocardial infarction?

How do doctors teach you about prevention? In short, angina pectoris should be noted regularly**, sometimes angina eccentric muscle infarction is one step away; But if it's under control, the heart attack won't stop!

1.Angina pectoris is a clinical syndrome with episodic chest pain or chest discomfort caused by coronary insufficiency, acute, temporary ischemia and hypoxia of myocardium. It is characterized by paroxysmal anterior chest squeezing pain, which may be accompanied by other symptoms, mainly posterior to the sternum, radiating to the precordium and left upper extremity, often during exertion or emotional agitation.

2.Myocardial infarction refers to the avascular necrosis of the myocardium, which is a sharp reduction or interruption of the blood flow of the coronary arteries on the basis of coronary artery lesions, so that the corresponding myocardium has severe and lasting acute ischemia, and eventually leads to avascular necrosis of the myocardium.

One is coronary insufficiency, the other is myocardial avascular necrosis, but generally speaking, it is cardiac ischemia, one blood vessel is insufficient, and the other is myocardial ischemia.

Angina pectoris has the following two characteristics:

1.Systolic colic in the precordial area does not last more than 15 minutes;

2.The colic is quickly relieved after sublingual administration of acid-glycerin tablets.

Acute myocardial infarction has the following four characteristics:

1.Precordial colic is more intense, unbearable, often accompanied by irritability;

2.Colic lasts more than 15 minutes, some up to half an hour or more;

3.angina does not decrease after rest;

4.Colic does not relieve after sublingual nitroglycerin tablets. Once the patient has myocardial infarction, he should immediately go to the hospital for diagnosis and treatment without delay.

From the perspective of pain time, angina pectoris is short and can be relieved with medication or rest, but myocardial infarction pain is long and intense, and medication cannot relieve it or the relief is slow.

From the point of view of lesion loss, angina pectoris does not have significant damage to the myocardium, whereas myocardial infarction is avascular necrosis of the myocardium and is more difficult to recover from.

From a laboratory point of view, myocardial infarction has enzymatic changes.

Angina pectoris is caused by transient ischemia of the heart muscle, but if it is too long, it can cause necrosis of the heart muscle, which is called myocardial infarction. The difference between the two is that the duration of pain is not the same - angina pectoris, which can be relieved after resting after about a few minutes, can be relieved by taking nitroglycerin, but myocardial infarction will take more than 30 minutes, and eating nitroglycerin does not relieve the ECG. If myocardial infarction is suspected, it is important to go to the hospital as soon as possible, as acute myocardial infarction has a high mortality rate.

It should be diagnosed correctly and correct**. There is a big difference between the two, and angina pectoris can develop into myocardial infarction under certain circumstances.

Angina pectoris is relieved by oral nitrates for 5-10 minutes.

If the heart attack does not go away, it may be a heart attack if it lasts for 30 minutes.

Angina pectoris: the main clinical manifestations are episodic chest pain, ST-segment depression of subendocardial ischemia is common on electrocardiogram, and there are risk factors for coronary heart disease.

Myocardial infarction: more intense in nature, lasting up to several hours, with arrhythmias, heart failure shock, and abnormal G-band elevation of the ST-segment in the ECG towards the infarction site.

The difference is here, I hope it works for you.