What are the clinical manifestations of cor pulmonale and heart failure?

Cor pulmonale heart failure is mainly right heart failure, and its clinical manifestations are as follows:

1) Symptoms: The patient has shortness of breath, palpitations, palpitations, increased heart rate, cyanosis and dyspnea further aggravated, and even orthopnea, epigastric distension and pain, ascites, oliguria or anuria, edema of both lower limbs, and loss of appetite, nausea, vomiting, liver pain, and even jaundice.

2) Signs: jugular venous distension or distention. In the semi-recumbent or sitting position, the external jugular vein can be filled with the external jugular vein above the clavicle, and in severe cases, the arm or other superficial veins can also be filled and distended;

Liver enlargement and tenderness. Right-sided heart failure, jaundice with elevated aminotransferases, can lead to cardiogenic cirrhosis, often accompanied by jaundice and ascites;

Dropsy. It is a drooping depression, with the inner side of the foot and ankle and the anterior tibia more obvious, and in severe cases, it can develop into generalized edema;

Pleural effusion. Right-sided pleural effusion is more common, but it can also be bilateral;

Ascites. Ascites mostly occurs in advanced stages;

Cyanosis. It is more common in patients with long-term right-sided heart failure and is peripheral cyanosis.

These are mainly damage to the heart muscle and an increase in the volume load of the body. There are two aspects of myocardial damage: 1. Primary myocardial damage, including myocardial ischemia, such as myocardial infarction and coronary heart disease.

Myocardial damage caused by inflammation or immune reactions to the heart muscle, such as myocarditis or dilated cardiomyopathy, as well as hereditary diseases, such as hypertrophic cardiomyopathy and myocardial densification, can cause myocardial damage. 2. Secondary myocardial damage, such as metabolic cardiomyopathy, such as diabetes mellitus and hyperthyroidism, and then systemic psychiatric diseases, such as myocardial amyloidosis, resulting in myocardial damage. There are also connective tissue disorders, as well as cardiotoxic drugs, that can also cause damage to the heart muscle.

In addition, cardiac overload, such as long-term renal insufficiency, leads to limited drainage and increased myocardial volume load, which can also cause myocardial damage and induce heart failure. There are also long-term hypertension, pulmonary hypertension, and aortic stenosis, as well as coronary valve insufficiency, anemia, or congenital cardiovascular disease, which can cause an increase in heart workload. In addition, some mitral valve stenosis, or cardiac tamponade, and pericarditis, can also cause heart failure.

Common causes of heart failure include severe hypertension, coronary heart disease, cardiomyopathy, congenital heart disease, etc. Recently, my grandmother has been saying that she is weak, it is becoming more and more difficult to walk, she is always dizzy, her chest has always been stuffy and painful, and she always feels breathless when she lies down and sleeps at night, and sometimes she needs to be half-lying to fall asleep. The family was really worried, so they took their grandmother to the hospital for a check-up, and the grandmother had been hospitalized for a minor heart attack, and the results of the examination came out, saying that she had already developed heart failure.

The doctor prescribed Qili Qiangxin Capsule to my grandmother, saying that it is good for temperature and yang, blood circulation and circulation, water dilution and swelling, which can alleviate the symptoms of heart failure such as asthma, fatigue and edema caused by heart failure, and can also improve heart function.

<> heart failure is a group of complex clinical syndromes caused by abnormal changes in the structure or function of the heart caused by a variety of reasons, which causes dysfunction of ventricular contraction and diastolic function, mainly manifested as respiratory distress, fatigue and fluid retention (pulmonary congestion, systemic congestion and peripheral edema), etc., and the above drawings are symptoms of heart failure.

(1) Early symptoms of heart failure: manifested in general physical activity, the patient feels fatigue and weakness of the limbs, which can be reduced or eliminated after rest. When doing slightly strenuous activities, such as:

If you walk a little faster and go upstairs, you will have difficulty breathing, and you will need to stop and rest for a while before moving on. These findings suggest early symptoms of left-sided heart failure. As the disease progresses, heart function gradually declines, and even when doing some light physical work or activity, you will feel short of breath and difficulty breathing.

When you sleep at night, you always feel that the pillow is low or you want the mattress on the side of your head to be higher. Sometimes you are awakened by shortness of breath during sleep, and you can fall back asleep after getting up and sitting for a while. The early symptoms of right heart failure are mainly loss of appetite, abdominal distension and discomfort, sometimes pain in the right upper quadrant, decreased urine output, gradual weight gain, and tightness in the hands and feet.

2) The main signs of early heart failure are: the liver begins to stasis and enlargement; jugular venous distention; early diastolic gallop rhythm; "mobile" rales at the base of the lungs; Rapid heart rate; Hypersecond heart sound is heard in the pulmonary valve auscultation area; Alternate pulses appear, etc.

Heart failure is divided into left heart failure and right heart failure. Left heart failure is mainly pulmonary congestion, manifested by dyspnea, coughing and wheezing, chest tightness, and a feeling of suffocation.

Heart failure is heart failure, which is divided into acute heart failure and chronic heart failure, and the specific symptoms are as follows:

1. Acute heart failure: including acute left heart failure, acute right heart failure and total heart failure, etc., the onset is more acute, often manifested as extreme dyspnea, orthopnea, frequent cough, and coughing up a large amount of white or pink foamy sputum, pale, bruised, sweating, and thin pulse; Some may have a fast heart rate, high blood pressure, and to a certain extent, signs of shock, mainly cardiogenic shock;

2. Chronic heart failure: compared with acute heart failure, the course of the disease is relatively long, and it is often manifested as chronic dyspnea, cough, sputum production, chest tightness, shortness of breath, pulmonary congestion, pulmonary edema and edema of both lower limbs.

The decompensated symptoms of cor pulmonale are twofold:

1. Respiratory failure: dyspnea progressively worsens, especially at night, often with headache, insomnia, decreased appetite, daytime mental trance, drowsiness, and symptoms of pulmonary encephalopathy.

2. Heart failure: Qi promotes aggravation of walking. Heart signs such as palpitations and nausea. or edema of the lower extremities, or even ascites.

In the late stage, I got **ah, compound licorice drama spicy soup** pulmonary heart disease is good. **The effect is also good.

1: The concept of chronic cor pulmonale, and **;

1 Concept. Chronic cor pulmonale (cor pulmonale) refers to the chronic lesions of the lungs, thoracic cage or pulmonary arteries, resulting in increased pulmonary circulatory resistance and increased pulmonary artery pressure, resulting in right ventricular hypertrophy and enlargement, with or without right heart failure.

2．**。1) Bronchial and lung diseases: chronic bronchitis (referred to as chronic bronchia) complicated by obstructive emphysema is the most common type of cor pulmonale, accounting for 80% to 90%. It is followed by bronchial asthma, bronchiectasis, severe tuberculosis, pneumoconiosis, and diffuse pulmonary interstitial fibrosis.

These conditions can lead to ventilation and ventilation dysfunction.

2) Thoracic dyskinesia disease: rare, including spinal deformity caused by various reasons, as well as severe thoracic deformity caused by extensive pleural thickening and adhesions, mainly leading to restrictive ventilatory dysfunction. In addition, due to lung compression, the bronchial tubes are twisted or deformed, resulting in poor airway drainage, recurrent lung infections, and emphysema or pulmonary fibrosis, which develops into pulmonary heart pain.

3) Pulmonary vascular disease: rare. Diseases such as multiple pulmonary arteriolar embolism and pulmonary arteritis can cause narrowing and blockage of pulmonary arterioles.