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Chicken taeniasis is caused by a variety of tapeworms of the genus Lyres parasitic in the duodenum of chickens, and the common reliant tapeworms include Reilly tapeworm in the groove of spinos, Reilly tapeworm quadrangularis and Reilly tapeworm in wheels.
Because various tapeworms such as Reilly tapeworm in the groove are parasitic in the small intestine of chickens, the integrity of the intestinal wall is destroyed with the scolice, causing mucosal bleeding, intestinal inflammation, and seriously affecting digestive function. Sick chickens present with diarrhea, sometimes mixed with bloody mucus in the feces. Mild infections cause stunted chick development and adult egg production decreases or stops.
When the number of parasitic tapeworms is high, it can block the intestinal tube, and the passage of intestinal contents is blocked, causing intestinal rupture and peritonitis. Tapeworm metabolites can cause poisoning and neurological symptoms in chickens. Sick chickens lose their appetite, are depressed, anemic, have pale combs and mucous membranes, are extremely weak, often paralyzed in both feet, unable to stand, and finally die of exhaustion.
Young chicks are severe, and adult chickens are lighter.
Sick chickens are not energetic, their appetite increases in the early stage, and when they are poisoned, they lose their appetite, but their appetite increases, they are emaciated and anemic, their feathers are loose, and they are discharged with white thin feces with mucus and foam, mixed with white tapeworm proglottids.
When seriously infected, some cases often have progressive paralysis, starting from the two feet, gradually affecting the whole body, that is, paralyzed chickens, sometimes some cases after a period of time after the symptoms of chicken poisoning are lifted and do not heal themselves, but affect the future productivity.
Adult chickens infected with the disease generally do not show symptoms, but affect the production of antibodies when immunization vaccines, in severe cases, the egg production decreases or the egg production rate fluctuates, individual serious cases appear in the abdomen that is, water stall chickens and neurological symptoms that are paralyzed chickens, often due to the stimulation of bacterial or viral diseases and failure and death.
The diagnosis of chicken taeniasis is usually done by autopsy: cut open the intestine, and in sufficient light, white bands or scattered proglottids can be found. If the intestine is placed in a large dish of water with a black background, the worms are more recognizable.
Because the scolices of tapeworms are extremely important for species identification, they should be carefully sought. To peel off the scolice, the mucosa with the head segment can be deeply cut with a scalpel and the mucosa is peeled off with two needles under a dissecting scope. In elongated hymenoles, the scolex must be quickly picked out to prevent it from self-extricating.
Since chicken tapeworms must have a specific intermediate host involved in their life cycle, the key to preventing and controlling chicken tapeworm disease is to eliminate the intermediate host, thereby interrupting the life history of the tapeworm. Intensive chicken farms, with cage management practices that allow flocks to avoid intermediate hosts, can be an easy-to-implement preventative measure. It is more difficult to use pesticides to eliminate intermediate hosts.
Young chicks are raised separately from adult chickens, and finally an all-in, all-out system is adopted.
Medicated deworming is performed regularly, and prophylactic deworming is recommended at 60 and 120 days of age.
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First of all, it is necessary to ensure cleanliness and hygiene, the chicken manure needs to be removed, the treatment of the noodle camp must be carried out, some drugs can be sprayed for disinfection, and the chickens need to be dewormed, so as to ensure safety.
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It is necessary to ensure cleanliness and hygiene, clean some feces, some mosquitoes, and spray relevant solutions for disinfection.
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(1) Life history.
Adult worms parasitize in the small intestine of poultry, mature pregnant egg proglottids fall off automatically, excrete with feces, and are swallowed by suitable intermediate hosts, and develop into cysticerci with infectious ability in their bodies after 2 to 3 weeks. Birds are infected by eating this cysticerci-like intermediate host.
2) Popular features.
Taeniasis in poultry is widely distributed and harmful. Infections mostly occur in the middle of the intervening period from 4 to September. However, the susceptibility of chicks is stronger, and the morbidity and mortality of chicks at the age of 25 to 40 days are the highest, and the adult birds are mostly carriers of worms.
3) Clinical symptoms and pathological changes.
Chickens suffer from indigestion, dysentery, thin feces or mixed with bloody mucus, increased thirst, depressed spirit, drooping wings, backward feathers, emaciation, and slow growth. Severe anemia, pale mucous membranes and crown, and finally debilitating death. Laying hens even stop laying eggs.
Increased mucus, foul-smelling, thickened mucous membranes, and bleeding spots in the small intestine. In severe infections, worms can block the intestines. In the case of Reilly Spinosa infection, a depressed nodule can be seen on the intestinal wall, and the nodule contains a yellow-brown cheese-like substance.
4) Prevention and control programs.
In the stool, white rice-like proglottids can be found. In summer, when temperatures are high, proglottids can be seen squirming around the stool. Microscopic examination of this type of pregnancy may reveal a large number of eggs. Some seriously ill chickens can be diagnosed by autopsy.
Prevent and improve environmental sanitation and strengthen manure management. Pay attention to the infection situation at any time, and carry out drug deworming in time.
**The preferred drug, praziquantel, is 10 mg per kilogram of body weight once orally; Thiobis-dichlorophenol, at 150 200 mg per kilogram of body weight, chicks; Prothiobendazole, 20 mg per kilogram of body weight.
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Taeniasis is a parasitic disease caused by tapeworms that live in birds. The tapeworms parasitic in our country's poultry belong to a total of 21 genera of the order Cyclophylla Divinaceae, Bivalves, and Hymencapaceae, with lower animals as intermediate hosts, such as Davenia proglottids as the intermediate host is slugs or land snails, the intermediate host of Lyleys tapeworm is ant, the intermediate host of Lyleys tapeworm is houseflies, beetles and walking insects, etc., and the common ones in China are Denwen tapeworms and Lyleys tapeworms.
1.Prophylaxis. 1) Kill intermediate hosts on a regular basis.
2) Regular deworming, and harmless treatment of feces.
3) Regular anti-tapeworm drug prophylaxis.
2.**Praziquantel, niclosamide (ditafenol, Yumisheng, Xuefang 67), prothhimazole, etc., can be used, according to the instructions of the purchased anthelmintic drugs.
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**There are many drugs for this disease, mainly the following:
1) Thiochlorodichlorophenol (also known as allobutin) is 150 200 mg per kilogram of body weight, mixed into the feed and taken internally, and the chicks are reduced. When the whole group is treated, the medicine is mixed and fed in the feed, and the worm body can be discharged 3 to 7 hours after the treatment.
2) Chlorethaline is fed with 50 60 mg per kilogram of body weight, and can also be mixed into feed.
3) Areca nut decoction juice Add 1 gram of betel nut slices or powder to the chicken per kilogram of body weight, take it on an empty stomach in the morning, and then give enough drinking water, and remove worms within 1 3 days after taking the medicine. Nowadays, there are many new anthelmintic drugs on the market, with different names, which are generally used according to the doctor's instructions or new drug instructions, and can not be done on their own, so as not to affect production.
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Chicken taeniasis (cestodosis) is caused by a variety of tapeworms of the genus Lysley parasitic in the duodenum of chickens, and the common relily tapeworms include Reilly tapeworm in the groove of spinos, Reilly tapeworm quadrangular and Reilly tapeworm in wheel. Chickens of all ages can be infected, and other chickens such as turkeys, pheasants, pearl chickens, peacocks, etc. can also be infected, and chicks at the age of 17 to 40 days are the most susceptible and have the highest mortality rate.
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The larvae begin to form at the age of 50 days, and the adult worms are formed after 180 days, after the age of the tapeworm in the body the destruction and influence is very strong, first of all, the worm body absorbs nutrients in the body and destroys the normal digestion and absorption function of the intestines, resulting in the loss of nutrients in chickens, so that chickens produce eggs and low egg quality. Appearance: The cockscomb is white, the legs are white, which are the symptoms of anemia and seepage, and the intestinal tract is destroyed by insects and causes loose stools, causing unnecessary economic losses to the majority of breeding cracked households.
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