How is delayed myelin sheath treated? Delayed myelination Can myelination be cured?

In the process of growth and development of infants or children, there is myelin development retardation, myelin development delay or myelin development delay is also relatively common, such a situation, most children are due to obvious developmental delay, go to the hospital for routine cranial magnetic resonance examination, only to find that there are myelin development problems, the main method of such a situation can first consider the application of nutritional nerve drugs for assistance, such as mouse nerve growth factor, ganglioside, etc.

1. How to delay the development of myelin sheath**?

Myelin dysplasia refers to the impaired myelination due to the deficiency of certain enzymes during the development of the myelin sheath, also known as leukodystrophy. Common causes include adrenoleukodystrophy and metachromatic leukodystrophy, which are inherited metabolic diseases. At present, there is no better way to deal with these diseases.

Adrenocerebral leukodystrophy can be given with alternative adrenocorticotropic hormone** and symptomatic leukodystrophy**.

2. What is the cause of delayed myelin development?

1. Due to the influence of genetic factors, or local defects lead to myelination disorders, which affects the development of cerebral white matter and leads to the delay in the development of cerebral myelin sheath.

2. Delayed myelin development is due to the manifestation of cerebral dysplasia, which is caused by respiratory distress at birth and cerebral hypoxia, and the main emphasis should be on the early comprehensive system, delaying or even reducing the neurological deficits caused by myelin dysplasia in the white matter, and improving the patient's prognosis and quality of life.

To sum up: if there is a problem of myelin sheath retardation, it is necessary to go to the neurology department or **department** of the local tertiary hospital in time for early detection and treatment, so that the prognosis is better. If it has been a long time, it will be more difficult to recover.

If there is a problem of myelin retardation, it is necessary to go to the neurology department or **department** of the hospital in time for early detection and treatment, so that the prognosis is better. If it has been a long time, it will be more difficult to recover.

Myelin dysplasia usually doesn't get cured. Myelin dysplasia is usually caused by chromosomal abnormalities, genetic mutations and other factors, resulting in the failure of the myelin structure of the white matter of the brain to develop normally during the development of infants and young children, resulting in impaired nerve conduction function, which can lead to brain development disorders in children, accompanied by motor dysfunction and limited activity. Once the disease occurs, it is irreversible, and can only improve the symptoms through drugs and training, and try to improve the quality of life of the child.

1. Drugs**: For myelin dysplasia, glucocorticoids, such as dexamethasone acetate tablets, can play an anti-inflammatory and inhibition of immune response. Drugs that nourish nerve cells, such as vitamin B12 tablets, can also be used to promote the development of nerve cells; If there are secondary seizures, antiepileptic drugs such as sodium valproate tablets and carbamazepine tablets can be used to control seizures and avoid further damage to brain tissue;

2. Training: mainly including physical exercise training, skill training, language training, etc. It is mainly for the correct guidance of various movement disorders and abnormal postures of children, and to improve abnormal motor reflexes as much as possible.

Focus on fine motor training of the upper limbs and hands to improve children's life skills. In addition, it is necessary to carry out collaborative correction training for the child's language, pronunciation, and chewing and swallowing functions to improve the quality of life of the child as much as possible.

Women should strengthen prenatal examination during pregnancy to facilitate timely detection of abnormal conditions and facilitate early symptomatic treatment. Once it is confirmed that infants and young children have myelin dysplasia, they should actively cooperate with doctors to carry out medication** and functional ** training as soon as possible, and strive to maximize improvement and recovery.

Because the causes of the disease caused by delayed myelination are different, the effect is not particularly obvious, and individual symptoms will be partially relieved after control, and the effect may also vary greatly.

Normally, the myelin sheath is a membrane that wraps around the axons of nerve cells, acting as an insulating membrane. The first is to support the electrical insulation of axons from surrounding tissues, such as two adjacent axons, so as not to interfere with each other. At the same time, it can be called a skipping conduction mechanism that accelerates the transmission of electrical action potentials and can guide axonal regeneration in the case of axon damage.

If there may be a delay in development under the influence of the disease, some metabolic diseases or multiple sclerosis will have abnormal myelin development, and some diseases can be partially relieved with the improvement of the disease. For example, phenylketonuria may have a delay in myelination, and there will be a partial remission after **. The improvement of some diseases is not obvious, such as multiple sclerosis, which is also degenerative and aggravated, and it is not easy to alleviate it.

Once delayed myelin development is detected, it is recommended that the patient go to a regular hospital for examination, and magnetic resonance imaging can be used to find typical imaging features. Patients can eat more fresh vegetables and fruits, as well as foods that are high in vitamins.

There is no good way to have myelin dysplasia in the true sense. The myelin sheath is a structure that surrounds the axons of nerve fibers on the outside of nerve fibers, mainly to protect nerve axons and enhance the electrical conduction of nerves. Because the electrical conduction of nerves, in the process of myelin conduction, is skipping conduction, which can accelerate the conduction of nerves and accelerate the transmission of nerve signals.

The synthesis step of the nerve myelin sheath requires more than 30 biochemical reactions, and requires the assistance of a variety of chemicals and coenzymes to complete.

If the congenital dysplasia of myelin in the true sense is generally an enzyme defect, or an enzyme defect required for a certain reaction, or a synthase defect of a substance, or a defect in the action point of a certain coenzyme, if the enzyme defect is congenital, there is basically no solution. If congenital demyelination is diagnosed, the disease will basically last for life, and most of the myelin diseases will have severe neurological deficits, and most patients will have a relatively short lifespan.

1.Delayed myelin development refers to the fact that the development of the myelin sheath and the development of the cortex are not synchronized, and in the nervous system, the delay in the development of the myelin sheath leads to the development of malformations of the brain, which is manifested as gray matter malformation or symptoms of gray matter ectopia, which can be manifested as cerebral palsy, limb twitching, paresthesia or symptoms of seizures.

2.Patients also experience memory loss, unresponsiveness, poor numeracy, delayed speech development, and behavioral dysfunction. In addition, patients will also have limb mobility impairment, which is manifested by cerebral palsy-like symptoms and paresthesias, for such patients, the most important thing is to carry out symptomatic **, if there is epilepsy, anti-epileptic ** can be given.

Delayed myelination is a condition of myelination abnormalities and is generally considered to be delayed myelination of the white matter of the brain.

3.Myelination is the final stage of white matter development in the only wide brain. Delayed myelin development can lead to a child's brain development, often causing dizziness, headaches, and decreased cognitive performance.

Once we find that the development of the myelin sheath is delayed, it is recommended that the patient go to a regular hospital for examination, and the typical imaging features can be found through the magnetic resonance examination, and most of the patients' symptoms can be relieved to a certain extent through formal examination and **. Patients can eat a diet rich in fresh vegetables and fruits, as well as foods high in vitamins.

4.Delayed myelination in infants may be due to genetic factors. Myelin dysplasia is also known as white matter dysplasia.

It may be that in the process of myelination, due to the influence of genetic factors or hereditary defects of a certain enzyme, the formation of myelin disorder, this defect will affect the development of white matter in children, specifically manifested as developmental retardation, ataxia, convulsions, nystagmus, cortical blindness, optic nerve atrophy and sensory deafness.

Hello! Delayed myelination is a type of myelination abnormality, usually delayed myelination of the white matter of the brain.

Myelination is the final stage of white matter development, and the fetus forms myelin from the spinal nerve roots and notochord from the caudal to cephalad during the 3rd and 6th months of intrauterine life. At birth, a significant amount of myelin is already located in the brainstem, pontine brachi, hind limbs of the internal capsule, and the radiating crown in the center of the hemioval. The maturation process occurs mainly after birth and continues until the age of 20, and the myelin sheath of the white matter of the brain is remodeled throughout life.

Demyelinating lesions occur in children if they are affected by certain secondary factors such as viral infection, asphyxia, poisoning, autoimmune diseases, etc., or if some primary factors such as multiple sclerosis and Schilder's disease occur. Demyelination has a significant impact on the neurological development of children, often leading to disabilities such as mental retardation, epilepsy, language impairment, and limited mobility, and MRI can help diagnose them. In any case, it is recommended that you visit the neurology department of your local children's hospital, and long-term follow-up is required.

It is also a type of demyelinating disease.

Myelin dysplasia, also known as leukodystrophy, occurs when the white matter myelin sheath of the brain itself does not develop properly, so most of them occur in infancy, child, or adolescence. And there are often family genetic factors. Most people get better as the child grows and matures, and if the child has mental or physical decline, he or she needs to see a doctor.

Myelin dysplasia is more common in children with hypoxic encephalopathy, and if you can't rush to regenerate and repair the myelin sheath correctly, the myelin sheath will continue to lose and cannot conduct neuronal information and cause incomplete cerebral palsy. **It is necessary to find out the cause of the disease before doing it correctly** in order to achieve the best recovery.

Suggestion: Myelin dysplasia diseases are also called'White matter malnutrition', which occurs because the white matter myelin sheath of the brain itself does not develop properly, and therefore most occur in infancy, childhood, or adolescence. And there are often genetic factors.

Common types of leukodystrophy are as follows:

The most common one is called adrenoleukodystrophy (ALD), which is a genetic disease with X chromosome, which mostly occurs in boys (age and 12 years old are two peaks), mostly starting from vision and hearing impairment, indicating the onset of the second half of the brain, clinical symptoms are basically symmetrical, and can also be accompanied by signs of adrenal damage, such as **darkening, blood pressure drop, etc. CT or magnetic resonance imaging can be seen on large confluent lesions, and most of them are in the back of the brain, and if the plasma can detect an increase in very long-chain fatty acids (fatty acids above C20), or an increase in the ratio of C26 to C20, it has a definite diagnostic value.

This is followed by a recessive genetic disorder called metachromatic leukodystrophy (MLD), which occurs mostly in infants, young children, or children (a similar disease is often not found in previous generations when asked about the medical history), and the clinical manifestations range from.

At the age of one or two, there can be easy excitability, limb inactivity, and then the whole body muscle tone gradually decreases first and then increases, and there are staged movement disorders such as convulsions and limb paralysis, and angular arch reflexion and general muscle stiffness appear in the late stage. Tests of blood and urine are markedly reduced in aryl thioriesterase activity, and the presence of parachromatic particles (meaning that unusually red, acidic particles can be stained with a blue basic dye) in urine centrifugal sediment are reliable evidence for the diagnosis.

Yes, but you have to go to a regular hospital**.

Myelin dysplasia, also known as leukodysplasia, is a condition in which white matter does not develop normally, and symptomatic epilepsy is mainly treated with oral medications** such as valproate or oxcarbazepine, depending on the type of epilepsy.

Cerebral myelin dysplasia, i.e., white matter dysplasia. In the process of myelination, due to the influence of certain factors or hereditary defects of a certain enzyme, myelination disorder is caused, which affects the development of white matter in the brain.

Children with cerebral myelin dysplasia show significant motor delays at an early stage than normal children. Movements such as lying prone and raising the head, sitting, standing, and walking all start at a later age than normal children of the same age. Walking is especially noticeable, often not walking on its own until the age of 3-4 or 4-5 years old, and it is unsteady.

Cerebral myelin dysplasia is extremely harmful to children, delayed myelin development, a manifestation of cerebral dysplasia, the cause of respiratory distress at birth, brain hypoxia caused, early as soon as possible**!

Delayed myelin development, i.e., white matter dysplasia. The possible reason is that in the process of myelination, due to the influence of genetic factors or hereditary defects of a certain enzyme, the myelination disorder is caused, which affects the development of white matter in the brain, and the child can manifest as developmental retardation, convulsions, ataxia, nystagmus, optic nerve atrophy, cortical blindness and sensory deafness.

Guidance: 1. The possibility of self-healing of this disease is still very small, and anti-epileptic drugs should be given**.

2. Then you can use drugs that nourish the nerves, protect the brain, and promote intellectual development**.