Can cells not grow old and why do cells age

No. Aging and death are a process that nature gives to genes.

Growth, development, aging and death are the inevitable laws of the life process of every organism, and they are also the inevitable laws of the life process of cells. However, the death of cells is not the same thing as the death of the body, and the death of cells is not the same as the death of the body. And the death of the body does not mean that all cells die at the same time.

In the newly dead body, the white blood cells in the blood vessels are still moving, the epithelial cells of the trachea and bronchi are still swinging, and some organs can be transplanted or tissue culture because the cells are still alive. In the life process of the body, there is also the death of a large number of cells, even in the embryonic period, such as the continuous renewal of peripheral blood cells and epithelial cells in the human body, the disappearance of tadpole tails, and so on.

It can be seen that the aging and death of cells are not exactly the same thing as the aging and death of animals or human bodies. Nerve cells, cells that are particularly important for an individual, die first after an individual's death, and their death causes the death of other cells and the end of an individual's entire life.

How can cells not age? It's just that after this cell dies, another cell will grow, which is metabolism. When the metabolism is vigorous when you are young, the metabolism gradually declines when you get old, and when the new cells cannot replace the dead old cells in time, the person will die of old age

It is completely impossible, thousands of cells die and fall off every day.

No way, cellular senescence is normal. Senescent cells are constantly being replaced by new cell years.

Be physically and mentally healthy. Conditioning the body and so on... Eating our Infinitus health food can slow down aging...

Eat more protein foods.

There is a special antioxidant defense system in the human body, which is a variety of antioxidant enzymes that prevent free radical damage, such as SOD, which are constantly checking where DNA damage occurs in the human body and repairing it in time. It is precisely because of the presence of these enzymes that the human body always maintains its basic antioxidant function. However, the synthesis of these enzymes relies on a large number of amino acids, and only a steady supply of protein can keep the antioxidant enzymes at a high level, so people should consume protein appropriately.

Eat more antioxidant vitamins.

The scientific community recognizes that there are three major antioxidant vitamins: one is carotene, which is mainly a precursor to the synthesis of vitamin A, which is quite rich in yellow and green vegetables and fruits such as red grapes, spinach, kelp, sweet potatoes, mangoes, and pumpkins. The second is vitamin C, which is contained in various vegetables and fruits.

The third is vitamin E, which is higher in various nuts, wheat germ, eel, and vegetable oil. These vitamins help prevent free radical damage to the body's cells.

Avoid smoking and drinking, and eat less fried and processed foods.

Smoking and drinking alcohol can easily cause the formation of reactive oxygen species, fried foods are prone to produce lipid peroxide in the human body, and processed foods will also accelerate the aging of cells because they contain more chemicals.

1. Excessive macromolecular cross-linking is a major factor of aging, such as DNA cross-linking and collagen glue can damage their function and cause aging.

2. After the accumulation of cell metabolites to a certain amount, it will harm the cells and lead to the aging of the cells.

3. Free radicals contain unpaired electrons, which are highly reactive, which can trigger chain self-slag rotten radical reactions, such as denaturation and cross-linking of DNA, proteins and lipids, especially polyunsaturated fatty acids and other macromolecular substances, damaging DNA, biofilms and structural proteins and functional proteins, thereby causing the occurrence of various phenomena of aging.

4. The mitochondria that exist in the cells to provide energy are prone to mutations, and the accumulation of mutations is likely to be one of the reasons for the aging of the human body.

Normal point is a normal physiological phenomenon of cells.

Over the past few decades, many hypotheses have been proposed to explain the causes of cellular senescence in order to elucidate the mechanism of cellular senescence. Each of these theories has its own evidence, but the evidence tends to emphasize one aspect and is therefore one-sided. It is true that the cause of cellular senescence may be caused by multiple factors, so it is normal for there to be multiple theories about cellular senescence, which may be the reason why the mechanism of cellular senescence has not been truly revealed so far.

At present, there are two main theories with promising development: one is the theory of free radicals; The other is the theory of programmed cell death.

1.Free radical theory. The cellular metabolic process is inseparable from the presence of oxygen, and the biological oxidation process is the process by which the cell obtains energy, but at the same time as this biological oxidation process, some highly active compounds will be produced, which are by-products of the biological oxidation process.

Experiments have shown that the by-products or intermediates of these biological oxidation processes are directly related to cellular senescence, and they can lead to changes in cell structure and function, which is the free radical theory of cellular senescence.

The so-called free radicals are those chemicals with an odd number of electrons, i.e., they all carry unpaired free electrons that cause the high reactivity of these substances. There are a variety of reasons why free radicals are produced inside cells. For example, free radicals are released during processes such as biological oxidation, radiation, exposure to pollutants, and enzymatic reactions within cells.

The normal free radical reaction process in cells includes three processes: initiation, expansion, and termination. Once free radicals are generated, they can continue to multiply. Free radicals cause very much damage to cell membranes and also damage many cellular components.

However, the process of free radical amplification can be terminated at any time, and excess free radicals produced in the cell can be removed by adding exogenous compounds. For example, protective enzymes are added, mainly superoxide dismutase (SOD) and catalase (catalase), which work synergistically to play a protective role. In addition, other antioxidant molecules, such as vitamin E and vitamin C, are added, which are effective terminators of free radical reactions.

2.Programmed cell death theory. Most cells of an animal undergo normal natural death at a certain stage of development, known as programmed cell death. There is a fundamental difference between programmed cell death and cytopathological death.

The phenomenon of programmed cell death is ubiquitous and occurs in hormone-dependent tissues, as well as in lymphocytes, thymocytes, hepatocytes, **, and cells during embryogenesis.

Regarding the characteristics of the programmed cell death process, the most obvious morphological changes are chromatin condensation within the nucleus and degradation of DNA into oligonucleotide fragments, which are associated with the expression of certain specific proteins. Programmed cell death has been shown to be a physiological control mechanism of cells.

Cellular senescence and apoptosis are normal phenomena, and there are many theories of various mechanisms, but they mainly revolve around the regulation of the nucleus....

The causes of cellular senescence have always been a topic of great concern to researchers and difficult to answer. In recent decades, a variety of hypotheses have been proposed, such as somatic mutation and DNA damage theory, free radical theory, and cell programmed death theory, etc. However, there is no hypothesis that can really reveal the cause of cellular aging.

Current scientific research suggests that cellular senescence may be the result of a combination of internal and external factors.