How long does it take to get rid of hyperkalemia?

Generally, it can be cured in about three years, because this is a very serious disease, if it is not on time, the consequences are unimaginable, so we must protect our body.

Generally, it takes about half a year to heal, because this disease is actually more difficult, and there is no need for surgery at the time, so the time will be longer.

Generally speaking, it takes **1 to 2 weeks to heal, and this situation can be treated with drugs**, which is very convenient.

It takes a month. During the period, you must control your diet and take more medicines, so that the effect will be better.

You can take medicine**, or the most basic way**, and you can also drink traditional Chinese medicine to recuperate, these ways can alleviate the symptoms of such a disease, and can regulate the health problems of the body.

In the process of hyperkalemia, we must pay attention to diet and strictly control the intake of potassium in the diet. Then it is necessary to adjuvant the drug to carry out**. That's how you can **.

Hemodialysis or peritoneal dialysis may be used, but peritoneal dialysis** is less effective and slower.

By exchanging sodium and potassium ions in the colon, the absorption of potassium ions is reduced and fecal excretion is promoted; Hemodialysis can rapidly reduce serum potassium levels in patients for whom emergency dialysis is indicated. Hyperkalemia is rare in normal people because the above procedure can rapidly reduce hyperkalemia. The body rapidly excretes potassium ions from the urine.

Potassium is mainly transferred from intracellular to extracellular (common in acidosis). Calcium ions can stabilize the cell membrane, stabilize the cell membrane, reduce permeability, and reduce the flow of potassium ions. Patients with uremia often have varying degrees of anemia and upper gastrointestinal bleeding.

One way to reduce the symptoms of anemia is through blood transfusions. Blood transfusions develop from the trunk to the extremities and may affect breathing and movement. Typical changes on ECG include a high T wave, a prolonged Q-T interval, and an increase in the QRS complex.

Eliminate factors that may be causing a sustained increase in potassium in the blood. Stop eating potassium-containing foods and medications, including bananas, oranges, tangerines, potatoes, potassium-sparing diuretics, and ACE inhibitors. Use potassium-containing diuretics.

Foi-CAT exchange resins are used to promote potassium excretion. Vitamin C and probiotics can have unexpected effects over a period of time, hypokalemia can lead to increased acetylcholine release, abdominal pain, nausea and vomiting, and when the disease reaches a certain level, high potassium can have a toxic effect on the muscles, leading to quadriplegia and respiratory arrest.

Potassium is one of the nutrients that the body needs. It keeps the nerves and muscles, including the heart, working properly. In healthy people, the excretion of potassium increases with increasing intake.

Cardiovascular symptoms: high potassium inhibits myocardium, changes heart rate, easily induces malignant arrhythmia, and even cardiac arrest, which is the main cause of death from high potassium. Hypoadrenaline signs and osmotic disturbances, including hyperglycemia.

In addition, renal failure and/or increased potassium secretion in the distal tubules results in an increase in serum potassium.

Hyperkalemia is the concentration of potassium in the blood above milliliters, which is very common in the elderly, and symptoms such as muscle weakness and irregular heartbeat will occur after the disease. Not only does it cause great harm to the body, but it can also be life-threatening in serious cases, so the elderly should be active when they suffer from hyperkalemia. So, what is the approach to hyperkalemia in the elderly?

First of all, the cause of hyperkalemia in the elderly should be eliminated and foods with high potassium content should be avoided. Then the main purpose of using calcium preparations to lower blood potassium is to reduce blood potassium by diluting calcium gluconate and then injecting it intravenously to counter the toxicity of high potassium to the heart. This method of using insulin with intravenous glucose injections or using sodium bicarbonate to correct acidosis is fast and effective, can reduce the concentration of potassium in the blood in a short period of time, and is a very convenient and effective method that does not reduce the total amount of potassium in the body.

Second, drugs that promote potassium excretion, such as the diuretic prosemide, can be used to lower potassium in the blood through dialysis. To achieve the goal of hyperkalemia.

Hyperkalemia in older adults is characterized by muscle pain, slow heartbeat, and a tendency to irregular heartbeats. Hypercalcemia in the elderly is mainly divided into mild and severe. In mild cases, drugs containing potassium salts and potassium can be stopped immediately, and normal saline can be injected if necessary**.

In severe hyperkalemia, potassium antibodies** or glucose and insulin injections as directed by the doctor are given to promote potassium transfer to the cells. Hyperkalemia can also damage the kidneys, limit acidic excretion in the body, and predispose to acidosis symptoms.

Hyperkalemia is caused by diabetes mellitus and is accompanied by diabetic symptoms such as thirst, polyuria, and hunger. If the elderly accidentally suffer from hyperkalemia, it is recommended to actively go to the hospital to clarify the severity of the disease and choose the appropriate method** to prevent complications caused by the condition and avoid more serious damage to the body.

Patients with acute onset of hyperkalemia should take emergency measures, and different methods should be taken according to the severity of the disease.

1.Acute severe hyperkalemia.

1) High blood potassium can reduce the resting potential of cardiomyocytes while the threshold potential remains unchanged, so that the gap between the two decreases, so that the excitability of cardiomyocytes increases. Calcium ions may widen the gap between the resting potential and the threshold potential of the myocardial cell membrane, and stabilize the myocardial excitability. The emergency measure is an immediate intravenous bolus of 10% calcium gluconate 10 ml, which is completed in 5 to 10 minutes, and if necessary, 1 2 minutes later and another intravenous injection, which can quickly eliminate ventricular arrhythmia.

Because the action of calcium is short-lived, an intravenous infusion should be continued after an intravenous bolus. 10% calcium gluconate 20 40ml can be added to 500ml of normal saline or 5% glucose solution for intravenous infusion. Calcium has no effect on serum potassium concentration.

2) ** method of lowering serum potassium: Plasma with extracellular potassium is temporarily moved into the cell. Hypertonic glucose and insulin can be given intravenously.

In patients with heart failure or kidneys, the infusion rate should be slow. If you want to limit the amount of water you want to intake, you can increase the concentration of glucose solution to 25% to 50%. Potassium changes and hypoglycemic reactions are closely monitored during the instillation process.

Intravenous bolus of 5% sodium bicarbonate solution can also be given, followed by 5% sodium bicarbonate 150 250ml intravenous infusion. This approach is more appropriate in patients with metabolic acidosis. It both allows extracellular potassium to move into the cell and corrects metabolic acidosis.

The results are less satisfactory for patients with end-stage renal failure who are on dialysis to maintain life. In patients with end-stage renal failure, hemodialysis may be used to remove potassium from the body.

2.Mild-moderate hyperkalemia**.

1) Low-potassium diet Potassium intake is limited to 50-60mmol per day.

2) Stop precipitating medications Stop all medications that may cause potassium elevation in the blood.

3) Cation exchange resin to reduce intestinal potassium absorption and excretion of potassium from the body. For example, sodium ethylene sulfonate resin or sodium polyvinylbenzene can be taken orally, or an enema can be retained, but oral administration is more effective than an enema. During the enema, 40 grams of resin can be placed in 200 ml of 20% sorbitol solution as a retention enema, and the stool can be released after 1 hour.

4) Removal of triggers: Removal of hyperkalemia or diseases that cause hyperkalemia.

3.Dialysis. is the fastest and most effective method. Hemodialysis or peritoneal dialysis may be used, but the latter is less effective and slower. Hemodialysis with low- or non-potassium dialysate can cause potassium to drop almost immediately after the start of dialysis, and potassium will almost always return to normal after 1 to 2 hours.

Peritoneal dialysis can exchange about 5 mmol of potassium with an exchange of 2 L per hour with ordinary standard dialysate, and 180 240 mmol of potassium can be removed after 36 to 48 hours of continuous dialysis.

High potassium is just a symptom, and the most important thing is to find the cause of high potassium.

The kidneys are the organs that excrete potassium, and high levels of potassium in the blood may be related to kidney failure. Hyperkalemia may lead to cardiac arrest, and peritoneal dialysis or hemodialysis is preferable for hyperkalemia due to renal failure. Diuretics can be used appropriately in patients with oliguria.