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ataxia, or cerebellar disease.
This condition can be caused by a variety of diseases.
A detailed examination should be done at the neurology department of the hospital.
To do symptomatic treatment, it is best to find out**.
Cause**.
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Ataxia refers to the clinical manifestations of disordered coordination, instability and uncoordination of limb movements in the absence of muscle strength.
Causes of symptoms. a) Cerebellar ataxia.
b) Deep sensory impairment ataxia.
c) Cerebral ataxia.
4) Vestibular ataxia.
Common diseases. Cerebral palsy, congenital tumors, sleeping pill poisoning, dementia syndrome, Behcet's syndrome, multiple infarct dementia, temporal lobe tumors, rheumatic chorea, alcoholic cardiomyopathy, acute hydrogen sulfide poisoning, progressive multifocal leukoencephalopathy, intracranial metastases, acute disseminated encephalomyelitis, Langerhans cell histiocytosis, Alzheimer's disease, Meniere's disease, cerebral infarction, cysticercosis, cerebral tuberculoma, chronic progressive chorea, ascarin poisoning, angioreticulocytoma, pediatric hypertension, Pancreatic encephalopathy syndrome, chorea, primary immunodeficiency disease, sedation, diazepam, hypnotic drug poisoning, adenocarcinoma, epidermoid cyst, skull base tumor, glioma, Binswanger's disease, paralytic dementia, etc.
Diagnosis 1, ataxia.
Pay attention to the onset and course of the disease, which is usually acute onset ataxia and episodic, and is more likely to have vestibular lesions and vertigo epilepsy. Acute cerebellar lesions, inflammation of the central nervous system and traumatic brain injury are more common in patients who have a rapid onset and worsens in a short period of time. Cerebrovascular disease, traumatic brain injury, especially cerebellar hemorrhage, are more common in patients with acute onset and rapid deterioration, and sometimes life-threatening.
Ataxia due to alcoholism and vitamin deficiencies can improve ataxia after improving nutritional status. There is remission of ataxia with ** and multiple sclerosis is more common.
2. Age and family history.
It is of great reference in the diagnosis of ataxia. In childhood, he has congenital cerebellar hypoplasia, hereditary diseases, acute cerebellar ataxia in childhood, encephalitis and other polyperoneal muscular atrophy ataxia, hypertrophic interstitial neuropathy, syringomyelia, etc. Young and middle-aged patients with onset can be seen in dentate erythronuclear atrophy, olivet pontine cerebellar degeneration, subacute combined degeneration, telangiectasia ataxia, etc.
Middle-aged and elderly people are more common in cerebellar atrophy, vertebraebrobasilar insufficiency, cerebellar hemorrhage, cerebrovascular disease, etc. Ataxia has genetic factors such as congenital cerebellar hypoplasia, acute cerebellar ataxia in childhood, juvenile spinal hereditary ataxia, hereditary ataxia polyneuritis, hypertrophic interstitial neuropathy, dentate erythronuclear atrophy, olivet pontine cerebellar degeneration, telangiectasia ataxia, etc.
Check the physical exam.
Correct voluntary movement requires the involvement of many muscles, including agonist, synergistic, antagonist, and immobilizer muscles. The examination includes finger nose test, heel knee and shin test, rapid rotation test, rebound test, finger test, toe test, and start test.
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