Magnesium valproate sustained release tablets can t be crushed and eaten

Sustained-release tablets are long-acting drugs and cannot be crushed orally, and the duration of the effect will be shortened.

Magnesium valproate sustained-release tablets have made users happy. Magnesium valproate sustained-release tablets can adjust the mood, sodium magnesium valproate sustained-release tablets are used for various types of epilepsy, especially the following types, absence seizures, myoclonus, tonic-clonic seizures, atonic seizures, mixed seizures, partial seizures, simple or complex seizures, secondary generalized seizures, special types of syndromes, it can also be used for bipolar disorder manic episodes.

Precautions for magnesium valproate sustained-release tablets.

1. Valproate bonded magnesium sustained-release tablets must be used under the guidance of a physician, and when the doctor asks you about your special allergy history, physical condition, underlying diseases, recent medication and other issues, you should be truthful to help the doctor eliminate drug contraindications for you and avoid the risk of re-medication. Depending on the patient's constitution and severity of the condition, the dosage prescribed by the doctor will vary, so please follow the doctor's instructions for dosage.

2. Patients should pay attention to monitoring relevant indicators during medication, such as liver function, blood routine, platelet count, etc.

3. Because drinking alcohol can aggravate the sedative effect of magnesium valproate, do not drink alcohol during the use of rock seepage medicine.

Summary. The properties of magnesium valproate sustained-release tablets are white tablets or off-white tablets. Its main ingredient is magnesium valproate.

Magnesium valproate sustained-release tablets are used for ** various types of epilepsy, especially the following types: absence seizures; myoclonic seizures; tonic-clonic seizures; atonic episodes; mixed seizures; partial epilepsy; onset of simplicity or complexity; secondary, generalized seizures; Specific types of syndromes. It can also be used for manic episodes of bipolar disorder.

It is valid for 24 months.

Magnesium valproate extended-release tablets exposed to the outside for half an hour: will they be invalid? I put half a dot in the palm of my hand. Does the drug still work???

I was exposed to the pills several times, for more than half an hour. I also put the pill in the palm of my hand for half an hour.

Will the medicine fail?

Will the drug be less effective?

Please be sure to be honest with me. This medicine is very, very important for me. It's two bottles, not one or two tablets. Please, be sure to tell the truth.

Hello dear, I am Xinghai, I am very happy to answer the question for you. Not hail will fail the pro. This product should be sealed and stored.

The so-called sealing refers to the use of outer packaging to encapsulate another thing or the source of the old sedan car. Affects the storage conditions of the drug: temperature.

Ray. Oxygen and metal ions. Humidity and moisture.

Packaging materials. The properties of magnesium valproate sustained-release tablets are white tablets or off-white tablets. Its main ingredient is magnesium valproate. Magnesium valproate extended-release tablets are used for ** all types of epilepsy, especially the following types:

absence seizures; myoclonus seizures; tonic-clonic seizures; atonic episodes; mixed seizures; partial epilepsy; onset of simplicity or complexity; Followed by generalized seizures; Specific types of syndromes. It can also be used for manic episodes of bipolar disorder. It is valid for 24 months.

If you have any concerns, please consult your doctor.

I would like to ask if the medicine will not work?

The pro that will not fail.

Will the drug be less effective? I exposed the pill to the air and put it in the palm of my hand. Half an hour. Will the medicine fail?

Normal storage should be sealed. You take it out for half an hour, and the effect is still there.

There will be no reduction in queries here.

For your health.

Follow your doctor to take your medication.

(1) Common digestive disorders (nausea, stomach pain), mostly appear at the beginning, but do not need to stop, and the symptoms usually disappear within a few days. (2) Abnormal liver function: very few cases of severe liver damage or even death have been reported, and the most high-risk patients (especially those receiving a variety of antiepileptic drugs**) are infants and children under 3 years old with severe seizures, especially those with brain damage, mental retardation and hereditary metabolic or degenerative diseases, after 3 years of age, the risk decreases significantly, and further decreases with age, in most cases, liver damage appears in the first six months.

Suspicious symptoms: clinical symptoms are the basis for early diagnosis, especially before the onset of jaundice, and the presence of the following symptoms should be considered for liver damage, especially in high-risk patients. a Non-specific symptoms, usually sudden onset, such as fatigue, anorexia, lethargy, drowsiness, sometimes accompanied by recurrent vomiting and abdominal pain.

b Epilepsy**: Patients (or family members of the child) should be warned that when the above symptoms appear, they should report to the doctor in time, and immediately undergo clinical examination and liver function tests. Observation:

Liver function tests should be performed before **, and liver function tests should also be done regularly during the first 6 months, especially in high-risk patients, and in general examinations, tests reflecting protein synthesis, especially prothrombin rate, are the most relevant. When it is determined that the abnormal low prothrombin rate, especially if accompanied by other biochemical abnormalities (fibrinogen and coagulation factors are markedly reduced, bilirubin and aminotransferases are elevated), it is necessary to discontinue **, as a preventive measure, such as patients taking salicylate at the same time, should also be credited, because the metabolic pathways of these drugs are the same. (3) Neurological disorders, during the valproic acid drug **, a small number of patients develop drowsiness or stupor, and lead to transient coma (encephalopathy), when**, it can appear alone or at the same time as seizures, when the dose is reduced or stopped, these symptoms will be reduced, these cases are often found in combination **, especially with phenobarbital or after a sudden increase in the dose of valproic acid.

4) transient and/or dose-related adverse effects have been reported: alopecia, mild postural tremor, and drowsiness. (5) Decreased simple fibrinogen or prolonged bleeding time have been reported, usually without clinical signs, which mostly occur at high doses (valproic acid has an inhibitory effect on platelet aggregation in the second stage) (6) Blood system:

Most of them are thrombocytopenia, and rarely anemia, leukopenia, or pancytopenia. (7) Occasional reports of pancreatitis, sometimes leading to death. (8) Vasculitis has been reported (9) Simple and mild hyperammonemia without abnormal liver function occurs from time to time, but does not need to be stopped**.

10) Weight gain, amenorrhea and menstrual disorders have also been reported. (11) Reversible or irreversible hearing loss has occasionally been reported, but a causal relationship has not yet been clarified. (12) Valproic acid can cause ** reactions, such as rashes.

Toxic epithelial necrolysis, Stevens-Johnson syndrome, and erythema multiforme have also been reported in some cases. (13) Reversible fanconi syndrome with valproic acid** has been reported separately, but its mechanism of action is unknown.

Drug overdose, plasma valproic acid concentration of 120 g ml, *** significantly increased, early manifestations of nausea, vomiting, diarrhea, food phobia and other digestive symptoms, followed by muscle weakness, tremor of limbs, ataxia, drowsiness, confusion or coma, abnormal liver function, thrombocytopenia, pancreatitis, etc., once signs of poisoning are found, the drug should be stopped immediately. For patients who overdose, the hospital should take the following rescue measures: emptying the digestive tract and ensuring effective urination and cardiac respiratory testing.

In very severe cases, extracorporeal dialysis should be performed if necessary.