What is a fibrotic lesion of the right lung and what does partial fibrosis in the lungs mean?

Pulmonary fibrosis is the abbreviation of "diffuse interstitial lung disease", that is, "interstitial lung disease", pulmonary fibrosis is a difficult disease in the world, there is no specific drug at present, the disease can be effectively controlled if it is carried out in time, and if it is not accepted in time to standardize, it is likely to further develop lung parenchymal lesions. But there is no such thing as ** in medicine.

Hello 1 Pulmonary fibrous lesions lead to a decrease in the elasticity of local lung tissue, a decrease in function, and if the area increases, it can lead to dyspnea. l

Pulmonary fibrosis is not timely and easy to cause a variety of concurrent harm, its harm involves many aspects of the body, and has the characteristics of difficulty, slow recovery, etc., many patients fail to be timely for various reasons, resulting in aggravation of the disease, in order to improve everyone's attention to the disease, to reduce greater damage to the patient's health.

What are the harms of pulmonary fibrosis** if it is not timely?

1. Malignant diseases: patients with pulmonary fibrosis and scleroderma have an increased possibility of malignant diseases, especially lung adenocarcinoma, alveolar cell carcinoma, and oat cell carcinoma.

2. Cor pulmonale, and even heart failure: patients with pulmonary fibrosis suffer from chronic hypoxia and progressive pulmonary hypertension, often combined with right ventricular hypertrophy and cor pulmonale. Left-sided heart failure is also common and is associated with ischemic heart disease.

3. Bullae and spontaneous pneumothorax: The alveoli of patients with pulmonary fibrosis are highly distended, and the alveolar walls are thinner and easy to rupture, and they fuse with each other to form a large mass bullae. A sudden increase in pressure during sudden exertion, such as a violent cough, heavy lifting, or sports, causes the bullae to rupture, forming a spontaneous pneumothorax.

4. Lung infection and respiratory failure: patients with pulmonary fibrosis have decreased lung immunity due to weakened lung function, and most of them use glucocorticoids and immunosuppressants in the routine process of Western medicine, which increases the incidence of lung infection. When lung infections are not well controlled, respiratory failure can be induced.

1. Pulmonary fibrosis is a major class of end-stage changes in lung diseases characterized by fibroblast proliferation and accumulation of a large number of extracellular matrix, accompanied by inflammatory damage and tissue structure destruction, that is, normal alveolar tissue is damaged and repaired abnormally, resulting in abnormal structure (scarring). The vast majority of patients with pulmonary fibrosis** are unknown (idiopathic), and this group of diseases is called idiopathic interstitial pneumonia (IIP), which is a large group of interstitial lung diseases. Idiopathic interstitial pneumonia (IIP) is characterized by Idiopathic Pulmonary Fibrosis (IPF), which is a severe interstitial lung disease that can lead to progressive loss of lung function.

Pulmonary fibrosis seriously affects the respiratory function of the human body, manifested by dry cough, progressive dyspnea (self-denial of qi), and the patient's respiratory function continues to deteriorate as the condition and lung damage worsen. The incidence and mortality of idiopathic pulmonary fibrosis are increasing year by year, and the average survival time after diagnosis is only one year, and the mortality rate is higher than that of most tumors, which is called a "tumor-like disease".

2. **: When lung damage is caused by a variety of reasons, the interstitium will secrete collagen for repair, and if it is over-repaired, that is, fibroblasts are overproliferated and extracellular matrix is accumulated in large quantities, pulmonary fibrosis will be formed.

Symptom. First, in the early performance.

Pulmonary fibrosis is not obvious in the early stage of the disease, but the main manifestations of the disease are cough and dyspnea, and the symptoms will be aggravated with labor. Some patients also experience significant difficulty climbing stairs.

Symptom. 2. Manifestations of medium-term exacerbations.

Secondary infections develop and worsen with infectious diseases. At this point, the patient may feel rapid breathing without wheezing, hemoptysis or chest pain, and fatigue.

Pulmonary fibrosis is a malignant disease that can lead to many other serious diseases. Even heart failure, chronic hypoxia and progressive pulmonary hypertension in patients with pulmonary fibrosis are often combined with right ventricular hypertrophy and cor pulmonale. In patients with fibrosis, the alveoli are highly distended, and the alveolar walls become thin and easily ruptured and fuse with each other to form large mass bullae, which can rupture the bullae during sudden exertion, such as vigorous coughing, heavy lifting, or exercise, and form a spontaneous pneumothorax.

So go to the hospital as soon as possible**, I heard that the Beijing Armed Police Corps Hospital is good, you can consult it.

There are many classifications of pulmonary interstitial fibrosis, which are divided into two categories: known and unknown, and the onset of the disease is insidious, and the disease develops rapidly.

Among the known forms of pulmonary fibrosis, the most common ones are infections, occupational environmental factors, drugs, radiation, and connective tissue disorders. For example, inorganic dust including silica, asbestos, talc, antimony, aluminum, coal, beryllium, barium, iron, etc. Infectious**Infections with fungi, viruses, and atypical pathogens.

Drug factors include amiodarone, bleomycin, methotrexate, etc. Connective tissue disorders such as rheumatoid arthritis, polymyositis, dermatomyositis, Sjögren's syndrome, and systemic lupus erythematosus can also cause interstitial pulmonary fibrosis. Radioactive factors, on the other hand, usually refer to the radioactivity** of the tumour (referred to as radiotherapy).

Long-term high-concentration oxygen therapy can also cause pulmonary interstitial fibrosis.

The mechanism of pulmonary fibrosis is unknown**, and risk factors include smoking and environmental exposure. Relatively high-risk industries such as timber, metal mines, and metal product manufacturing can be uncertainties. Smoking is recognized as the most important hair element of the disease.

Patients with pulmonary interstitial fibrosis can exercise appropriately to enhance the body's resistance, but they should do what they can, do not exercise vigorously, they can do more abdominal pursed lip breathing, avoid colds as much as possible, and look for the cause of local fibrosis, ** underlying disease.

Common symptoms.

Pulmonary fibrosis usually develops between the ages of 40 and 50 years, and is more common in men than in women. Dyspnea is the most common symptom of pulmonary fibrosis. In mild pulmonary fibrosis, dyspnea often occurs during strenuous activity.

When pulmonary fibrosis progresses, dyspnea also occurs at rest, and patients with severe pulmonary fibrosis may have progressive dyspnea. Other symptoms include dry cough and fatigue. Some patients have clubbing and cyanosis.

Fibrosis of lung tissue is a serious consequence, resulting in structural changes in normal lung tissue and loss of function. When a large number of fibrotic tissues without gas exchange replace the alveoli, oxygen cannot enter the bloodstream. Patients suffer from poor breathing, hypoxia, acidosis, loss of labor, and in severe cases, death.

Generally, if pulmonary fibrosis occurs, it is mainly due to long-term chronic lung inflammation, which is not well controlled or not effectively controlled at that time, and lung calcification will occur later, that is, pulmonary fibrosis. Cough, sputum production. There is no cough in the early stage, and there may be a dry cough or a small amount of mucus sputum later, and secondary infection is susceptible.

Mucopurulent or purulent sputum is present, and occasionally bloody sputum is present. Dyspnea. Exertional dyspnea progressively worsens, shallow tachypnea may involve nasal alar twitching and accessory muscles, but most do not have orthopnea.

Pulmonary fibrosis is a major class of lung diseases characterized by fibroblast proliferation and a large number of extracellular matrix accumulation, accompanied by inflammatory damage, tissue structure destruction, that is, normal alveolar tissue is damaged and abnormally repaired, resulting in structural abnormalities. It has many clinical symptoms, which generally occur at the age of 40-50 years. Males are more common in females.

Dyspnea is the most common symptom of pulmonary fibrosis, and in mild pulmonary fibrosis, dyspnea often occurs during strenuous exercise, so it is often overlooked or misdiagnosed as another disease. When pulmonary fibrosis progresses, people can also have difficulty breathing at rest. Patients with more severe pulmonary fibrosis may have progressive dyspnea, and other symptoms include dry cough and fatigue.

Force, a serious consequence of fibrosis of lung tissue, can lead to changes in the structure of normal tissues and loss of function. When a large number of fibrotic tissues without gas exchange function replace the alveoli, the patient may have poor breathing, lack of oxygen, loss of labor, and in severe cases, death.