What should I pay attention to when I have renal insufficiency with gout?

In this case, it is especially important to pay attention to diet, many of which should be avoided or eaten less. Like some foods high in fats and beans, shiitake mushrooms are not good for gout.

First of all, it is especially important to drink more water, and water should be alkaline water, although lemonade has an acidic taste, but after metabolism produces more sodium, potassium, calcium and magnesium ions, it is actually "alkaline food". Also, try to avoid strong tea, strong coffee and drinks that contain high sugar. Pure milk, on the other hand, is actively recommended and has a certain uric acid-lowering effect.

Meat: Red meat such as beef, mutton and pork contains too many purines and should be restricted, while white meat such as chicken contains quite less purines and can be eaten appropriately. At the same time, be sure to avoid the intake of broth, such as mutton soup, beef broth, pork rib soup, hot pot, etc., which will seriously increase uric acid.

Seafood must be avoided, and freshwater fish such as herring and grass carp can be eaten appropriately and must not drink alcohol, and at the same time, soy products and salt can not be eaten.

1. Maintain uric acid levels.

Maintaining uric acid levels is important for gout**. In patients with chronic gouty nephropathy, it is particularly important to lower uric acid** with the aim of keeping serum uric acid below 327 mol L.

2. Lifestyle changes.

A good lifestyle can avoid the occurrence of disease, but when it does occur, it can be done by improving the lifestyle. If you have chronic gouty nephropathy, you should combine work and rest, go to bed early and get up early, and pay attention to a light diet, which is very helpful.

3. Drugs that promote uric acid metabolism.

Patients should take drugs that promote uric acid excretion** gouty nephropathy. Sodium bicarbonate can be taken under the guidance of a doctor, or drugs such as carboxybenzene sulfonamide, benzensulfonazolone, benzbromarone, etc., can effectively reduce blood uric acid concentration and improve hyperuricemia.

In summary, chronic gouty nephropathy refers to the long-term presence of a large amount of uric acid in the patient's blood, which needs to be excreted by the kidneys. Long-term use can cause renal tubular and renal interstitial damage, mainly low specific gravity urine, mild proteinuria and nocturia in the early stage, and renal failure in the later stage, and some patients can develop uremia. Therefore, it is necessary to avoid all kinds of animal offal, seafood, alcohol, and broth and other foods high in purines, and at the same time, patients should drink more water and excrete uric acid, so as to reduce the burden on the kidneys.

If it is chronic ventilation, most of this situation is due to the renal insufficiency, which leads to renal insufficiency, the ability to excrete some uric acid decreases, the uric acid increases in the blood, and the pain caused by the large joints is removed. In this case, you need to take some uric acid-lowering drugs orally**, and at the same time, it can improve kidney function, usually drink more boiled water, and the general situation will get better.

Renal insufficiency and gout affect each other, progressing each other, will cause the severity of each other's disease to aggravate, both have corresponding drug programs, gout must control uric acid, slow kidney medicine on those several, the platform is not easy to say, consult a doctor.

Answer]: B This question examines the choice of gout** drug. Colchicine, non-steroidal anti-inflammatory drugs and glucocorticoids are all used for acute gout**, and drugs that lower blood uric acid, including allopurines that inhibit uric acid production, and benzbromarone and probenecid, which promote the excretion of uric acid through the kidneys, are used in the interval between gout attacks, so the answer to question 3 is d.

Colchicine can be used for acute gout not tolerated by nonsteroidal anti-inflammatory drugs; Glucocorticoids** are effective in acute gout, but generally only in patients who cannot tolerate colchicine and non-steroidal anti-inflammatory drugs or have relative contraindications. When patients with renal insufficiency have acute gout and fibrillation, colchicine and non-steroidal anti-inflammatory drugs should be used instead of glucocorticoids, so the answer to question 1 is b.

What is the reason why the kidneys do not excrete acid in gout patients? Gout is an acute arthritis caused by a purine metabolism disorder. Most of this is due to excessive uric acid production and partly due to decreased uric acid excretion. **First of all, we should pay attention to dietary control and avoid fatty meat, seafood, and animal offal.

Long-term smoking and drinking, staying up late for a long time, eating high-sugar, high-salt and high-protein foods for a long time, and long-term strenuous exercise can cause damage to body organs.

If the concentration of uric acid in the human body is too high, crystals will form and precipitate in the kidneys, where they cannot be excreted.

Because gout is caused by the body's high uric acid, which is a disease of the metabolic system, it is likely to cause kidney failure, and the function of the kidneys to metabolize uric acid will become worse and worse.

Are there any other symptoms of gout besides pain?

Gout kidney damage – kidney damage due to increased blood uric acid, resulting in hematuria, proteinuria, and even renal insufficiency; In gout patients, a large amount of blood uric acid is excreted from the kidneys, which can also cause "kidney stones", which are mostly caused by hyperuricemia.

Therefore, patients with gout will have joint symptoms, such as "gout stones", and impaired kidney function, while other concomitant diseases such as hypertension, diabetes, and hyperlipidemia are often combined.

Many patients with gout do not have very good liver and kidney function, is this the cause of the disease itself or the damage of gout ** drugs?

This issue is complex and requires a case-by-case analysis. In general, poor liver and kidney function may be caused by gout itself, liver and kidney damage caused by drugs for hyperuricemia, and may also be caused by complications of gout, such as obesity, hyperlipidemia, hypertension, etc.

1.The disease itself is a factor.

Long-term hyperuricemia may lead to abnormal kidney function, and the excretion of large amounts of uric acid from the kidneys will increase the burden on the kidneys.

2.Comorbidities with other medical conditions.

If the patient has comorbid hypertension, his kidney function may also be poor, and long-term hypertension can cause damage to blood vessels, heart and kidneys. In addition, patients with hyperlipidemia are more likely to develop fatty liver, so their liver function may also be poor. If the patient has other underlying medical conditions such as hypertension, hyperlipidemia, and hyperglycemia, these diseases can also affect the patient's liver and kidney function.

3.Effects of uric acid-lowering drugs.

As we mentioned earlier, drugs like allopurinol, which inhibit uric acid synthesis, can damage liver function; Drugs that promote uric acid excretion, such as benzbromarone, can also increase the burden on the kidneys.

Therefore, for a certain patient, the damage to liver and kidney function may be the result of a combination of the above factors.

For patients with poor liver and kidney function, what adjustments need to be made to their ** regimen?

First of all, for patients with poor liver and kidney function, it is necessary to give a certain amount of liver protection and kidney protection**, and at the same time of gout, we must not forget the protective properties of these two organs**.

For patients with poor liver function, drugs with less liver damage should be preferred when choosing urate-lowering drugs. Allopurinol is very damaging to liver function, so we choose benzbromarone or febuxostat. Similarly, for patients with poor renal function, we use allopurinol or febuxostat.

The general principle is to avoid the weakest organs, and such ** is reasonable.

Gout is a sudden illness. Most people with gout have no aura before the onset of gout. They usually have elevated blood uric acid for many years before the onset of the disease, but usually have no symptoms.

There are no aura symptoms in an acute gout attack. A small number of patients present with symptoms of fatigue, allergies, and mild joint pain prior to the onset of illness. In the case of fatigue, it often occurs suddenly, and the pain is unbearable at the time of the attack.

At this time, they often go to the emergency room for medical attention.

Dear, gout is indeed a bad kidney, but the root cause of gout is the poor metabolism of the liver that causes the kidney to function badly! Therefore, to completely ** gout, it is necessary to start with the liver.

It has a lot to do with it, because the kidneys are not good, the body's ability to decompose and excrete purines decreases, and more purines in the blood will be converted into uric acid.

Gout is a relatively common and complex arthritic disease, at present** gout mainly includes relieving pain and inflammation during attacks, as well as preventing gout attacks, patients should maintain a reasonable weight, remember to drink plenty of water, avoid cold or moisture, and avoid joint damage.

It is recommended to choose an orthopedic hospital with medical insurance qualifications, which is more targeted, and can also be trained under the guidance of doctors, which is of great help to the condition, and the medical insurance charges are more reasonable, and can also reduce economic pressure, I hope it will help you.

It has a great impact, and the symptoms of gout are very harmful to the kidneys, and at the same time, it will also aggravate the difficulty of the disease, and it will also endanger the health of the body, and it will also increase the chance of disease.

Some lesions may occur, and cause renal insufficiency, because gout will have high uricemia, and these things have a very big impact on the kidneys.

The impact of gout on the kidneys is particularly large, it is very likely to cause kidney stones, it may also cause kidney disease, and it may also lead to renal insufficiency.

Gout can cause damage to the kidneys, causing gouty nephropathy, manifested by intermittent proteinuria, as the disease continues to progress, it can be accompanied by increased nocturia, hypertension, edema, azotemia, can also cause kidney failure, and can also cause uric acid nephrolithiasis.

The effects of gout on the kidneys include affecting the parenchymal function of the kidneys, such as glomerulonephritis, tubular damage, or abnormal kidney function. There is also a condition in which urinary stones appear, such as kidney stones, ureteral stones, etc.

Gout and kidney syndrome, gout is more severe. Elevated uric acid in the blood is the underlying cause of gout, and supersaturated uric acid precipitates and can be deposited in tissues, causing histological changes in gout. In the human body, there are many by-products of uric acid production, and reactive oxygen species are one of them.

Overproduction of reactive oxygen species can also damage various tissues except joints and kidneys. Therefore, joint and kidney diseases are only the tip of the iceberg caused by gout.

Gout or hyperuricemia, poorly controlled in the long term, can affect the kidneys and lead to kidney disease. The occurrence of renal lesions is directly proportional to the level and duration of serum uric acid. About one-third of patients with chronic gout will develop renal impairment, mainly sodium urate nephropathy, which manifests as low back pain, edema, hypertension, and proteinuria, which can progress to renal failure or uremia.

If urate is deposited in the kidneys, urinary stones such as renal colic, hematuria, and chronic urinary tract infections can also occur, which can eventually lead to kidney failure. If large amounts of urate are widely deposited in the renal tubules, acute urate nephropathy, oliguria, anuria, and even rapid renal failure can manifest.

Disorders of electrolyte metabolism. Repeated use of diuretics or unreasonable long-term salt deprivation can cause hyponatremia secondary to nephrotic syndrome; The use of adrenocorticosteroids and massive diuretics leads to profuse urination. If potassium is not replenished in a timely manner, hypokalemia is predisposed.

Coronary heart disease. Patients with nephrotic syndrome often have hyperlipidemia and hypercoagulability, and are therefore susceptible to coronary artery disease. The incidence of myocardial infarction in patients with nephrotic syndrome is 8 times higher than in normal people.

Coronary heart disease has become the third leading cause of death from nephrotic syndrome. Thrombosis. Patients with nephrotic syndrome are prone to thrombosis, especially membranous nephropathy, with an incidence of up to 25% to 40%.

Patients with gout and kidney syndrome are advised to drink plenty of water, because the main function of the kidneys is to produce urine and excrete various metabolic wastes. The amount of water you drink will have a direct impact on the amount of urine you produce. Insufficient urine production will inhibit the excretion of metabolic wastes such as uric acid, and metabolic wastes that cannot be excreted from the body in time will accumulate in the kidneys, resulting in impaired kidney function.

I think kidney syndrome is more severe. Because kidney syndrome can cause many complications, it is particularly detrimental to health.

Gout is more serious because the disease is likely to cause some kidney syndromes, and the impact on the body is relatively large.

Kidney syndrome is very serious, this disease is very difficult to ***, can produce serious ***, can kill people.

Kidney syndrome is more severe, can cause other complications, is difficult to be completely **, and brings more pain.