Why both hyperthyroidism and hypothyroidism cause myxedema

Hyperthyroidism: localized myxedema.

Localized myxedema is a toxic goiter.

Unique** symptoms, this is also a **symptom of common hyperthyroidism. Its occurrence probability accounts for about 5% of hyperthyroidism, because it occurs in the anterior part of the lower tibia, so it is also called myxedema in the anterior tibial region. This sign is mostly seen in the anterior and lower end of the tibia, sometimes extending to the dorsum of the foot and knees, and occasionally on the dorsum of the hands and abdomen, and the pre-onset lesion site ** The surface is uneven and brownish-red.

Dark red or reddish-brown plaque lesions, about 5-30 mm in diameter, up to several centimeters when connected into patches, then thickened and toughened, forming a swollen and coarse surface from the knee down, like rubber legs, and the later ** hair follicles are obviously enlarged and changed like bark.

Hypothyroidism: Thyroid hormone deficiency affects all tissues throughout the body, so the symptoms are multifaceted. The most important pathophysiological change caused by thyroid hormone deficiency is the presence of a large amount of extracellular mucinous material in the interstitial spaces of various tissues throughout the body, which is composed of acidic mucopolysaccharides (hyaluronic acid, chondroitin sulfate).

Mucin formed in combination with proteins. This mucin is extremely hydrophilic, absorbs a large amount of water, and deposits it in the interstitial spaces of various tissues. It is manifested in **, gradually infiltrated by mucin and acidic mucopolysaccharides and swelling, **surface keratinization, atrophy, hair follicles and sweat glands.

Keratinized embolism is specific.

Please go to the local regular hospital for treatment, and do not believe the best method and the miraculous effect of a certain prescription said by a certain Chinese medicine practitioner in a hospital.

Hello; Chronic lymphocytic thyroiditis, Hashimoto's disease. (1) Early stage, hyperthyroidism. (2) In the medium term, hyperthyroidism and hypothyroidism coexist.

3) Late, hypothyroid. Most of the reasons are caused by the inflammatory destruction of the thyroid gland and the increased release of thyroid hormone into the blood. Recommended Chinese Medicine**. If you are satisfied with the answer, please promptly.

If necessary**, please contact us and we will do our best to help.

Myxedema coma is a complication of life-threatening hypothyroidism. Features include a long course of hypothyroidism, coma with very low body temperature, loss of reflexia, and seizures.

1. Hypothyroidism, myxedema, myxedema, is caused by the appearance of more mucopolysaccharides, which are common in hypothyroidism. Chronic thyroiditis, thyroidectomy and other reasons can lead to a decrease in thyroid cells, which will induce thyroxine insufficiency and new hypothyroidism. When hypothyroidism occurs, it will affect the normal metabolism of fat, sugar and protein, resulting in the accumulation of mucopolysaccharides and other components under the skin, resulting in swelling, and the color will mostly appear pale or sallow, which is called myxedema.

Second, there are many cases of hypothyroidism myxedema causing this condition, so you should go to the hospital in time to check in a regular hospital, and carry out ** under the guidance of a doctor. Regular life can avoid the occurrence of some diseases, insist on exercising to improve the body's immunity, eat more vegetables and fruits, and eat less high-quality and high-protein foods, which can enhance the body's fitness.

3. Symptoms of hypothyroidism, myxedema.

Patients with hypothyroidism often have typical symptoms such as fatigue, unresponsiveness, slowed movements, general weakness, and loss of appetite. If it is not diagnosed in time, the disease will develop further and myxedema, also known as hypothyroidism crisis, which is a serious stage of the patient's disease development due to the long-term insufficiency or lack of thyroid hormone in the internal circulation. If it is not effective, it will be life-threatening.

The main manifestations are loss of consciousness, low body temperature, low blood pressure, bradycardia, hyponatremia, hypoglycemia, hypoventilation, and patients may also have **dryness, thinning hair, hoarseness, tendon reflexia, macroglossia, edema, goiter, etc.

It can cause hypoglycemia, enlarged thyroid, cerebral hemorrhage, epilepsy, and even edema.

Possible complications are generalized edema, hypoproteinemia, reduced blood thickness, nephrogenic edema, and renal insufficiency, all of which are complications of hypothyroidism.

It can lead to epilepsy, edema, hoarseness, slow reactions, low body temperature, and loss of mind, which can lead to many diseases.

Hyperthyroidism generally refers to hyperthyroidism, and patients with hyperthyroidism have edema symptoms, which may be related to their own hyperthyroidism. It may also be associated with hyperthyroidism involving other systems, and prompt endocrinology is recommended.

Hyperthyroidism is a multisystem, complex autoimmune disease that can cause inflammatory cell infiltration and cytokine and chemokine production. A large number of immune complexes are deposited in the blood vessels, causing capillary inflammation, increased capillary permeability, and large amounts of fluid extravasation leading to the formation of effusion, resulting in edema symptoms.

In addition, severe hyperthyroidism may lead to heart failure, increased capillary hydrostatic pressure, increased fluid exudation to form effusion, and may cause increased central venous pressure, impaired lymphatic return, decreased collateral circulation, and large amounts of fluid into the serous cavity, which may cause edema. Hyperthyroidism may also cause abnormal protein metabolism due to chongshiqing, resulting in hypoproteinemia, decreased plasma colloidal osmotic pressure, and excessive exudation of fluid in the capillaries, resulting in edema symptoms.

After suffering from hyperthyroidism, you need to take care of your daily diet in time, and do not eat foods high in iodine, such as kelp, seaweed, jellyfish, etc. And it should be targeted under the guidance of a doctor, such as taking methimazole tablets, propylthiouracil tablets and other drugs according to the doctor's instructions to control the condition within the normal range.

1. Understand the mood of hypothyroidism myxedema: fear of illness and rush to the doctor, hate illness for quick results, spare money and do not seek medical treatment, etc., should help patients eliminate their thoughts and establish self-confidence.

2. For hypothyroidism, myxedema should pay attention to the changes in the patient's breathing, pulse, heartbeat and blood pressure. If you have concurrent psychiatric symptoms, pleural and ascites and pericardial effusion, intractable angina, heart failure, myxedema coma, you should be sent to the hospital immediately**.

3. Patients with hypothyroidism have a deficiency and cold constitution, and cold drugs should be prohibited or used with caution when using sleeping drugs, so as to prevent coma from being induced. Diuretics are easy to use intermittently, pay attention to urine output, whether there are electrolyte imbalances, and prevent hypokalemia.

4. Pay attention to dietary care when hypothyroidism myxedema**: most hypothyroid patients have a deficiency and cold constitution, so they should not eat raw, cold and ice food. The diet is mainly vitamin, high protein, and high in calories. Eat plenty of iodine-rich foods such as fruits, fresh vegetables, and kelp.

This problem is 13 years old, but I would like to correct the one upstairs.

Page 357 of the Physiology Book, decreased TH secretion, impaired protein synthesis, and interstitial mucin deposition, which allows water molecules to remain subcutaneous, causing myxedema.

The above are the physiological mechanisms of myxedema.

Hypothyroidism due to graft nail inflammation. Thyroxine supplementation** is required. At present, you can take 50ug of Alpha** every morning, and the thyroid function will be rechecked after one month, and the dose of the drug will be adjusted according to the results.

Department of Nuclear Medicine, Second Xiangya Hospital-Liu Guofeng, Attending Physician.

Chronic lymphocytic thy-roiditis, also known as Hashimoto thyroiditis, is an autoimmune disease.

This disease is more common in middle-aged women, manifested as goiter, slow onset, often unintentionally stuffy pants, the volume is about 2 3 times that of the normal thyroid gland, the surface is smooth, the texture is tough and elastic like rubber, and the obvious nodules are pure and rare, no tenderness, no adhesions around it, and can move with swallowing. In the late stages, a small number of symptoms of mild local compression may occur.

The disease progresses slowly, and sometimes the goiter does not seem to change significantly for several years. Initially, the thyroid gland is functioning normally. In the course of the disease, sometimes hyperthyroidism can also appear, followed by normal function, hypothyroidism, and then normal, the process is similar to subacute thyroiditis, but not accompanied by pain, fever, etc., so this state is called ** thyroiditis, and the postpartum onset is called postpartum thyroiditis.

However, when the thyroid gland is destroyed to a certain extent, many patients gradually develop hypothyroidism, and a few have myxedema. The disease is sometimes associated with pernicious anemia due to the presence of autoantibodies to the parietal cells of the stomach.