Gout takes febuxostat, uric acid drops to 240, can you stop the drug

OK. It can also be halved.

Cut two pieces of medicine.

Eat for a while. Look again.

The crystals have not yet dissolved. Eating non-step also depends on your physique, not all physiques can eat. Long-term eating will have ***.

Whether it is allopurinol and febuxostat, which inhibit the production of uric acid, or benzbromarone, which promotes uric acid excretion, they are only "symptomatic"**, and they all act on the downstream of the uric acid elevation mechanism, and do not fundamentally solve the cause of uric acid elevation. In other words, despite the use of acid-lowering drugs, the cause of hyperuricemia is not **.

At the same time, after the administration of uric acid-lowering drugs in patients with gout, the effect of the drugs causes the uric acid in the blood to drop to the target level, but urate crystals deposited in joints or other tissues may still remain. When the dose of acid-lowering drugs is reduced or stopped, the concentration difference between the joint and the blood due to the presence of uric acid crystals produces a difference in the concentration of uric acid, which makes the uric acid in the joint transfer to the blood, and at this time, the ability of the drug to control the blood uric acid is no longer enough, so the level of uric acid in the blood will rise sharply.

The transfer of uric acid in the joints to the blood, coupled with the persistence of hyperuricemia, makes the uric acid appear after the drug is reduced or stopped in gout patients.

At this time, you can choose to reduce the dosage and continue to take it or cooperate with traditional Chinese medicine to repair the damaged body functions.

Summary. Hello, glad to come to your questions. You're basically back to normal now.

First of all, I would like to send you my blessings. Wishing you good health!

Your previous uric acid reached more than 700, which is indeed too high. Then you are instructed by the doctor to take the medicine. It has now reached 295 and has returned to a very normal value.

In general, if the uric acid level is below 480, you can basically stop taking the drug.

So, I hope you keep a good attitude. But those can be left without medication.

However, I have to go to the doctor for regular check-ups.

I hope mine helps you and I wish you good health.

I used to have more than 700 uric acid, and after taking febuxostat for 1 month, I added it from 10 grams to 40 mg, and now my uric acid is 295, do I still need to continue taking medicine.

Hello, glad to come to your questions. You're basically back to normal now. First of all, I would like to send you my blessings.

Wishing you good health! Your previous uric acid reached more than 700, which is indeed too high. Then you are instructed by the doctor to take the medicine.

It has now reached 295 and has returned to a very normal value. In general, if the uric acid level is below 480, you can basically stop taking the drug. So, I hope you keep a good attitude.

But those can be left without medication. However, I have to go to the doctor for regular check-ups. I hope mine helps you and I wish you good health.

The doctor also let me eat for 1 month without reducing the amount.

Well, then according to the doctor's instructions. The doctor wants you to strengthen and strengthen.

Febuxostat, which is familiar to gout patients, is a commonly used drug to lower uric acid, although the effect is relatively obvious, but improper use will still bring many problems and even ***. Like many patients, why does uric acid still ** after taking febuxostat?

How does febuxostat lower uric acid?

Uric acid is a product of purine metabolism, and the component of febuxostat is a xanthine oxidase inhibitor, which mainly works by reducing the concentration of uric acid or urate in the blood, so as to avoid the attack of gout. This type of drug is suitable for most patients with high uric acid and gout. Compared with other acid-lowering drugs such as allopurinol, it can reduce the level of uric acid in the blood more quickly.

Why does febuxostat uric acid still happen?

Although febuxostat has obvious effects, it only blocks the second half of the uric acid synthesis pathway and prevents excessive uric acid synthesis, but cannot fundamentally solve the increase in uric acid synthesis. Therefore, once the drug is stopped, the synthesis of uric acid will not be blocked, and over time, it will cause uric acid to rise, urate crystallization, gouty arthritis attacks, etc.

Precautions for taking febuxostat.

1) Pay attention to taking it on time and in accordance with the amount, and the uric acid is more stable.

In order to maintain long-term stability and avoid the situation of **, it is necessary to take it on time and in accordance with the amount, and the dosage can be gradually increased with the change of the condition. Please follow your doctor's instructions for specific dosages.

2) Note***, uric acid lowering is safer and more effective.

Because the drug is metabolized through the liver, it can be used appropriately in patients with mild to moderate liver damage and high uric acid, but it is not recommended for patients with severe liver damage.

3) Pay attention to the combination of dietary management, the effect of acid reduction is better.

During a gout attack or when uric acid is too high, it is forbidden to take high-purine foods, and low-purine foods and medium-purine foods can be appropriately consumed. The three recognized categories of high-purine foods include seafood, soups and offal.

Simply taking these uric acid-lowering drugs can only temporarily alleviate the symptoms of gout, and there will be recurrence after stopping the drug, which is a symptom but not a cure, and will only cause recurrence and prolongation. It is recommended to try traditional Chinese medicine, which can systematically regulate symptoms and syndrome differentiation, reduce swelling, relieve pain, reduce uric acid, regulate the spleen and strengthen the kidney.

The high level of uric acid in the blood can be caused by excessive uric acid production, poor uric acid excretion, and both.

Uric acid is a purine metabolic end product in the human body. Therefore, the current low-purine diet advocated is actually a way to reduce the synthesis of uric acid from the source, which is very important and is the premise and basis for the prevention of gout. However, its ** effect is very limited, and it can only reduce the level of uric acid in the blood.

mol l, which is often just a drop in the bucket for many patients with hyperuricemia.

In addition, it is also possible to reduce the production of endogenous uric acid by taking drugs such as allopurinol and febuxostat to prevent our human body from processing and synthesizing uric acid. This is the most advanced method often used by doctors at present, and from the perspective of clinical practice, its effect is still very limited for a considerable number of gout patients.

This is because, in the attack mechanism of gout, the weight of excessive uric acid production is actually not high, and more patients have varying degrees of poor uric acid excretion: the body cannot effectively remove excessive uric acid, so that uric acid accumulates in the blood, which eventually leads to the occurrence of gout and its related complications.

For this reason, urate-stimulating acid excretion drugs have a wider range of adaptations, and its representative drug, benzbromarone, is currently the first-line drug for gout**.

Refutation of rumors: Febuxostat is strong in lowering uric acid, no ***, you can eat it with confidence, listen to what the doctor says.

Yes, that can be eaten for a long time, where did you buy 20mg