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Cerebellar atrophy is a non-organic disease, which is an incurable disease, and most of them are caused by genetics. It will lead to unsteady walking, ataxia, slurred speech, inability to take care of oneself, and seriously affect life expectancy.
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Cerebellar atrophy is a phenomenon in which the cerebellum shrinks in size and the sulcus widens. It can manifest as hereditary, neurological deformities, caused by acute cerebellitis or poisoning, and can induce cerebellar atrophy due to carbon monoxide, drugs, alcoholism, etc. This disorder causes symptoms such as gait instability, ataxia, and dysarthria.
Pathology: cerebellar atrophy is a neuroimaging manifestation in which the cerebellum is reduced in size and the sulci are widened. The disease's **:
Genetics: This condition can be hereditary, and if an immediate family member is a member of the cerebellar atrophy, then someone else will have a greater chance of developing the disease than the general population, such as spinocerebellar degeneration or ataxia atrophy. Poisoning:
Carbon monoxide poisoning can induce cerebellar atrophy, and drug poisoning can also predispose to these disorders, such as phenytoin poisoning. For regular drinkers, it is also possible to induce cerebellar atrophy, which is easy to atrophy because alcoholism is prone to alcoholic cerebellar degeneration. Disease:
Certain other types of disorders can affect the cerebellum, and symptoms of cerebellar atrophy, such as neuroparaneoplastic syndrome, may lead to a state of cerebellar atrophy. Symptoms: The cerebellum coordinates the balance of the body, so it is easy to affect cerebellar atrophy such as standing, walking, etc.
It may also affect upper limb movements, for example, trembling writing. It can also affect the language system, there will be a slow speech or lip ataxia.
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Cerebellar atrophy is not a disease but a neuroimaging finding. It can be seen not only in some hereditary and degenerative diseases, but also in some acute courses such as the late stage of acute cerebellitis and some drug poisoning, and even in some clinically asymptomatic people, cerebellar atrophy can also be seen in imaging examinations, especially in the elderly. Common features are decreased cerebellar volume and widened sulci on neuroimaging.
It can be divided into localized and generalized cerebellar atrophy. Because the cerebellum is mainly involved in the regulation of somatic balance and muscle tone, patients with cerebellar atrophy often have symptoms such as gait instability, ataxia, and slurred speech.
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Western medicine currently lacks a special **** method, telling that this disease cannot be cured, and can only take some nutritional neurohormone drugs or **** to improve symptoms, and these methods have no effect. The use of traditional Chinese medicine Rongjin Xing Yi decoction is suitable for the patient's condition to prescribe**, in order to achieve the medicine to the disease! Early detection, early attention, early recovery!!
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Cerebellar atrophy can cause symptoms such as unsteady walking, slurred speech, etc., and this condition is the first to occur.
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This disease is not very scary, and now it can also be effective** and delay the continuation of atrophy.
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Cerebellar atrophy is a chronic degenerative nervous system lesion of the brain caused by a variety of reasons, and symptoms worsen progressively with age, and brain atrophy is usually difficult**.
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Spinocerebellaratrophy, also known as spinocerebellar atrophy or spinocerebellar ataxia (SCA), is a chronic, progressive brain disease that mainly damages the spinal cord and cerebellum, and is mostly inherited in families.
Cerebellar atrophy is characterized by a series of genetic symptoms, mainly progressive uncoordinated steps, accompanied by disorders of hand movements, speech, and eye movements. The cerebellum usually shrinks.
Due to the different scope and development process of the lesions, there are many types of clinical signs of cerebellar atrophy, and its main symptoms are unsteady walking, poor movement, weakness in gripping, slurred speech, dizziness, heavy head, head swelling, headache, accompanied by diplopia or blurred vision, swallowing, trembling in writing, and urinary and urinary disorders.
Like other movement disorders, it is a condition that causes difficulty in moving the body and other associated symptoms due to the inability to coordinate fine muscle movements. This disease can be divided into cerebellar disorders, spinal cord dysfunction, autonomic nervous system disorders, and involuntary movement disorders.
The symptoms of each type are slightly different and vary from patient to patient. In general, the patient's mental capacity is not affected, but the body gradually loses control.
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Cerebellar ataxia is one of the more common diseases. The occurrence of the disease has brought a lot of impact to the patient. It can even make the patient lose the ability to take care of himself.
Changes in muscle tone can change from decreased lesion to spasticity, and the ataxic gait can change to spastic ataxia. Unsteady standing, leaning forward or swaying from side to side, more prominent when standing on tiptoe or heel, and easy to fall are often the most common symptoms in the early stage of the patient.
Name: Mr. Guan.
Age: 43 years old.
Course of disease: suffered from cerebellar ataxia for 4 years, main symptoms: dizziness, blurred vision, slurred speech, inaccurate hand grasping, small strength, unsteady legs, shaky walking, diarrhea, cold thighs and calves, poor sleep, dreaming, easy to wake up, sweating at night, hot hands and feet at night, constipation, burning sensation in urine, heart discomfort, palpitation, occasional tinnitus.
Often dry cough, insufficient gas, choking cough when drinking water, heavy salt taste like to drink water, heavy menstrual flow and blackness, easy temper tantrum, tongue with tooth marks, tongue coating cracks.
Previously**: After taking clonazepam, butylphthalide, methylcobalamin and other drugs, the condition was controlled, but *** was too large, but the condition never improved.
Now**: Brain Walking Soup**.
After a course of treatment: dizziness and cough improved slightly, sleep quality recovered, blurred vision, blurred ghosting improved.
After three courses of treatment: walking more steadily, insomnia and dreams better, urine and urine improved, and the feeling of cold legs and feet was better.
After six courses of treatment: walking steadily, drinking water and choking, difficulty in swallowing recovered, speaking clearly, dizziness was gone, the whole body muscle strength was basically restored, blurred vision was basically recovered, the legs and feet were cold, night sweats, heart, tinnitus improved, and then continued to consolidate a course of treatment, the symptoms were basically none, and now there is no recurrence in the return visit.
Suggestion: Patients with cerebellar ataxia should also maintain daily nutrition and calorie intake, and at the same time, patients with many sequelae of cerebral hemorrhage are prone to aphasia and inability to express their physical condition, and cerebellar ataxia occurs, resulting in a lack of nutrients in the patient's body and need to be ingested from food
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Cerebellar atrophy, ataxia, and multiple system atrophy are progressive diseases that can eventually lead to paralysis and inability to take care of themselves.
1. Adjust the airflow in the mouth through breathing exercises.
Breathing exercises can be used to strengthen the adjustment of airflow, oral movements to drive the movement of the tongue, exercise the coordination of local muscle movements, and maintain normal speech function, which can promote the recovery of patients with cerebellar atrophy who already have speech disorders.
2. Improve patients' interest through various forms of exercise.
It is difficult for patients to adhere to simple speech training for a long time, so it is necessary to enrich the training content in the form of tongue twisters, storytelling, questions, etc., to stimulate the patient's interest in practice, and family members should also supervise the patient to help the patient recover faster.
3. Stand and step.
Stand with your legs together, hold the back of the chair with your hands, and do a stepping motion, raising your legs as high as possible, repeating 10 times on each side.
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Cerebellar atrophy is mainly due to changes in the ventricles, sulci and gyrus of the cerebellum caused by various reasons, which is mainly manifested as the widening of the ventricles of the cerebellum and the deepening of the sulci of the cerebellum and the thinning of the gyrus, which is manifested as the appearance of a cavity in the cerebellum, the space becomes larger, and then the cerebellar tissue becomes smaller, which is called cerebellar atrophy.
Usually it can be seen in congenital factors, one is called cerebellar ataxia, which is related to genes and causes cerebellar atrophy. Acquired factors are called neurodegenerative diseases, which can also manifest as cerebellar atrophy. Cerebellar atrophy causes ataxia symptoms such as drunken gait, nystagmus, poetic speech, and impaired motor coordination, including the inability to counterfinger and decreased muscle tone.
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Cerebellar atrophy is divided into congenital cerebellar atrophy and acquired cerebellar atrophy, which requires early intervention to avoid further development of the disease and serious adverse effects on life, as follows:
1. Congenital cerebellar atrophy: usually caused by genetic lesions, such as congenital avascular cerebellar atrophy ataxia, patients present with obvious cerebellar atrophy symptoms, and pregnant women are usually required to undergo genetic screening in advance to avoid cerebellar atrophy caused by congenital gene deficiency in the fetus;
2. Acquired cerebellar atrophy: common in poisoned and elderly patients, such as cerebral vascular stenosis caused by insufficient cerebral blood supply and oxygen supply, cerebellar atrophy can occur.
In addition, it may be caused by drug poisoning, neurological lesions, etc., which can usually progress with the disease, resulting in a sharp decline in the patient's quality of life, and symptoms such as unsteady walking and difficulty in moving.
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It is a mental debilitating disease.
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Typical symptoms of cerebellar atrophy are unsteady gait, ataxia, slurred speech, oculomotor disorders, and nystagmus.
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Slight atrophy is a progressive disease and there is no way to be **.
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The continuous onset of cerebellar atrophy not only affects the life of patients, but also brings unnecessary troubles to the society and family, it is recommended that everyone should have a clear understanding of the whole disease of cerebellar atrophy, the elderly love Alzheimer's disease, the brain cells of the elderly begin to become dead and memory loss, which is the symptom of cerebellar atrophy.
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Ataxia refers to a disorder of coordination of movement with normal muscle strength. Disturbance of the amplitude and coordination of voluntary movements of the limbs, and inability to maintain body posture and balance.
However, it does not include coordination impairment in mild limb paralysis, voluntary movement deviation due to ophthalmoplegia, difficulty in voluntary movement due to visual impairment, and apraxia due to cerebral lesions.
Depending on the location of the lesion, ataxia can be divided into four types: deep sensory impairment ataxia; cerebellar ataxia; vestibular labyrinthine ataxia; Cerebral ataxia. The commonly known as "ataxia" refers to cerebellar ataxia.
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Cerebellar atrophy is not a disease to be precise, but rather a neuroimaging manifestation that manifests itself as unsteady walking and slurred speech.
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There are many precursors of cerebellar atrophy: First, when going up and down the stairs, the legs are uncoordinated, the muscles are relatively stiff, and the movements such as running, climbing, and playing ball cannot be completed accurately. Second, when standing still, the body may shake back and forth, and the water is unstable, and it is easy to hit the door or wall when walking.
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Cerebellar atrophy is a common condition of aging.
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Cerebellar atrophy is a neurological disorder that runs in families, and there is a 50% chance that one of the parents will inherit the disease. On the basis of years of clinical summarization and test prescriptions, Dr. Tian Feng, a 78-year-old Chinese medicine doctor, carried out dialectical classification according to the patient's personal physique, and studied a new idea of encephalopathy - "Five Elements Tongmai".
According to the different characteristics of encephalopathy, pathology and symptoms, it is mainly based on the principle of nourishing the liver and kidney, strengthening the muscles and bones, invigorating qi and strengthening the spleen, activating stasis and dissolving phlegm, and opening the body, regulating acetylcholine receptors, dissolving deposits, inhibiting the production of acetylcholine receptors, removing antibodies, immune complexes and other generosities in the patient's plasma, adjusting immune function, making the effect more consolidated and stable, and continuously improving the clinical recovery rate, relieving the troubles of many patients with ataxia and cerebellar atrophy that have not been cured for a long time.
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Cerebellar atrophy is a neuroimaging expression that can be seen in a variety of disorders.
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A chronic disease that is getting more serious the more it drags on, and it is recommended as soon as possible**
It's just that the child's brain has atrophied.
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