What is insulin resistance What should I do about the causes of insulin resistance?

The hypoglycemic effect after using insulin is not good--- please see my main post"Health Balance Interpretation"

Everyone is very enthusiastic, I am very touched! But still haven't figured out whether "insulin resistance" is a substance or a phenomenon?

At present, it is believed that IR is not only the pathogenesis of type 2 diabetes, but also the main thread that runs through a variety of metabolic related diseases, and is the link that connects them and is the common pathophysiological basis of these diseases.

Although the research on IR in traditional Chinese medicine started late, the research has become more active in recent years, and has gradually become a hot spot in the scientific research of traditional Chinese medicine in the prevention and treatment of diabetes.

The root cause of insulin resistance is that there is a problem with the insulin receiver in the human body, so if the insulin receiver is not completely repaired, and the secretion of insulin alone cannot fundamentally lead to insulin resistance.

The biguanide metformin is the golden key to solving this problem, which has been confirmed by several clinical and laboratory studies.

At present, the main methods that can reduce insulin resistance are biguanide metformin, angiotensin-converting enzyme inhibitors, insulin sensitizers, etc.

Metformin acts on the pancreatic nucleus? Pro-scandium, benzene carbuncle, Napu magpie nucleus, Zhi ditch sac, Zheng? Divide and divide?

0 years of history, since 1995 in the United States: ACE inhibitors can enhance postprandial insulin sensitivity and lower blood sugar; Insulin sensitizers do not secrete insulin and can enhance the sensitivity of the liver and muscles to insulin, but they are contraindicated in patients with liver and kidney insufficiency.

These drugs** have different mechanisms of insulin resistance and different efficacy, and metformin is the most used and the most experienced in research.

Summary. Hello, the most common insulin resistance is related to obesity, and if the body has insulin resistance, it can be improved by **, especially in individuals with visceral fat, large abdomen, and significant obesity. Most patients have high baseline insulin levels and then a decrease in fasting insulin levels, so it is critical for patients with insulin resistance.

If you have the conditions and are a relatively obese patient, you must lose weight. If insulin resistance persists after weight loss, drugs that improve insulin sensitivity, including thiazolidinediones, such as pioglitazone and rosiglitazone, can be very good at improving insulin sensitivity and insulin resistance. In addition, metformin also has the effect of partially improving insulin sensitivity and relieving insulin resistance.

Hello, the most common insulin resistance is related to obesity, and if the body has insulin resistance, it can be improved by **, especially in individuals with visceral fat, large abdomen, and significant obesity. Most patients have a high level of insulin in the underlying state, and the fasting insulin level decreases after reducing the weight of the body, so it is very important for patients with insulin resistance in the auspicious body. If you have the conditions and are a relatively obese patient, you must lose weight.

If insulin resistance persists after weight loss, drugs that improve insulin sensitivity, including thiazolidinediones, such as pioglitazone and rosiglitazone, can be very good at improving insulin sensitivity and insulin resistance. In addition, metformin also has the effect of partially improving insulin sensitivity and relieving insulin resistance.

What should I do if I treat canker sores with canker abrasion?

Eat vitamin C plus vitamin B

What to do with canker sores.

Eat vitamin C plus vitamin B

Usually eat a little more fruit.

Insulin resistance is a state in which the body's sensitivity to insulin's hypoglycemic effects decreases, resulting in the need for an excess of insulin to achieve a normal hypoglycemic effect. For example, 10 units of insulin would normally be useful if there is insulin resistance, but 520 units of insulin would be required. To put it more colloquially, it is a unit of insulin that does not exceed one force, and it takes several to do it.

Therefore, in order to maintain the body's normal blood sugar level, pancreatic islets have to work hard to produce more insulin to complete the task of lowering and maintaining normal blood sugar levels. This will inevitably cause too much insulin in the blood, far exceeding the normal amount, and too much insulin in the blood is called hyperinsulinemia in medicine. It can be seen that hyperinsulinemia is the final result of insulin resistance.

Hyperinsulinemia can persist for a longer period of time, several years, or more than a decade. During this time, the pancreatic islets are overloaded and secrete much more insulin than normal, keeping the blood sugar level basically within the normal range, so the blood sugar is not high, and there will be no symptoms of diabetes. It is difficult to detect and diagnose this intrinsic abnormality that has in fact already appeared.

However, the ability of the human pancreatic islets to secrete insulin is limited, and it will not be inexhaustible, especially with such long-term, excessive and overloaded activities, the pancreatic islet function begins to decline and gradually develops in the direction of failure. The secretion of insulin is less and less, the hyperinsulinemia state can no longer be maintained, and the blood sugar gradually increases, first with abnormal fasting blood glucose, followed by impaired glucose tolerance, and finally becomes a diabetic patient. Therefore, insulin resistance is currently considered to be one of the main causes of type diabetes.

With the prolongation of the course of type diabetes, the progression of the disease, and the failure of pancreatic islet function, the secretion of insulin is getting less and less, and hyperinsulinemia in the early stage of the disease gradually becomes hypoinsulinemia, which is why insulin injections are also required for type diabetes. If hyperinsulinemia is gone, is there no insulin resistance? Of course not, because insulin resistance is the cause, and as long as the cause is not resolved, insulin resistance will always be there.

So, insulin resistance is not exactly the same as hyperinsulinemia. Hyperinsulinemia is a transitional state in which pancreatic islets are able to compensate for insulin resistance.